Graves
Management of Neonates Born to Mothers With Graves’ Disease
Authors and Affiliations:
Daniëlle C.M. van der Kaay, MD, PhD, et al. from the Division of Endocrinology, The Hospital for Sick Children, University of Toronto.
Emphasizes the collaborative effort in planning, drafting, and reviewing the manuscript among the authors.
Overview
Risk to Newborns: Newborns of mothers with Graves’ disease (GD) are at increased risk for developing neonatal hyperthyroidism, which can lead to significant morbidity and mortality.
Current Guidelines: Lack of consensus guidelines for managing these cases necessitated a literature review to establish a management approach.
Background
Prevalence:
Maternal hyperthyroidism prevalence: 0.1% - 2.7%.
Transient GD in infants ranges from 1.5% - 2.5%, potentially up to 20% in observational studies.
Causative Mechanisms:
TSH receptor antibodies (TRAb) are responsible for GD and freely cross the placenta during the latter half of pregnancy.
Two types of TRAb: TSH-stimulating antibodies induce excess thyroid hormone production, while blocking antibodies do not initiate signaling.
Management Algorithm
Initial Risk Assessment:
Base assessment on maternal TRAb levels:
Negative → no follow-up needed.
Positive or unavailable → newborn considered "at risk" for hyperthyroidism.
Cord Blood Testing:
Determine TRAb levels as soon as possible to guide discharge decisions.
Thyroid Function Tests:
Not necessary to measure cord TSH and fT4 levels.
Follow-Up Testing:
Perform fT4 and TSH levels at days 3-5 and then again at days 10-14, with clinical follow-up until 2-3 months.
Treatment Protocol:
Methimazole (MMI) as first line; adjunctive β-blockers for sympathetic hyperactivity when necessary.
Potassium iodide may be used in refractory cases.
Monitoring Frequency:
Weekly assessment of MMI-treated infants until stable.
Understanding Potential Hypothyroidism:
Be aware of the risks of central or primary hypothyroidism in affected newborns.
Clinical Significance of Maternal TRAb
Maternal TRAb levels may persist post-treatment, affecting the newborn.
High maternal TRAb correlates with increased hyperthyroidism risk in newborns.
Evaluating TRAb levels at 20-24 weeks gestation is recommended.
Neonatal Signs and Symptoms of GD
Symptoms include:
Goiter, low birth weight, irritability, diarrhea, and cardiac issues, among others.
Diagnosis can be complicated due to overlaps with other conditions such as sepsis.
Severe cases have been associated with high mortality rates.
Treatment Considerations
Treatment initiation may prevent complications such as heart failure or developmental issues.
Guidelines suggest MMI treatment based on objective measures of hyperthyroidism.
There is no clear consensus on treating asymptomatic newborns with hyperthyroidism.
Ongoing monitoring of thyroid function is critical in this population.
Timing and Frequency of Assessments
Initial TFTs should be performed between days 3-5 of life. If normal, follow-ups may be ceased after 2 weeks.
For at-risk infants, continued monitoring up to 3 months is advisable.
Conclusion
Effective management of neonatal hyperthyroidism due to maternal GD is crucial for preventing morbidity and mortality.
Ongoing refinement of management algorithms based on research findings will enhance care for these patients.
Key Terms
ATD: Antithyroid drug.
TB: Thyroid Binding.
TFT: Thyroid function test.
TRAb: TSH receptor antibodies.
GD: Graves’ disease.
MMI: Methimazole.
PTU: Propylthiouracil.