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Abnormal Rhythms
Introduction
The focus of this section is on Arrhythmia (Abnormal Rhythm) which is synonymous with Dysrhythmia (bad rhythm).
Arrhythmia Overview
Definition:
- Arrhythmia means 'without rhythm'.
- Dysrhythmia refers to a 'bad rhythm', which is equivalent to arrhythmia.
Types of Arrhythmia
Common Types:
- Premature Beats
- Paroxysmal Tachycardia
- Supraventricular Arrhythmias
- Ventricular Arrhythmias
Rate of Tachy-Arrhythmia
Definitions of Tachycardia:
- Tachycardia: 150-250 beats/min
- Flutter: 250-350 beats/min
- Fibrillation: > 350 beats/min
Premature Beats
Definition:
- Premature beats are early beats that interrupt the underlying rhythm by occurring earlier than expected.Types of Premature Beats:
1. PACs (Premature Atrial Contractions)
2. PJCs (Premature Junctional Contractions)
3. PVCs (Premature Ventricular Contractions)
Premature Atrial Contractions (PACs)
Characteristics:
- Early beats arising from areas in the atria other than the SA node, resulting in a different shape of the P Wave.
- Conducted with a narrow QRS complex followed by a pause.Etiologies:
- Increased sympathetic input, exogenous stimulants, drug interactions, acute myocardial infarction (AMI), cardiac ischemia, idiopathic causes.
Premature Junctional Contractions (PJCs)
Characteristics:
- Early beats stemming from the AV node or junction.
- Often conducted with a narrow QRS complex.Common Etiologies:
- Ischemia, hypoxemia, valvular disease, digitalis toxicity, or normal variant conditions.
Premature Ventricular Contractions (PVCs)
Characteristics:
- Early beats arising from the ventricles; display wide QRS complexes (>0.12 sec) that look different from normal sinus-conducted beats.
- PVCs can occur as isolated events or may appear in patterns (pairs, triplets, repeating sequences).Clinical Significance:
- More than 6 PVCs/min suggests pathological conditions.
- Three or more PVCs consecutively is termed a run of Ventricular Tachycardia (VT).
- If VT persists for more than 30 seconds, it is classified as Sustained VT.
Comparison: PAC vs PVC
PACs:
- Usually followed by an incomplete compensatory pause because the PAC resets the sinus node timing, leading to the next sinus P wave appearing earlier than expected.PVCs:
- Typically followed by a complete compensatory pause since the sinus node timing is not interrupted; the subsequent sinus impulse aligns with the sinus rate.
PVC Patterns
Couplets and Triplets:
- PVCs can appear as couplets (two consecutive PVCs) or triplets (three consecutive PVCs).Ventricular Bigeminy: PVCs occurring in patterns such that each normal complex is followed by a PVC.
Trigeminy and Quadrigeminy PVCs
Definitions:
- Bigeminy: Occurrence of a PVC every other beat.
- Trigeminy: Occurrence of a PVC every third beat.
- Quadrigeminy: Occurrence of a PVC every fourth beat.Clinical Implications:
- Increased frequency of PVCs in patients with heart disease is predictive of ventricular fibrillation and sudden death.Causes of PVCs:
- Electrolyte imbalance, hypoxia, ischemia, acute myocardial infarction, and medication toxicity.
Runs of PVCs
Clinical Significance:
- Presentation of runs indicates increased irritability within the ventricles, potentially leading to more severe arrhythmias.
Multifocal PVCs
Definition:
- PVCs that show different shapes and sizes, indicating multiple ectopic foci in the ventricles.
R-on-T Phenomenon
Definition:
- A phenomenon where a PVC occurs superimposing on the T wave of the previous beat's repolarization.
- PVCs can occur early, after the T wave, or late in the cycle, occasionally resulting in a fusion beat.Clinical Risk:
- Particularly hazardous in acute ischemic conditions, as they heighten the risk of progression to ventricular tachycardia or fibrillation.
Summary of Various Forms of PVCs
Forms Covered:
- Multifocal PVCs
- Couplet
- R-on-T phenomenon
- Ventricular tachycardia
- Ventricular bigeminy
- Ventricular trigeminy
Paroxysmal Tachycardia
Definitions:
- Paroxysmal Atrial Tachycardia
- Paroxysmal Junctional Tachycardia
- Paroxysmal Ventricular Tachycardia
Mechanisms of Arrhythmia
Four Mechanisms Identified:
1. Reentry (most common)
2. Automaticity
3. Triggered Activity
4. Parasystole
Detailed Mechanisms of Arrhythmias
Reentry Mechanism:
- Illustrated by the analogy of a dog chasing its tail; involves abnormal conduction patterns or scar tissue that doesn’t conduct.
- Episodes of arrhythmias due to reentrant circuits often start and end suddenly.
AV Node Re-Entrant Tachycardia
Pathway Details:
- The fast pathway in the AV node conducts rapidly but has a longer recovery time.
- The slow pathway conducts slowly but recovers quickly to initiate further beats.Mechanism: PAC blocks in the fast pathway, conducts down the slow pathway, and then retrogrades back up the fast pathway.
Arrhythmias in Clinical Context
Common Arrhythmias:
- Ventricular Tachycardia
- Atrial Tachycardia
- Atrial Fibrillation
- Atrial Flutter
- AV Reentry (such as in Wolff-Parkinson-White syndrome)
Paroxysmal Atrial Tachycardia (PAT)
Definition:
- An event characterized by a sudden start and stop of tachycardia often occurring at 160-220 beats/min.
- Result of reentrant activation fronts within the atria.Causes:
- Anxiety, stimulants (e.g., epinephrine), hypoxia, hyperthyroidism, menstrual onset in women.
Ectopic Atrial Tachycardia
Definition:
- Identified by a dissimilar P wave compared to previous ECG traces, indicating an ectopic focus other than the SA node.Paroxysmal Nature: Sudden and unpredictable outbursts of tachycardia.
Paroxysmal Junctional Tachycardia
Characteristics:
- Fast and slow pathways within the AV junction create a reentrant rhythm exceeding 150 BPM, stimulating normal conduction pathways of the ventricles.
- Regular R-R intervals, normal QRS complexes, and often inverted P waves buried within the QRS or following it.
Conclusion
Significance: Understanding of arrhythmias is essential for proper diagnosis and management, as well as recognizing their clinical implications in various contexts, including pre-existing heart conditions.