Cardiac Notes
Blood Flow Through the Heart
Blood flows from the body to the right atrium, then to the right ventricle, and then to the lungs for oxygenation.
Oxygenated blood returns from the lungs to the left atrium, then to the left ventricle, and finally out to the body.
Heart Valves
Mitral Valve: Located between the left atrium and left ventricle.
Tricuspid Valve: Located between the right atrium and right ventricle.
Pulmonic Valve: (Semilunar) Located between the right ventricle and pulmonary artery.
Aortic Valve: (Semilunar) Located between the left ventricle and aorta.
Chordae Tendineae: Tendon-like cords that attach the mitral and tricuspid valves to the papillary muscles.
Papillary Muscles: Located in the ventricles; they contract to prevent the valves from inverting during ventricular contraction.
Conduction System
SA Node: Sinoatrial node; the heart's natural pacemaker.
AV Node: Atrioventricular node; delays the electrical impulse to allow the atria to contract before the ventricles.
Bundle of His: Conducts the electrical impulse from the AV node to the bundle branches.
Left Bundle Branch: Divides into the anterior and posterior fascicles to conduct impulses to the left ventricle.
Right Bundle Branch: Conducts impulses to the right ventricle.
Purkinje Fibers: Distribute the electrical impulse throughout the ventricles, causing them to contract.
History and Physical Exam of the Adult
History of Current Complaint:
Chest pain: Onset, location, duration, character, aggravating/alleviating factors.
Irregularities of heart rhythm (palpitations).
Respiratory manifestations: Dyspnea (shortness of breath), especially exertional dyspnea and orthopnea (difficulty breathing when lying down).
Paroxysmal nocturnal dyspnea (sudden, severe shortness of breath at night).
Fatigue or dizziness.
Past Health History:
Major illnesses and cardiac surgical history.
Family heart history/childhood & infectious diseases.
Psychosocial history.
Medications & Allergies:
Cardiac medications, diuretics, aspirin, and chewable antacids.
Risk Factors:
Dietary/nutrition habits.
Exercise and social habits.
Occupation, geographic location, and environment.
Cardiac Assessment: Physical Signs
Skin Color: Dusky skin, pallor (pale skin), or cyanosis (bluish skin).
Syncope, Dizziness, Vertigo: Fainting, lightheadedness, or a sensation of spinning.
Generalized Edema: Swelling throughout the body.
Weight Gain and Dependent, Pitting Edema: Swelling in the legs, feet, or arms that leaves a pit when pressed.
Respiratory Distress: Difficulty breathing.
Focused Cardiac Assessment
Blood Pressure: Measured bilaterally in both arms.
Orthostatic VS: Blood pressure and heart rate measurements taken in different positions (lying, sitting, standing) to assess for changes.
Pulse Pressure: Calculated as . Assess pulse rhythm and force.
Pulse Deficit: Difference between the apical pulse and the peripheral pulse; indicates a dysrhythmia.
Respirations: Assessment includes rate, rhythm, depth, and quality.
Heart Sounds - Location
S1 and S2:
S2 Loudest: Base of the heart (Aortic & Pulmonic areas).
S1 & S2 Sound Equally: Erb's Point
S1 Loudest: Apex of the Heart (Mitral & Tricuspid areas).
Normal Heart Sounds: S1 & S2
S1 = "lub"
Tricuspid and Mitral valves close nearly simultaneously.
Best heard at the apex of the heart.
Marks the beginning of systole (ventricular contraction).
S2 = "dub"
Aortic and Pulmonic valves close nearly simultaneously.
Best heard at the base of the heart.
Marks the beginning of diastole (ventricular relaxation).
Heart Sound: S3: Ventricular Gallop
Turbulence of blood going into ventricles (passive filling, not a contraction).
Low-intensity vibration of ventricle walls.
May be normal in young adults.
Common with left ventricular heart failure or mitral valve regurgitation.
Abnormal Heart Sounds
S3 – Ventricular Gallop: Early diastolic filling of the left ventricle.
S4 – Atrial Gallop: Left atrial contraction filling into stiff ventricles.
Pericardial Friction Rub: Indicates inflamed pericardium.
Murmurs: Indicate ineffective valves; can be systolic or diastolic.
Heart Sounds: S4—Atrial Gallop
Heard with atrial contraction if there is resistance to ventricular filling (active filling).
Decreased ventricular compliance.
Common in patients with CAD (coronary artery disease), cardiomyopathy, left ventricular hypertrophy, or aortic stenosis.
