M4 Lead Identification and Cell-Based Assays
Lead Identification
Learning Objectives
Develop a cell-based assay based on the identified disease target and molecular target.
Examine features of cell-based assays.
Utilize cell-based assays to screen hits and convert them into leads (HITs to leads).
From Hits to Leads
The previous module detailed techniques for measuring interactions between small molecules and molecular targets.
Molecules that interact with the target are called "hits."
Hits vary in their strength of interaction with the target.
Screening 50,000 compounds might yield 0.1% positives (compounds that interact with the molecular target).
This could result in about 500 compounds to screen, which is still a large number.
Reducing This Number:
Categorizing compounds into classes.
Screening those with binding strength above a certain threshold.
Developing a Cell-Based Assay
When developing a bioassay:
It should be cell-based.
It should be simple to perform (few steps).
It should be based on a measurable biological activity of the target molecule.
Critically, it should be able to be miniaturized.
Cystic Fibrosis Example
Target disease: Cystic Fibrosis (CF)
Target molecule: Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) protein, a chloride channel.
CFTR Mutations:
Class 1: No protein synthesized.
Class 2: Truncated protein synthesized.
Class 3: Protein synthesized with mutation, stuck in the ER.
Class 4 & 5: Other types of mutations
Class II mutations are found in nearly 80% of CF patients.
Details of Class II Mutation (Delta F508)
CFTR is made in the ER.
There is a deletion at residue number 508 (delta F508 - deletion of phenylalanine at position 508).
This deletion causes the protein to misfold and be retained in the ER.
Only 5% of the protein reaches the plasma membrane.
Crucially, the 5% that reaches the membrane is fully functional.
Developing a Cell-Based Assay for CFTR
Start with messenger RNA (mRNA).
Use RT-PCR to clone the wild-type and delta F508 forms of the protein.
Clone these into mammalian expression vectors.
Transfect these vectors into epithelial cell lines.
Plate cells expressing either wild-type or delta F508 CFTR in a 96-well plate.
Measure chloride flux across the membrane in the presence or absence of identified hits.
Hits that stimulate CFTR activity become leads.
Stimulating CFTR activity implies promoting trafficking of CFTR from the ER to the membrane.