Module 3 – Personality & Eating Disorders Study Notes
Conceptual Frameworks & Influencing Factors
- Functional Ability themes underlying course content:
- Homeostasis & Regulation (Fluid/Electrolytes, Thermoregulation, Cellular, Hormonal, Glucose, Gas Exchange, Perfusion, Clotting)
- Protection & Movement (Immunity, Infection, Tissue Integrity, Sensory Perception, Mobility, Resilience, Stress & Coping, Nutrition)
- Maladaptive Behaviour (Family Dynamics, Mood & Cognition, Mood & Affect, Anxiety, Cognition, Psychosis, Sexuality & Reproduction, Addiction)
- Systems-level concepts for professional nursing practice:
- Patient Education, Health Promotion & Self-Management
- Health Disparities, Adherence, Care Coordination & Collaboration
Personality Disorders (PD) – Overview
- Definition: Personality traits become so exaggerated/rigid that they create functional impairment or distress, particularly in relationships.
- Prevalence & co-morbidity:
- >50% of all patients presenting for medical care may have a PD.
- In psychiatric populations, 30–50% have a co-occurring PD.
- Associated with emotional, social & occupational disability.
- Onset & etiology:
- Traits visible from infancy ➔ disorder usually emerges in adolescence.
- Multifactorial: genetic (twin studies show stronger similarity in identical twins), neurobiological (impulsivity, affective instability), psychological (childhood neglect/trauma, harsh discipline, chaotic or sexually abusive homes).
General Clinical Features of PD
- Core impairments: identity, self-direction, empathy, intimacy.
- Hallmarks:
- Fear of rejection, avoidance OR fusion & boundary blurring.
- Demanding, fault-finding, manipulative, passive-aggressive, distrusting.
- Limited insight—patients see others as “the problem.”
- Relationships often volatile; potential for self/other-directed violence.
- Suffering, under-achievement, social isolation, bizarre/anxious/withdrawn behaviour.
Symptom Clusters
- Cluster A (“odd/eccentric”): avoidant of relationships, unusual beliefs.
- Cluster B (“dramatic/manipulative”): emotional reactivity, poor impulse control, unclear identity.
- Cluster C (“anxious/fearful”): high anxiety, self-blame, inhibition.
Cluster-Specific Disorders & Key Characteristics
- Cluster A
- Schizotypal PD – appears like schizophrenia without frank psychosis.
- Paranoid PD – pervasive unjustified suspiciousness/hostility.
- Schizoid PD – flat affect, indifferent to praise/criticism, detached relationships.
- Cluster B
- Antisocial PD – persistent disregard/violation of others’ rights.
- Borderline PD – unstable intense relationships, affective lability, splitting (good vs. bad).
- Narcissistic PD – grandiosity, entitlement, lack of empathy.
- Histrionic PD – excessive emotion & attention seeking, must be centre stage.
- Cluster C
- Avoidant PD – feelings of inadequacy, hypersensitive to criticism.
- Obsessive-Compulsive PD – perfectionism, orderliness, indecisiveness, emotional constriction.
- Dependent PD – excessive need for care, fears of being alone.
Passive-Aggressive Personality Trait
- Chronic irritability, blame-shifting, verbal aggression, negativism, obstructionism.
Primitive Defences in PD (esp. BPD & Antisocial)
- Used to control inner chaos due to “ego weakness.”
- Defences: splitting, dissociation, psychotic denial, projective identification, primitive idealisation, omnipotence/devaluation.
- Manifestations: unmodulated rage, envy, shame ➔ attacking, clinging, lying, impulsivity, identity diffusion, irrationality.
Assessment Guidelines for PD
- Always assess for suicidal/homicidal ideation.
- Rule out medical/substance causes.
- Consider cultural/ethnic context.
- Look for personality change in mid-life (possible substance use disorder).
- Anticipate strong counter-transference (nurse’s negative emotions).
Expected Outcomes (Realistic & Modest)
- Minimise self-destructive acts.
- Reduce manipulation; link behaviour to consequences.
- Teach alternatives to crisis; sustain emotional management.
- Foster a regression-preventing lifestyle.
