Interpretation of Lesion
What should I do first?
You need to determine what there are Red Flags present, Yellow Flags present, in order to take appropriate action
Decide if the problem is in the scope of PT practice
Is there a need for medical referral
Examination
What you do in the the initial examination depends on the Stage of Healing
Appropriate measures to assess body structure and function
Appropriate assessment of activity limitations: outcome measures
Appropriate physical performance measures: when you do these depends on healing
Synthesize info. gathered and see if there are clusters of findings that will give you a working hypothesis and PT diagnosis
Review of Acute vs. Chronic Lesions
Acute inflammation
SHARP, pain, swelling, heat, redness, and loss of function
Chronic inflammation
characterized by an attempt to repair with increased numbers of fibrocytes and other tissue building cells and the Prescence of granulation tissue
Acute Lesion
Have the normal physiological response to adverse chemical, thermal, or mechanical stimulus
New and never experienced before
Pain is relatively constant or reoccurs frequently
Often severe and disabling
Characterized by anxiety
Acute
Impaired movement; mov’t aggravates
With PROM of the related joint there is muscle spasm end feel or empty end feel; may have guarding or muscle spasm
Pain is referred and diffuse over the related segment
Increase in skin temp
Effusion (joint) or swelling
Difficulty falling asleep or remaining asleep
Intervention Acute Lesion
Goals
Decrease pain
Control inflammation/edema
Protect structures from further damage
Prevent the deleterious effects of rest
Intervention Acute Lesions
PRICEMEM
P - Protection: avoid excess tissue loading
R - Rest: absence from abuse rather than absence from activity
I - Ice: 10-15 minutes every 1-2 hours
C - Compression: elastic bandage, pneumatic device, supports to decrease swelling
E - Elevation: do until swelling is resolved
M - Manual techniques: variety of them
E - Early motion: passive, active assisted, active; depends on the lesion
M - Medication: NSAIDS (PT can’t prescribe meds, refer to doctor)
Review of Chronic
With PROM there is no muscle spasm or empty end feel
Little or no temp. change
Usually no problem with sleep, exception is the shoulder and hip due to lying down
Chronic Lesions
Pain is typically more aggravating than worrying
No alarming associated symptoms
Lasts longer than 6 months
Others >3months other >4-7 wks
Pain experienced before and has reoccured
Pain is increased by specific activities and relieved by rest
Usually mild/mod
Intervention Chronic Lesions
Not inflammatory
Minimal modalities
Exercise
ROM
Strengthening: needs to be progressive
Balance/proprioception
Function
Posture
Body mechanics: functional activites
Return to activity training
Conditioning
Manual Therapy - massive, jt. mobilizations/manipulation, MET, Craniosacral etc.
Education
Pain as conceptual inference; body’s best guess that protective action is required
Explaining that pain is not a matter of tissue damage or pathology, matter of the perceived need to protect body tissue
Discuss that pain is modulated by other sensory inputs and cognitive, social and emotional factors
Need activity modification or postures to minimize pain
Expected outcome
Why one needs to exercise: Patient Related Instruction
Cognitive Functional Therapy (CFT) - is an approach that aims to address the multidimensional nature of pain was simultaneously addressing and biomechanical contributors and acknowledging the important roles of pain-inhibitory processes related to reducing the fear of pain
Determine painful postures and painful movements and then modify defending postures or movement while introducing helpful techniques such as relaxed breathing, during the movement. aim is decrease symptoms for pt.
Graded Motor Imagery (GMI) - starts where person views images of body parts that are displayed and determine if left or right, progresses to explicit motor imagery where person imagines adopting posture shown in the image and progresses to mirror box training
Quick Review of Findings
Response to resistive testing
Strong and painless → normal response
Weak and painless → Nerve palsy or complete rupture
Strong and painful → Grade 1 contractile
Weak and painful → More significant contractile lesion grade 2; possible other serious pathology
SEE CHART ON SLIDE 26 AND USE FLASHCARDS
Fractures
Review types of fractures and fracture healing from patho
Lesion of Bone: Fracture
Fracture
Usually associated with trauma or repeated trauma
Osteoporosis and other bone diseases are exceptions; fragility fracture can occur without trauma
ID by examination, plain films (x-rays), CAT scan, bone scan
Symptoms
Usually VERY painful, deep pain
Look at jt. position, malalignment
Muscle guarding
Swelling secondary to tissue damage
Pt will not want to move part because of pain
Management
First goal is stabilization of the fracture site, medical management: cast, traction, splint, surgery, etc.
Early PT Intervention: needs to understand the medical management of the fracture and soft tissue
Check for DVT
Safety with bed mobility (UE/LE), transfers (UE/LE), gait and stairs if LE
After healing look for signs of malunion; inspect bone structure and alignment. What you do in initial examination depends on the stage of healing
Look at stage of healing - Refer to patho lecture for bone healing; Pg. 47-50 Dutton
Intervention are for the underlying impairments!