May be heard in the elderly, but is always pathological.
Major Purpose of the Heart Valves
Prevent the backward flow of blood
Pericardial Friction Rub
Caused by pericarditis (inflammation of the pericardium).
Best heard at the apex of the heart (apical).
Associated with chest pain and shortness of breath (SOB).
Managed with EKG, NSAIDs, ASA, and drainage.
Murmurs in Adults
Abnormal sound in the heart or neighboring large blood vessels.
Heard during systole, diastole, or both.
Graded on a 1-6 point grading system.
Causes: cardiac valve disorder (atherosclerosis, regurgitation) & abnormal blood flow patterns.
Can occur during pregnancy or exercise.
Physical Assessment Head and Neck
Neck Veins: Assess for Jugular Vein Distention (JVD).
Evaluate with the head of the bed (HOB) at 30-45 degrees.
Chest:
Auscultate heart sounds.
Palpate carotid, abdominal aorta, and femoral arteries.
Auscultation: Bruit
Listen over major arteries:
Carotid arteries.
Abdominal aorta.
Listen for a bruit: a blowing sound.
Palpate for a thrill: a vibration.
Causes:
Narrowed vessel
Aneurysm
Physical Assessment - Lungs
Breath sounds: Assess for abnormalities.
Left-sided heart failure: Tachypnea (rapid breathing), crackles, blood-tinged sputum, pulmonary edema.
Right-sided heart failure: Lungs generally not affected, but may have heart palpitations and generalized edema.
Physical Assessment - Abdomen
Inspection:
Distension: ascites (fluid accumulation in the abdomen).
Auscultation:
Aortic bruits/thrills.
Renal bruits/thrills.
Palpation:
Enlarged liver.
Physical Assessment - Edema
Generalized Edema: Swelling throughout the whole body.
Dependent Edema: Swelling confined to dependent areas (e.g., legs, feet).
Pitting Edema: Swelling that leaves a "pit" when pressed.
Scale:
Grade 1+ x 2 = 2mm indentation
Grade 2+ (x 2 mm) = 4mm
Grade 3+ (x2 mm) = 6mm
Grade 4+ (x2 mm) = 8mm
Age-Related Changes
Cardiac Valves:
Thicker & stiffer.
Infections & Vegetation.
Murmurs more common.
Conduction System:
Bradycardia & dysrhythmias.
Seen on EKG: increase in PR and QRS interval.
Left Ventricle:
Increased size.
Stiffer, less contractility.
Decreased response to workload.
Arteries:
Thicker and stiffer (atherosclerosis).
Increased systemic vascular resistance.
Increased sensitivity to ADH (Antidiuretic Hormone) which leads to HTN.
Orthostatic HTN.
History and Physical Exam of the Child
Acute Assessment: Observe the child for signs of distress
Pallor, sweating, cyanosis, or increased work of breathing
Level of activity, do they appear uncomfortable?
Interactions with parents/caregivers
History – General Health
Feeding difficulties
Growth delays
Decreased exercise/play tolerance
Prenatal history (exposure to drugs, lithium, phenytoin, alcohol)
Maternal illness: Diabetes, rubella, systemic lupus erythematosus
Born premature or other family history
Cardiac Assessment
Periods of cyanosis, sweating, SOB, palpitations, and edema
Chest pain and syncope (rare)
Cardiac monitor
Heart Sounds: Congenital displacement
Heart Sounds in children
Move across the chest in smaller increments.
Assess lying, sitting, and standing.
Murmurs are common.
Affects 50% of children
S1 (apex) and S2 (base, 2nd intercostal): normal.
S3: could be innocent.
S4: atrial gallop.
Target apical heart rates by age:
newborn: 100-170
6 months 1 yr 90-130
2-3 years: 80-120
10 years and older: 60-120
Diagnostic Tests
Non-invasive:
EKG
Echocardiogram
Chest X-ray
Holter Monitor
Electrocardiogram: ECG (EKG)
Measures electrical conduction in the heart.
Diagnoses abnormal heart rhythms, heart size, position.