Communication & Nursing Strategies
- Core tools: limit-setting, consistency, authenticity, trustworthiness.
- Teach patients to:
- Identify triggers & cues preceding impulsive acts.
- Explore impact on self/others; develop alternative responses.
- Practise coping skills (anger management, assertiveness).
- Avoid: secret-keeping, gift acceptance, discussing staff personal info, special favours.
Managing Manipulative or Impulsive Behaviour
- Brief observation before labelling “manipulative.”
- Clear boundaries & documented consequences; enforce consistently.
- Examples of manipulative tactics: arguing, flattery, pitting staff, power struggles.
Milieu & Group Approaches
- Community meetings, problem-solving, coping skills & socialisation groups.
- Desensitisation through safe group experience.
Therapeutic Modalities
- Therapeutic relationship: difficult d/t suspicion/hostility; must understand complaints arise from fear/threat perceptions.
- STEPPS (Systems Training for Emotional Predictability & Problem-Solving):
- 20-week manualised adjunct for BPD.
- Lowers core symptom intensity; ↓ ED visits & suicide attempts (no change in hospital utilisation/suicidal ideation frequency).
- Dialectical Behaviour Therapy (DBT) – core for BPD: mindfulness, distress tolerance, emotion regulation, interpersonal effectiveness.
Pharmacologic Symptom Management in PD
- No medication cures PD; target symptoms with low-toxicity drugs.
- Avoid long-term benzodiazepines (abuse/OD risk).
- Options:
- SSRIs – depression & panic; ↓ aggression when combined with lithium/anticonvulsants.
- SARI (Trazodone) & SNRI (Venlafaxine) – safer in overdose.
- Carbamazepine – impulsivity/self-harm.
- Atypical antipsychotics – transient psychosis under stress.
Neurobiology of Borderline PD & Emotional Dysregulation
- Altered serotonin (5-HTT short alleles) ➔ ↓ serotonin, ↑ impulsive aggression.
- Functional MRI: prefrontal cortex hypo-activity (poor regulation), hyper-reactive amygdala/limbic system (prolonged fight-or-flight).
- Elevated norepinephrine & dopamine during stress.
- Treatments: DBT (shifts from sympathetic → parasympathetic), SSRIs, anticonvulsants, SGAs, lithium dampen dysregulation.
Case Study – “Gale”
- 58-year-old professor; Dx: Depression & BPD.
- Incident: verbal aggression toward faculty, threw books, assaulted staff ➔ crisis unit admission.
- Behaviour on unit: yelling, threw hot coffee; required seclusion & medication.
- Audience-response pearls:
- Best initial nurse intervention: Introduce self & establish working relationship (Option D) – builds trust before limits.
- When splitting another nurse as “good” vs. primary nurse “cold” ➔ respond with reflecting feelings without reinforcing split: “You seem concerned about how you’re being treated…” (Option A).
- Day 4 violent outburst, refusing PO meds ➔ likely need IM Haloperidol + IM anxiolytic (Option B) for immediate control.
Self-Care for Nurses Working with PD
- Common feelings: frustration, anger, helplessness, being “manipulated,” fear of violence, exhaustion.
- Essential to debrief, set limits, recognise counter-transference, seek supervision.
Eating Disorders – Overview
- Unified feature: irrational misperception of body image.
- DSM-5 categories: Anorexia Nervosa, Bulimia Nervosa, Binge-Eating Disorder (BED).
- High co-morbidity with mood/anxiety disorders, substance abuse, body dysmorphic disorder, impulse control & PDs (esp. BPD & OCPD).
- AED guideline: always assess for suicide/self-harm risk.
Hospitalisation Criteria for ED
- Weight loss >30% in 6 months.
- Hypothermia, dehydration, loss of sub-Q fat.
- Bradycardia <40\text{ bpm}; Systolic BP <70\text{ mmHg}.
- Hypokalaemia <3\text{ mEq/L} unresponsive to PO supplement.
- Psychiatric: suicidal, severe depression, psychosis, family crisis, uncontrolled purging/abuse of laxatives/diuretics/emetics/street drugs.
