43 - Patella Luxation
general
genetic → heritable
developmental disease → not congenital
65% are bilateral
80% are medial luxation
small breed
toy and mini breeds
medial luxation
large breed
lateral luxation → more complex to tx
increasing in prevalence
anatomy
patella is a sesamoid bone embedded within the patellar tendon
musculature → quadriceps insert on patella and continue as patellar ligament to insert on tibial tuberosity
normal patellar mechanism
provides smooth surface for tendon to glide over moving joint
acts like pulley mechanism → changes the direction of force + magnifies force with less effort
must be properly aligned for normal function → femoral shaft, trochlear groove, and tibial tuberosity
medial patellar luxation
luxation = malalignment of quadriceps mechanism
one of the most common rear limb lamenesses
often bilateral
MPL = higher incidence of concurrent CCLR
small breeds
usually initially detected in young dogs without CCLR
can be detected in older dogs with CCLR
no hx of trauma
“skipping” hindlimb lameness
NWB lamness can indicate MPL + CCLR
femoral malformations
coxa vara
decreased angle of femoral head
femur pulled up and tilted, may cause extensor muscle laxity
decreased angle of anteversion
femoral head/neck are tilted
limb rotates internally → knee and foot twist toward midline body
distal external torsion
tendon pulled out of alignment
MPL = external rotations
LPL = internal rotation
distal varus
rotated femur
hypoplasia of medial condyle
femur leans on malformed condyle
secondary to luxated patella during growth → no pressure on trochlear groove
tibial malformations
medial displacement of tibial tuberosity
most common
internal proximal tibial torsion
proximal tibial valgus
distal varus
less-common causes
secondary to trauma
patellar alta → patella sits too proximally in groove
tightness or atrophy of quadriceps muscles
exam findings
normal to decreased muscle mass in HL
normal to decreased weight bearing in HL
unilateral or bilateral skipping gait
intermittent lamness
dogs may limp then extend leg
discomfort with palpation and ROM
may feel grinding as patella moves
stifle effusion
crepitus with ROM
check for tibial thrust and/or cranial drawer
grading
increased grade associated with more severe femoral/tibial deformities
grade I
minimal malaignment
can be luxated by force but automatically return to placement
asyptomatic → often incidental finding
no treatment necessary
grade II
intermittent luxation
patella luxates easily on palpation but still wants to stay in groove
spends more time in place than out
surgery indicated in clinical signs present or are progressive
Grade III
patella permanently luxated but can be manually reduced → reluxates when released
significant lameness and gait abnormalities
surgery is indicated
Grade IV
may need to sedate to grade
permanent luxation and cannot be reduced manually
trochlear groove absent or severely eroded trochlear ridges
severe gait abnormalities
often has concurrent CCLRs
surgery indicated → guarded prognosis
surgical repair
assess trochlear groove
assess tibial tuberosity
case selection
small dogs with minimal to moderate limb deformities
large dogs with minimal to no major limb deformities
trochleoplasty
trochlear block recession
preserves more cartilage surface area
trochlear wedge resection
easy to perform with hand saw
preserves hyaline articular cartilage
deepens trochlear groove
shapes: wedge or block
abrasion sulcoplasty
removes cartilage and replaces with fibrocartilage → poorer overall quality
tibial crest transposition
re-aligns quadriceps by shifting insertion of patellar ligament
risks
may create LPL
implant failure
tibial tuberosity avulsion
fracture
soft-tissue aspects
releasing incison
anti-rotation suture if concurrent CCLR
retinacular imbrication
corrects retinacular laxity
excessive tension predisposes luxation in opposite direction → goal is equal tension medial and lateral
opposite side as direction of luxation
performed as final step of luxation correction