3 Types: 1, 3, and 12 lead
12 Lead used for diagnostic
EKG/ECG
Performed at the bedside by trained nurse/tech
Used to diagnose:
Abnormal heart rhythms
Electrical abnormalities
Narrowed arteries
Effects of medications
Ischemia, injury, infarctions, prior MI
Pacemaker status
Cardiac Action Potential & Automaticity
Electrical Activity—The action potential:
Depolarization
Sodium () moves into the cell and potassium () moves out
Calcium () inside cells causes an increase in cardiac contraction of the heart
Inside of cells becomes more positively charged
S1: Contraction
Repolarization
Potassium () moves back into the cell and sodium () moves back out
Inside of cells moves back to a more negative charge
S2: Relaxation
Spontaneous depolarization
SA Node Automaticity
Cardiac Cycle on ECG
Includes systole, diastole, atrial depolarization, ventricular depolarization, and ventricular repolarization.
Key components: P wave, QRS complex, T wave, and R-R interval.
EKG interpretation
Key components: P wave, QRS complex, T wave.
Measurements:
0.04 sec
0.20 sec
Intervals:
PR interval
QRS interval
QT interval
Cardiac Cycle on EKG/ECG. Intervals and Segments
The image shows the different waves on an EKG to include P wave, QRS complex, and T wave.
It shows the different segments and intervals to include PR segment, PR interval, ST segment, QRS interval, and QT interval.
EKG/ECG
P wave
Atrial depolarization
PR interval
Time for electrical impulse conduction through the atria and the AV node
Duration 0.12 – 0.20 sec
Baseline between P wave and QRS complex is the isoelectric line
QRS complex
Ventricular depolarization
Normal Duration <0.12 sec
Wide QRS = hyperkalemia
EKG/ECG
ST Segment
Normally Isoelectric (no electrical charge)
ST Depression usually hypokalemia
Elevated ST: myocardial ischemia
T wave
Ventricular repolarization
Flattened T wave: hypokalemia
Peaked T wave: hyperkalemia
Inverted T wave: hypokalemia
U wave (if present)
hypokalemia
Hypo/hyperkalemia: changes to the EKG
HYPOkalemia:
P wave = peaked
T wave (flattening, shallow or inverted)
ST-segment depression
U waves = prominent
PR interval = prolonged
HYPERkalemia:
P wave = flattened
QRS = Prolonged or wide
T wave = peaked
PR Interval = Prolonged
ST-Segment Depression
Holter Monitoring
24- 48-hour wearable EKG while performing daily routine
Records arrythmias
Evaluate medications or pacemaker therapy
Remove for showers
Diary Card (must be maintained accurately)
Activity
Symptom
Exact time
Zio Patch monitor
Single Lead
Water resistant
Readings up to 14 days
Captures a variety of cardiac dysrhythmias
Better accuracy than Holter
Echocardiogram
Evaluates:
Heart size & shape
Valve structure & function
Blood flow & pumping function
Ejection fraction
Client Instructions:
Lie Still; No talking
Breath normally
Diagnostic Tests: Invasive Cardiac Catheterization
View of aorta through coronary arteries
Evaluates heart pressures/function
Stent placement
Biopsy
Diagnosis
Cardiac Catheterization
Local anesthetic or sedation given, general in certain cases
Right Heart
Insert through vein (jugular, AC)
Left Heart
Insert through a femoral (most common) brachial, radial, or axillary artery
Coronary Angiography: Cardiac cath with injection of contrast medium
Cardiac Cath: Pre-Op Procedures
Informed consent
Assess allergies. Assess for Shellfish and Iodine Allergies
NPO 6-8 hrs prior
VS
Mark peripheral pulses
Labs:
Creatinine, BUN levels
PT/INR, aPTT
Education
Lie still
Fluttery feeling
Flushing/ warmth
Cough
Palpitations
Remain flat on back for 2-12 hours postprocedure
Cardiac Catheterization: Post-procedure Interventions
Monitor VS
Assess CMS (circulation, motion, sensation)
Cardiac monitoring
Maintain pressure dressing
Monitor for bleeding
Encourage fluids
Assess S/S of dye hypersensitivity
Keep extremity extended (no bending)
Do not elevate HOB – (lay supine)
No getting up for 6-12 hours (lay supine)
Lab values
Lipids:
Triglycerides:
40 to <160 mg/dL (males)
35 to <135 mg/dL (females)
Total Cholesterol: <200 mg/dL
LDL: (bad cholesterol) <130 mg/dL
HDL: (good cholesterol)
Male: >45 mg/dL
Female: >55mg/dL
ADH 1-5 pg/mL
Lab values (cont’d)
RBC
men – 4.0 to 6.0 x /L.
women – 3.8 to 5.2 x /L.
