Drug Therapy for Coronary Heart Disease
Drug Therapy for Coronary Heart Disease
Overview of Coronary Heart Disease
Clinical syndrome characterized by episodes of chest pain
Occurs with myocardial oxygen (O2) deficit
Produces myocardial ischemia
Causes of coronary heart disease:
Atherosclerotic plaque in coronary arteries
Coronary vasospasm
Development of Coronary Artery Disease (CAD)
Atherosclerotic plaque development and progression
Represents a continuum that progresses from angina to myocardial infarction (MI)
Three main types of angina:
Classic angina
Typically occurs with exertion or stress
Variant angina (also known as Prinzmetal’s or vasospastic angina)
Caused by coronary artery spasm
Unstable angina
Occurs at rest or with minimal exertion and is associated with worsening symptoms
Angina Pectoris
Definition: Angina pectoris is a clinical syndrome characterized by episodes of chest pain due to a myocardial oxygen supply deficit relative to demand.
Classic Anginal Pain
Characteristics:
Substernal chest pain that is:
Constricting
Squeezing
Suffocating
Pain may radiate to:
Jaw
Neck
Shoulder
Both arms
Back
Symptoms may be confused with:
Arthritis
Indigestion
Associated symptoms:
Nausea and vomiting (N/V)
Dizziness
Diaphoresis
Shortness of breath (SOB)
Fear of impending doom
Gender differences exist in symptom quality:
Women may report different symptoms, such as epigastric or back discomfort.
True or False Questions
Angina pectoris is a clinical syndrome characterized by episodes of chest pain.
Answer: True
Rationale: Angina pectoris occurs due to an imbalance between myocardial oxygen supply and demand.
Cardiac symptoms are pretty much the same in everyone.
Answer: False
Rationale: Symptoms differ according to gender; women may have varied symptomatology.
Overlapping Factors in CAD
Coronary atherosclerosis
Myocardial ischemia
Associated with acute coronary syndrome (ACS)
Resultant cardiovascular impairments
Nonpharmacologic Management of CAD
Risk factor modification:
Implement lifestyle changes and medications
Address obesity
Smoking cessation
Manage elevated triglycerides and cholesterol
Control elevated blood pressure and fasting glucose
Patient education regarding the management of CAD
Revascularization and interventional procedures for advanced cases
True or False Question
Optimal management of CAD involves lifestyle changes and medications.
Answer: True
Rationale: Management of CAD, irrespective of MI symptoms, requires changes in lifestyle and medications when necessary.
Pharmacologic Management of CAD
Classes of drugs used:
Organic nitrates (e.g., nitroglycerin)
Beta-adrenergic blocking agents (e.g., atenolol)
Calcium channel blocking agents (e.g., nifedipine):
Actions:
Reduce myocardial oxygen demand
Increase myocardial blood supply
Metabolic modulators (e.g., ranolazine):
Function to preserve cardiac function without decreasing heart rate or blood pressure (BP)
Adjunctive Antianginal Drugs
Drugs with antithrombotic effects:
Aspirin
Adenosine diphosphate receptor antagonists
Glycoprotein IIb/IIIa receptor antagonists
Antilipidemics:
Reduce serum cholesterol
Antihypertensives:
Reduce blood pressure
Angiotensin-converting enzyme inhibitors:
Cause vasodilation
Morphine:
Relieves pain, reduces anxiety, decreases preload
Thrombolytic drugs:
Dissolve thrombi and restore blood flow
Goals of Therapy for CAD
Relieve acute anginal pain
Reduce the number and severity of acute anginal attacks
Improve exercise tolerance and quality of life
Delay progression of CAD
Prevent myocardial infarction and sudden cardiac death
Special Populations in Antianginal Drug Use
Considerations for:
Children
Older adults
Individuals with renal impairment
Individuals with hepatic impairment
Patients in critical illness
Home care settings
Nursing Implications
Key nursing responsibilities:
Preventing drug interactions
Administering medications
Assessing for therapeutic effects
Assessing for adverse effects
Patient teaching related to CAD and its management.