Cell Biology: Enzymes
Properties of Catalysts
- Accelerate chemical reactions without being changed themselves * Most catalysts are enzymes but some ribozymes (RNA)
Properties of Enzymes
- Usually present in small quantities
- Not irreversibly altered during reaction
- Used many times
- Enzymes can be saturated or unsaturated
- May be extremely substrate specific
- Accelerate a favorable reaction
- Can be regulated
- Have no effect on the thermodynamic properties of a reaction
Why Don’t Most Thermodynamically FavorableReactions Occur Quickly?
- Most reactions require covalent bonds to be broken or formed * Requires certain amount of energy
- Energy of Activation * Minimal energy of reacting molecules * Few molecules have the amount of energy required
- Can increase the amount of reacting molecules by adding thermal energy * But is not practical in organisms
- Must lower EA → by an enzyme (catalyst)
Enzymes and ΔG
- Enzymes have no effect on ΔG
- If there was no EA barrier, reactions would proceed unregulated
How Enzymes Work
- Bring together reactants favorably
- Accelerate bond breaking and bond forming
- Enzyme-Substrate Complex * E+S ←→ ES ←→ E+P
- Substrate interacts with active site * Active site: groove or pocket on the surface of the enzyme * Made up of small clusters of amino acids that aren’t linear in the amino acid sequence
- Accelerating Reactions * Orientation: needs to be in a specific orientation for reaction * Reactivity: charges (may be electron donation between active site and substrate * Inducing Strain: changing position of substrate to put strain on it and make it easier to break
Substrate Binding and Activation * Substrate bonding usually ionic or hydrogen * Usually results in a conformational change in the enzyme * Lock and key model is outdated * Induced Fit is used model
Enzyme Activity and Regulation * Dependent on factors * pH * Temperature * Can only perform duties in certain parts of the body where the pH and temperature is good for the enzyme
Catalytic Event * 1. Substrate bound and activated * 2. Temporary bonds formed between substrate and active site * 3. Covalent bonds in substrate adjusted * 4. Product released * 5. Cycle repeats * Turnover number: number of time per second an enzyme can go through the entire sequence * Specific for every enzyme * And substrate * Varies widely depending on the requirements and needs of the cell * Max rate / enzyme concentration
Enzyme Kinetics * Graphs * Product vs time * Initial Reaction Velocity vs [Substrate]
Michaelis-Menten
- \ * K1- K4 are rate constants
- Assumptions * Only looking at initial conditions * K4 << K3 → product does not rebind to enzyme * [ES] is nearly constant * [Etotal] = [E free] + [E bound]; [E bound] = [ES] * K1 >> K3 → formation of ES is much faster than breakdown of ES to E and P
- Equation * K1 [E] [S] = K3 [ES] + K2 [ES]
(rate of making) = (rate of breaking down)
K2 and K3 = 1/time
K1 = 1/time*concentration
V = Vmax [S] / Km + [S] * V = initial velocity of product formation * Rate of enzyme reaction, increases with increased substrate until Vmax * [S] = initial substrate concentration * Vmax = maximum possible reaction velocity * Enzymes working as fast as possible * Km = kinetic parameter (k2 + k3) / k1 * Concentration of substrate at which rate is half Vmax
Vmax * Will occur when all enzyme is present in an ES complex * Velocity of product formation = k3 [ES] * Vmax = K3 [Etotal] * K3 = Kcat = Turnover Number
Km * At very low [S] * V = Vmax [S] / Km * → [S] << Km so Km + [S] ~ Km * Ignoring [S] value * First order reaction with a linear slope (Vmax/Km) * Rate dependent on the [S] * Enzyme reaches Vmax at low [S] and enzyme binds tightly * At very high [S] * V = Vmax [S] / [S] → Vmax * → [S] >> Km so Km + [S] ~ [S] * Ignoring Km * Zero order reaction (plateauing) * Vmax is initial velocity when [S] approaches infinity * Enzyme reaches Vmax at higher [S] and binds weakly * When Km = [S] * V = Vmax [S] / 2[S] → Vmax/2 * → [S] = Km so Km + [S] ~ 2[S]
Roles of Km and Vmax
- Km is independent of enzyme concentration; does not change when [E] changes * Consider it as playing for just one enzyme?
- Km is specific for each enzyme catalyzed reaction
- Vmax depends on the reaction conditions * Depends on enzyme concentration * More enzymes raise Vmax if the substrate stays the same?
- Km and Enzyme Affinity for Substrate * High affinity: K1 = high, K2 = low * Low affinity: K1 = low, K2 = high * When K1 is smaller, Km is bigger * Km is bigger → affinity is low * Km is smaller → enzyme affinity is high
Enzyme Inhibition
- Irreversible * Caused by outside forces * Forms covalent bonds with enzyme * Causes loss of enzyme activity * Usually involves an amino acid at active site * Toxic to cells
- Reversible
* Binds to enzyme in a way that can be reversed
* Active enzyme depends on inhibitor concentration of stability of Enzyme-inhibitor Complex
* Competitive Inhibition
* Mimics structure of the substrate
* Binds to active site but cannot be made into product
* Contests with the substrate for active site
* Reduces enzyme activity
* Apparent change in Km → looks greater
* Takes a higher concentration of substrate to meet Vmax
* Makes product slower -- need more substrate
* Substrate is fighting the inhibitor
* No change in Vmax * Makes enzyme less efficient * Noncompetitive Inhibition * Binds away from action site * Does not mimic substrate * Changes shape of active site so product cannot be formed * Substrate doesn’t fit into active site * Reduces enzymatic activity * Enzyme is not functioning * No relationship to concentration of substrate * Increasing substrate doesn’t help because it isn’t competing * Vmax is reduced because many enzymes unavailable * Less enzymes to work with
\n Regulation of Enzyme Activity
pH
Compartmentalization: where in the cell the enzymes used * Ex: lysosomal enzymes made in lysosomes * In acidic conditions as well
Inhibitors
Control concentration or availability of substrate
Allosteric regulation
Modification
Proteolytic Cleavage
Substrate Level Regulation * Glucose + ATP → Glucose-6-Phosphate + ADP * Hexokinase used for reaction * Hexokinase is inhibited when there is a lot of product
Allosteric Regulation * Regulatory molecules can cause enzymes to become active or inactive * Can bind and cause inactivity or activity * PKA example * Changing shape of the enzyme to either have or not have an active site available * Active: high affinity for substrate * Inactive: low affinity for substrate * Makes very little product
- Feedback Inhibition
* Product of one enzyme interacts with the enzyme that made it or another enzyme upstream in the pathway and inhibits enzyme activity
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* Version of allosteric and non competitive * Could affect any of these enzymes
- Regulation by Covalent Modification * Addition or removal of specific chemical groups * Phosphorylation * Phosphate group from ATP * Usually activates but sometimes inactivates
- Proteolytic Cleavage * Enzymes start as inactive (precursors) when going into small intestine * Enteropeptidase comes in and chops some of the inactive enzyme off -- causing activation * Now activated trypsin turns on the other precursors by cutting off parts of the enzyme \n \n \n \n \n \n \n
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