hemostasis
Overview of Hemostasis
Definition: Hemostasis refers to the arrest of bleeding or prevention of blood loss following injury to a blood vessel.
Importance: It is crucial for the body to react and respond to injuries (e.g., cuts) to prevent excessive blood loss.
Structure of Blood Vessels
Arteries:
Function: Carry oxygenated blood away from the heart to the body.
Characteristics:
Carry blood under high pressure, hence have thick elastic walls.
Do not have valves.
Blood pressure measurement reflects arterial pressure.
The pulse felt in arteries indicates blood flow.
Veins:
Function: Return deoxygenated blood to the heart.
Characteristics:
Operate under low pressure; hence when injured, blood oozes rather than spurts.
Have thinner walls than arteries and contain valves to prevent backflow.
Visible through the skin surface, often appearing bluish due to deoxygenation.
Do not have a pulse.
Capillaries:
Function: Site of gas, nutrient, and waste exchange.
Characteristics:
Smallest type of blood vessels with tiny diameters.
Susceptible to damage (e.g., resulting in petechiae).
Overview of Platelets (Thrombocytes)
Normal Count: 150,000 to 400,000 platelets per cubic millimeter.
Production:
Platelets are produced in the bone marrow.
Stored in the liver and spleen for ready access during hemorrhaging.
Lifespan: 7 to 12 days; undergo apoptosis upon expiry.
Macrohages: Responsible for cleaning up the dead platelets.
Laboratory Test: Platelet count is a part of the Complete Blood Count (CBC).
Coagulation Cascade
Concept: The cascade is triggered by either external tissue trauma or internal vessel damage.
Pathways:
Extrinsic Pathway:
Triggered by external injury leading to the immediate release of coagulation factors from the liver.
Measured by PT (Prothrombin Time) test, normal range: 11 to 13.5 seconds.
Intrinsic Pathway:
Activated by internal vessel damage.
Measured by aPTT (Activated Partial Thromboplastin Time) test, normal range: 25 to 35 seconds.
Common Pathway: Coagulation factors converge to form fibrin, ultimately contributing to clot formation to help healing.
PT/INR Testing:
PT time assesses extrinsic pathway functionality.
INR indicates blood coagulation levels, especially relevant for patients on anticoagulants (e.g., warfarin).
Normal INR: 0.8 - 1.1; therapeutic range for anticoagulant therapy: 2 - 3.
Coagulation Disorders
Thrombocytopenia (Low Platelet Count):
Causes: Autoimmune disorders (e.g., lupus), trauma (e.g., prosthetic heart valves), dialysis (fistula), chronic alcoholism (liver damage), and chemotherapy-induced suppression of bone marrow.
Thrombocytosis (High Platelet Count):
Causes: Stress, cancers, infections, and sepsis (systemic response).
Specific Coagulation Disorders
Hemophilia:
Common Types: Hemophilia A (factor VIII deficiency) is most prevalent.
Symptoms: Extended bleeding, spontaneous bruising, and prolonged aPTT.
Vitamin K Deficiency:
Caused by malnutrition, poor absorption, or liver disease.
Involves prolonged clotting time due to the role of vitamin K in the coagulation cascade. Binds to factor VII, affecting extrinsic pathway; abnormal PT.
Disseminated Intravascular Coagulation (DIC):
Life-threatening condition where clotting and bleeding occur simultaneously, often triggered by severe trauma, burns, or septic shock.
Results in rapid consumption of coagulation factors and simultaneous activation of fibrinolysis.
Recognition of Bleeding Disorders
Petechiae: Small pinpoints of bleeding from broken capillaries, typically seen on the skin.
Hematomas: Larger accumulations of blood outside blood vessels, indicating significant trauma.
Ecchymosis: Bruises indicating bleeding under the skin due to ruptured larger vessels.
Purpura: Larger areas of petechiae-like discoloration on skin, indicating more extensive capillary breakage.
Conclusion and Practical Application
The clotting process involves primary hemostasis (temporary plug formation) and secondary hemostasis (fibrin clot formation).
Nurses should recognize and react appropriately to signs of bleeding, including the need for pressure dressings post-venipuncture.
Understanding the coagulation cascade is essential for addressing coagulation disorders and tailoring treatments accordingly.