patho exam 3
Overview
This section focuses on the liver, its functions, diseases, and related pathology such as infection, inflammation, and cirrhosis.
Topics covered will appear on exam three.
Anatomy and Physiology of the Liver
Location:
Positioned in the right upper quadrant of the abdomen, beneath the diaphragm.
Weight:
Approximately 1,500 grams in adults.
Protection and Structure:
Encased with Gleason's capsule, made of thick connective tissue.
Organized into lobules.
Blood Supply:
Dual blood supply comprising:
Hepatic Artery: Supplies 25% of blood, a branch of the aorta.
Portal Vein: Drains venous blood from the gastrointestinal (GI) tract and supplies 75% of blood.
Portal Triad: Contains branches of the artery, venous blood vessels, and bile duct.
Liver Cell Types:
Hepatocytes: Functional liver cells responsible for several metabolic processes.
Kupffer Cells: Specialized macrophages that detoxify harmful substances; described as the "cleanup crew."
Sinusoids: Vascular spaces between hepatocytes containing a mixed blood supply.
Functions of the Liver
General Functions:
Digestion:
Manufactures bile, critical for digestion, secreted into the hepatic duct and common bile duct.
Bilirubin Conjugation:
Converts bilirubin into its water-soluble form with glucuronic acid.
Fat Metabolism:
Transforms triglycerides into fatty acids, cholesterol, and glycerol. Supports energy production.
Protein Metabolism:
Synthesizes and breaks down proteins. Produces most of the body's albumin; deamination converts proteins into ammonia, excreted in urine.
Carbohydrate Metabolism:
Stores glucose as glycogen. Processes glycogenolysis (breakdown of glycogen) and gluconeogenesis (creation of glucose).
Hematological Functions:
Produces coagulation factors using vitamin K.
Endocrine Role:
Pancreatic glucagon stimulates lipolysis and manages glycogen breakdown.
Detoxification:
Biotransformation occurs, changing substances for excretion, influenced by the first-pass effect.
Storage:
Stores vitamins (A, D, B12), iron, copper, and produces B lymphocytes.
Liver Dysfunction
Cholestasis:
Condition where bile flow is obstructed, causing a blockage similar to a traffic jam.
Hepatocellular Injury:
Inflammation damages liver cells, disrupting detoxification, leading to accumulation of drugs and metabolites.
Common Causes of Liver Dysfunction:
Viral infections (e.g., hepatitis), drugs, toxic environmental substances, excessive alcohol consumption.
Hepatitis Family
General information on Hepatitis:
Systemic liver infection primarily caused by viruses:
Hepatitis A: RNA virus, fecal-oral route, commonly through contaminated food/water.
Hepatitis B: DNA virus, transmitted via blood/body fluids; risk for liver cancer; has specific markers (s, c, e).
Hepatitis C: RNA virus, blood transmission, can remain dormant until symptomatic.
Hepatitis D: Depends on HBV to infect, accelerates damage.
Hepatitis E: Similar to Hepatitis A, associated with contaminated water in developing countries.
Clinical Symptoms & Examination:
Jaundice is a significant symptom, characterized by yellowing of the skin and eyes, first visible in the sclera. This stems from hyperbilirubinemia (elevated bilirubin levels in blood).
Causes of Jaundice:
Prehepatic: Excessive breakdown of red blood cells causing bilirubin overload.
Intrahepatic: Hepatocellular injury impeding conjugation.
Posthepatic: Obstruction of bile duct preventing bilirubin excretion (e.g., gallstones, tumors).
Chronic Hepatitis:
Lasting longer than six months, leading to continuing liver inflammation.
Hepatitis Overview
Hepatitis A (HAV):
Contaminated food/water (fecal-oral route); incubation period 14-21 days; prevention via vaccination and hygiene practice.
Hepatitis B (HBV):
Blood and bodily fluid transmission; can lead to cancer; four stages with a vaccine available.
Hepatitis C (HCV):
RNA virus, blood transmission, can remain asymptomatic for years.
Hepatitis D (HDV):
Dependency on HBV for replication; worsens liver disease.
Hepatitis E (HEV):
Fecal-oral route similar to HAV; predominant in developing countries, often related to water sanitation.
Treatment and Preventive Measures
Educate patients about the risks of medications like Tylenol, the importance of adhering to dosing instructions, and potential liver damage.
Promote lifestyle modifications such as weight loss, healthy diets, diabetes management, and increased physical activity for those with nonalcoholic fatty liver disease.
Highlight the potential dangers of mixing alcohol with medications, particularly with hepatotoxic drugs.