Reward ppt
Biol2052 Neuroscience
Focus: Complex brain functions related to pleasure, reward, and food.
Instructor: James Dillon (jcd@soton.ac.uk)
Learning Outcomes
Key competencies to demonstrate:
Describe components of reward: hedonic vs motivational aspects.
Identify brain structures involved in the rewarding effects of food.
Explain neural substrates related to different reward components.
Introduction to Pleasure and Food Reward
Overview of:
Pleasure derived from food and its impact on feeding behavior.
Increase in the prevalence of eating disorders.
Key brain structures coordinating feeding and the distinction between homeostatic and reward circuits.
Insights into eating disorders and their neural basis.
Epicurus' Perspective on Pleasure
Quote: “We recognize pleasure as the first good innate in us…”
Importance of pleasure in guiding future behavior choices related to food intake.
Eating pleasurable foods reinforces the desire to repeat the behavior.
Importance of Eating
Eating is vital for survival and maintaining homeostasis.
Pleasurable effects of food can drive consumption beyond metabolic needs.
Hedonic circuits ('liking') play a crucial role in the pleasurable experience of eating.
Eating and Reward Components
'LIKING'
Represents pleasure associated with food consumption.
'WANTING'
Motivational drive to seek food.
Both components contribute to the overall reward experience of eating.
Overview of Eating Disorders
Types include obesity, bulimia, and anorexia.
Severe obesity rates have nearly doubled among children in the past decade.
Understanding neural mechanisms behind food reward can lead to new therapeutic approaches.
Brain Systems Controlling Food Intake
Homeostatic System:
Orexigenic: Increases appetite.
Anorexigenic: Decreases appetite.
Key regions involved:
Hypothalamus, intestine, liver, brainstem, pancreas.
Impact of ablating orexigenic neurons: causes cessation of eating and mortality in rodent models.
Reward Circuit Studies
Brain stimulation and behavioral analysis techniques in animal models used to study reward.
Olds and Milner (1954) identified reward centers in rodent brains.
Specific electrode placements allowed mapping of reward circuitry.
Reward System Characteristics
The reward system evaluates rewarding properties of stimuli comprising multiple neural circuits.
Major areas involved include:
Nucleus Accumbens (NAcc)
Lateral Hypothalamus (LH)
Ventral Tegmental Area (VTA)
Medial forebrain bundle (MFB) enables robust self-stimulation behavior.
Neural Substrates of Hedonic and Motivational Reward
Questions on:
How to measure 'liking' reactions?
Berridge et al. (2020) quantified affective orofacial reactions to assess substance preference.
Endogenous Opioids in Reward Pathways
Types include:
Enkephalins, dynorphins, endorphins.
Opioid receptor subtypes:
mu, kappa, delta, are G-protein coupled receptors.
Agonists (e.g., morphine) vs. antagonists (e.g., naloxone) impact reward signaling.
Understanding Opioid Pathways
Morphine enhances 'liking' responses for palatable foods.
Naloxone decreases food intake, particularly with sucrose.
Identified 'hedonic hotspots' in the nucleus accumbens crucial for reward processing.
Hedonic Hotspots and Their Connectivity
There are additional 'liking' hotspots beyond NAcc.
Activation of mu opioid receptors enhances hedonic responses.
Hotspots interconnected, contributing to a circuit for pleasure.
Functional Connectivity Assessment
Fos plume method to examine functional connectivity among hotspots.
Injection with DAMGO and measurement of c-Fos expression as an indicator of neuronal activation.
The Role of Hotspots in 'Liking'
Recruitment of hotspots crucial for increased 'liking' responses.
Disruption of one hotspot alters overall circuit functioning.
Endocannabinoids in Reward Signaling
THCA enhances food intake, particularly of sweet substances.
Anandamide targeted injections increase liking expressions for sucrose in NAcc.
CB1 and mu receptors co-localized within neurons of NAcc.
Dopaminergic Pathways and 'Wanting'
Dopamine identified as a key neurotransmitter in reward system (Olds and Milner).
Evidence that dopamine is vital for the motivational aspect ('wanting') of food rewards rather than pleasure itself.
Summary of Key Concepts
Dopaminergic pathways generate 'wanting':
Anatomically larger than hedonic networks.
Can create strong motivation without affecting pleasure.
'Liking' circuits:
Composed of interconnected hotspots that can enhance pleasure responses.
Cross-talk between Homeostatic and Reward Circuits
Lateral hypothalamic area facilitates communication between homeostatic and reward systems.
Orexin as a candidate signaling molecule for this cross-talk, enhancing liking reactions.
Implications for Understanding Eating Disorders
Hypothesis: Excessive 'wanting' can surpass 'liking' leading to overeating.
Incentive sensitization related to hyperactivity in dopaminergic pathways can trigger food desires.
Future research utilizing neuroimaging may enhance understanding of dynamics leading to obesity.