DNA Translation
Learning Outcomes
Describe genetic code context (codons and amino acids).
Stages of translation: initiation, elongation, termination.
Effects of phosphorylation on protein activity, function, localization.
Importance of regulating protein levels and forms of post-translational modification.
Key Steps in Information Processing
Transcription: DNA to mRNA.
Translation: mRNA to protein (amino acids).
Translation Components
mRNA: Template encoding protein information.
tRNA: Carrier, brings amino acids to mRNA.
rRNA: Structural and functional roles in ribosome.
Ribosomal proteins.
Release/termination factors.
Genetic Code and Codons
Codon: 3 nucleotides coding for 1 amino acid.
64 possible codons from 4 nucleotides.
AUG codon initiates protein synthesis coding for methionine.
Open Reading Frame (ORF)
Continuous codon sequence between initiation and termination codons.
Characteristics of Genetic Code
Specific, universal, redundant, and non-overlapping.
Mutations and Disease Examples
Codon amplification may lead to diseases (e.g., Huntington's).
Sickle Cell Anemia from a missense mutation in the β-globin gene.
Transfer RNA (tRNA)
Cloverleaf structure with anticodon for mRNA.
Wobble pairing allows flexibility in matching codons.
Ribosome Structure and Function
Sites: A (Aminoacyl), P (Peptidyl), E (Exit).
Ribosome assembles tRNA and mRNA for amino acid translation.
Process of Translation
Initiation
Assembly of ribosomal units, mRNA, and tRNA.
Recognition of start codon by tRNA (AUG).
Elongation
Aminoacyl-tRNA arrives at A site.
Peptide bond formation between amino acids.
Ribosome translocates, moving the chain.
Termination
Stop codon in A site triggers release factor, disassembling complex.
Dynamic Regulation of Protein Activity
Influenced by transcription/translation rates, degradation, and localization.
Protein Degradation Mechanisms
Lysosomal: For imported proteins.
Proteasomal: For cytosolic proteins marked by polyubiquitination.
Post-Translational Modifications (PTMs)
Include phosphorylation, ligand binding, cleavage, ubiquitination.
Phosphorylation Mechanism
Adds phosphate to serine, threonine, tyrosine residues.
Alters protein localization, function, and degradation markers.
Kinases as Therapeutic Targets
Example: Gleevec for Chronic Myelogenous Leukemia targeting BCR-ABL fusion kinase.