Hgb
men: 13.2 and 16.6 (g/dL)
women: 11.6 and 15 g/dL
Cardiac Markers:
CK-MB: normal 0%
Troponin-I: < 0.03 ng/mL
Troponin-T: <0.2 ng/mL
BNP – b-type natriuretic peptide: <100 pg/mL
Cardiac Medications
Classes: Beta Blockers, Calcium Channel Blockers, Cardiac Glycosides
Beta-Adrenergic Blockers (Beta-Blockers)
B1 selective
Relaxes the heart
e.g., Metoprolol (Lopressor) Atenolol, Esmolol
B1 and B2 Non-selective
Relaxes the heart and lungs
eg. Propranolol (Inderal), Carvedilol (Coreg)
Two Types:
Hypertension
Supraventricular tachydysrhythmias
Heart Failure; Angina; Myocardial Infarction
Migraine (Inderal)
Anxiety
Beta Blockers
Mechanism of action:
Blocks beta-receptors of the sympathetic nervous system.
Prevent adrenaline from binding to receptors.
Effects:
Reduce heart rate
Reduce blood pressure
Decrease myocardial contractility by slowing AV conduction
Beta Blockers
Adverse Effects:
Hypotension, bradycardia,
Bronchospasm, dyspnea (Lt HF)
JVD, edema, weight gain (Rt HF)
Cold extremities, exacerbation of Raynaud's disease
Fatigue, dizziness, orthostatic hypotension
Nursing Implications:
Hold Apical Rate < 60
Hold Systolic < 90
Heart Failure
Asthmatics
Pregnancy
Patient Teaching
Pulse, BP checks
Diabetics =Glucose checks
Do not stop abruptly
Calcium Channel Blockers
Amlodipine, Cardizem, Procardia, Verapamil
Affect smooth muscle contraction by blocking calcium during depolarization, relaxing smooth muscles in the heart
Treatment:
Angina
Hypertension
Supraventricular tachycardia
Calcium Channel Blockers
Less in the muscle = less force of contraction
Slows the SA/AV node = decreased HR (diltiazem & verapamil)
Vasodilation- (peripheral and/or coronary)
Relaxes smooth muscle
Calcium Channel Blockers
Adverse Effects:
Bradycardia, hypotension
Chest pain
Bronchospasm, dyspnea
Peripheral edema
Fatigue
Dizziness, orthostatic hypotension, flushing
Headache
Reflex tachycardia
Nursing Implications:
Hold if Systolic <90
Hold if Pulse <50
Monitor Labs; Liver and renal studies
Administration issues
Patient Teaching
Pulse, BP checks
All Adverse effects
Do not stop abruptly
No grapefruit/juice
Cardiac Glycoside: Inotropic Agents - Digoxin
Mechanism of Action: Inhibits Na K+ ATPase enzyme, Increases force of heart's contractions
Used to Treat: Atrial Fibrillation, Atrial Flutter, Congestive Heart Failure
Side Effects: Unusual Tiredness and Fatigue, Anxiety, Hallucinations
Poisoning and Toxicity: Visual Disturbances, Nausea or Vomiting, Arrhythmias, Electrolyte Imbalances, Can be worsened by other medications
Contraindication: Ventricular Fibrillations
Digoxin: Nursing Implications
Monitor:
S/Sx of toxicity
Serum digoxin level
Hold if > 2 ng/ml = toxic
Hold medication if apical pulse < 60
Monitor electrolytes: Potassium, Calcium, and Magnesium level
Have antidote available for dig toxicity
Digoxin immune fab (digibind)
Patient teaching:
Avoid foods high in K+ potassium (unless on diuretics)
Check pulse daily, hold if <60
s/s toxicity & report to PCP
Abdominal pain
N/V, anorexia
Visual disturbances
Arrhythmias
Must have dig levels drawn as prescribed
Nursing Process and Care Plan
Assessment:
General physical assessment with focused cardiac assessment –Chief Complaint
Health history, family history, psychosocial
Diagnostic tests and labs: EKG, Chest Xray, cardiac enzymes
Respiratory signs/symptoms
Nursing Diagnosis
Decreased Cardiac Output
Decreased Activity Tolerance
Acute Pain
Fear/Anxiety
Risk for Impaired Gas Exchange
Fatigue
Planning (Expected Outcome)
Patient will maintain BP within an individually acceptable range.
Implementation
Reduce activities that put stress on the heart such as limit visitors each hour.
Monitor and record BP in both arms q 2 hours.
Administer propranolol (beta blocker) as directed.
Evaluation
(Outcome met/not met/partially met)