PTM
Proteolytic cleavage
Carried out by proteases
Irreversible
Changes the tertiary/quaternary protein structure
Can activate/deactivate proteins depending on the protein
Example: protease activation - chymotrypsinogen and chymotrypsin
Zymogen is inactive when first secreted by the pancreas, this prevents premature digestion, for example prevents digestion of body proteins required for function
Inactive precursor is chymotrypsinogen
Activated by proteolytic cleavage. Produces 3 polypeptide chains and these are held together by disulphide bridges. The rearrangement and production of a quaternary structure orientates the catalytic triad correctly in the active site enabling nucleophilic attack and protein digestion
Lipidation
Attachment of hydrophobic groups/fatty acid chains to a protein
Allows membrane localisation
Changes how the molecule interacts with the membrane and other molecules, changes properties by introducing a hydrophobic group
Example: prenylation
Attachment of isoprene (conjugated 2 double bonds) containing polymers to cysteine residues of proteins
Farnesylation - 15C added
Geranylgeranylation - 20C added
Can be removed, the protein will no longer be anchored to the membrane, important in the regulation of protein localisation in the membrane due to its reversibility
Important in signalling
Example: myristylation
Irreversible attachment of a 14C fatty acid chain to the amino terminus of protein - generally a glycine residue
Irreversible
Protein targeting to membranes
Example: palmitoylation
Reversible attachment of fatty acid chain to cysteine residue
Localises proteins to the cytoplasmic leaflet of the plasma membrane
Example: GPI anchor
Protein attached through C terminus to carbohydrate groups and phosphatidylinositol group with various fatty acid groups
Common amongst adhesion molecules and membrane receptors
Helps membrane loacalisation
Phosphorylation
Addition of the terminal phsphate from ATP to a molecule
Covalent attachment
Very stable PTM
Done by kinases, specific, serine, threonine or tyrosine kinases, attach onto the OH of a side chain
Addition adds a large negative charge onto the molecule, this can induce large conformational changes on the protein which can be activating or deactivating to a protein depedning on what they change
Can produce binding sites for other proteins - such as SH2 domain proteins that can recognise the phosphprylated state of the protein due to the altered charge and electrostatic potential
Removed by phosphatatses, hydrolyse the covalent bond and revert the protein back to its original structure
Involved in protein kinase cascades
Example: Signalling and MAPK pathways
Ligand binfing to a RTK causes dimerisation of the intracellular domains, this causes transphosphorylation to occur
This produces a binding site for a SH2 domain proteins
Phsphprylates these and activats further downstream pathways
Example: phosphorylation of a transcirption factor activates it and allows it to bind to DNA asnd cause transcription of mRNA for a specific protein - eg enzyme for metabolism
Acetylation/deacetylation
Addition of acetly group to a protein, more specifically on a lysine residue
Alters the amino acids around it
Associated with histones, alters the reading of DNA
Done by acetyltransferases and reversed by deacetylases
Example: histone acetylation
Addition of acetyl group onto lysine residue causes the conversion of heterochromatin to euchromatin
Addition removes the +ve charge on lysine, therefore less attracted to the negatively charged DNA backbone, less attraction means increased accessibility by RNA polymerase for transcription
Methylation
Addtion of a methyl group, commonly onto arginine and lysine, can be signle, double or triple
Mehtylation remains the +ve charge on lysine
Importsnt for tranxriptional repression or activation
Example: mehtylation for repression
Produces binding sites for proteins that induce compaction of genes
Changes the conformation of the transcription factor binding site, reducing transcription and therefore expression