Anorexia Nervosa
- Clinical signs
- Terror of weight gain, food pre-occupation, cutting food into tiny bits.
- Possible vigorous exercise, vomiting, laxatives/diuretics.
- Severely underweight (cachexia), lanugo hair, amenorrhoea.
- CV: bradycardia, murmurs, ECG changes ➔ sudden MI.
- Hypotension (orthostatic), blood dyscrasias, liver changes, elevated cholesterol, thyroid abnormalities, proteinuria/haematuria.
- Nursing Diagnoses: imbalanced nutrition < requirements, ↓ cardiac output, risk for electrolyte injury, fluid volume imbalance, distorted body image, anxiety, low self-esteem, ineffective coping, hopelessness.
- Interventions
- Acute phase (ICU/ED/CCU): build trust, strict weight & intake monitoring, milieu therapy, link privileges to compliance.
- Long-term: combo of individual/group/family therapy; periodic brief re-hospitalisations.
- Cognitive distortions to address: over-generalisation, all-or-nothing, catastrophising, personalisation, emotional reasoning.
- Example of all-or-nothing: “If I allow myself to gain weight, I’ll be huge.”
- Medications: limited evidence; Olanzapine may ↑ weight & improve body image; Fluoxetine may aid maintenance/prevent relapse.
Bulimia Nervosa
- Clinical signs
- Recurrent bingeing with compensatory purging (vomiting, laxatives, diuretics).
- History of anorexia common (¼–⅓).
- Depression, interpersonal issues, impulsivity, possible substance abuse.
- Physical: parotid enlargement, dental erosion, esophageal tears, Russell’s sign (callused knuckles), ECG changes, metabolic alkalosis, hypokalaemia, dehydration.
- Nursing Diagnoses: ↓ cardiac output, disturbed body image, powerlessness, chronic low self-esteem, anxiety, ineffective coping.
- Interventions
- Acute: inpatient CBT, interrupt binge–purge cycle, normalise eating, treat co-morbidities.
- Long-term: ongoing therapy, connect with resources (www.anred.com), family involvement.
- Patient more able to form therapeutic alliance (behaviour is ego-dystonic).
- Audience Q: Dehydration from vomiting most likely causes hypokalaemia (Option B).
Binge-Eating Disorder (BED)
- Variant of compulsive overeating; now discrete DSM-5 diagnosis.
- Episodes of large food intake in short time with loss of control; followed by guilt, disgust, depression.
- Often linked with obesity & depression.
- Long-term outpatient focus: healthy weight maintenance, individual/family/group therapy, pharmacology, nutrition counselling; treat co-morbid depression, substance use, PDs.
- Teach constructive coping, relaxation, social & problem-solving skills.
- Meal planning, healthy diet/exercise, effects of binging/purging, cognitive distortions.
- Supervised food shopping, eating out, “forbidden food” exposure.
- Discharge: living, school, work, finances, outpatient follow-up.
Team-Based Care for ED
- Core team: Nurse, Psychiatrist, LMHC, Dietician, Internist/Pediatrician, Psychologist, Social Worker, Mental Health Tech.
- Family, social network & support groups integral.
Medication Notes in ED
- No agent universally efficacious for EDs.
- Weight gain/appetite improve when underlying anxiety is treated.
- Olanzapine: weight gain, cognitive/body-image benefit.
- Fluoxetine: mixed results in maintenance/relapse prevention.
Milieu, Health Teaching & Psychotherapy Goals (ED)
- Interrupt pathological eating behaviours, normalise patterns, teach relaxation & coping, correct distortions, discharge planning.
Quick Reference to Audience Response Answers
- Gale – best initial intervention ➔ Introduce self / establish nurse relationship (D).
- Gale splitting staff ➔ Reflect concern (A).
- STEPPS applicability ➔ Borderline PD (C).
- Gale violent outburst med ➔ IM Haloperidol + anxiolytic (B).
- All-or-nothing cognition ➔ “If I allow myself to gain weight, I’ll be huge.” (A).
- Bulimic dehydration complication ➔ Hypokalaemia (B).
- Therapeutic alliance blocker in anorexia ➔ Authoritarian disciplined eating approach (A) is least helpful.