We want to know if part of the medical management was Demineralized Bone Matrix: DBM
Modalities to assist fracture healing
Pulsed Electromagnetic Field (PEMF)
Pulsed US
Direct Current
Safe and effective positioning and movement
LE assistive device and gait training (crutches)
Fracture Overview:
Decrease in ROM, AAROM, AROM, PROM; later stretching. Where you start depends on the stage of healing. Need to be in contact with MD; how will progress?
Decrease in Strength: depends on healing stage and soft tissue involvement; how will we know?
Decrease in function: ADL, IADL, gait, transfers, work and leisure activities. In early PT looking for protection; will address restoration of function as bone heals
Balance and proprioception come with later stages
Lesion of Bone: Dislocation
Dislocation: joint integrity comprised, and bone moves out of place, usually associated with injury
ID by examination and plain films (x-rays)
Maybe obvious on inspection, malalignment
Usually very painful
Protective positioning
Unable to move
Remember soft tissue injury occurs to the supporting structures
Labral tear
Mm tears
Capsule involvement
Management
first goal is reduction of dislocation; may need to go under anesthesia (medical management)
depends on severity of dislocation, single vs. recurrent dislocation; will have surrounding soft tissue injury
Immobilization, may need surgery for stabilization
Healing stages of soft tissue; need protection of structures
Common impairments: pain, swelling, loss of motion, impaired motor performance, if LE gait
PT Interventions depends on Impairments and tissue healing
Protection stage for healing:
Pain control:
Educate pt. on resting position
Modalities for pain
Reduce swelling
Positioning
Modalities
Safe transfers/safe gait
ADL modification
Lesion of Bone: Demineralization
Refer to patho, Dutton pgs. 218-219
Result of Insufficient bone formation, excessive bone re-absorption, or both
Decrease bone mass, narrowing of the bone shaft, widening of the medullary canal
ID with DEXA scan
DEXA Scan:
Osteopenia = -1 to -2.5 T score
Osteoporosis = <2.5 T score
Most common areas
Thoracic spine, ribs, proximal femur, distal forearm
Best method is PREVENTION
Stop smoking
Decrease alcohol consumption
Appropriate body fat
Increase calcium and Vit D
Decrease caffeine consumption
Minimize corticosteroid
Exercise: weight-bearing activities, strength training
Patient education
General Interventions for Osteoporosis
Posture “zone”
Strengthening, flexibility, and aerobic conditioning
Balance training
Safe ADL and IADL
Specific CPG Definitions (Don’t need to know for test)
SWB → Static weight-bearing exercise including single-leg standing
DWBLF → Dynamic weight-bearing exercise with low force including walking and tai chi
DWBHF → Dynamic weight-bearing exercise with high force including jogging, jumping, running, dancing, and vibration platform
NWBLF → Non-weight bearing exercise with low force, example low-load high repetition step training
NWBHF → Non-weight bearing exercise with high force, example progressive resisted strength training
Combination → Combination of exercises types with more than one of the above exercise interventions
Bone: Osteomyelitis - Acute or chronic inflammatory process of the bone secondary to infection with pyogenic organisms or other sources of infections, such as TB, fungal infection, parasitic infection, viral infection
Individuals who are pre-disposed
DM
Sickle cell
Acquired immune deficiency syndrome
IV drug abuse
Alcoholism
Chronic steroid use
Immunosuppression
Chronic joint disease
Clinical signs and Symptoms
Fever
Fatigue
Edema
Erythema
Constant pain
Tenderness over the involved bone
Decrease use of the extremity
Intervention: Medical management with the appropriate medications; may require surgical management
PT: May be involved with bed mobility, transfers, and gait training with assistive device. Maybe involved after cleared to address impairments (ROM, strength, balance/proprio, function)
Osteomalacia - NOT ON TEST
Paget’s Disease - NOT ON TEST
Chondral/Cartilage: Types
Fibrocartilage
Elastic
Articular
Know the time frames for the healing of these different cartilages ^^^ pg 44-46 Dutton
Etiology of Chondral/Cartilage Issues
Trauma - acute injuries such as dislocation, fractures, sport impact injuries
Usually younger, more physically active people
Usually in only 1 area; mild to severe
Degenerative changes - chronic wear and tear, repetitive microtrauma, underlying conditions such as osteoarthritis
Structural abnormalities - that lead to altered joint mechanics
Inflammatory disorders - such as RA other inflammatory arthritis
Diagnosis
History
Clinical assessment
Imaging (MRI, CT)
Arthroscopic evaluation
Management
Conservative management - activity modification, PT, anti-inflammatory medications, intra-articular injections such as corticosteroids or hyaluronic acid
PT management will depend on acuity, specific impairments and functional limitations. Will also depend on what was done as medical management
physical agents: decrease pain swelling
ROM
Ther ex
Education
Balance/coordination
Gait training
Surgical intervention - for symptomatic progressive chronal lesions that fail to respond to conservative
Scope (is there a POC in place)
Osteotomy: realignment
Joint replacement
Microfracture - minimally invasive procedure where small holes are created in subchondral bone to stimulation formation of fibrocartilage, promoting repair and regeneration NOT ON TEST
DO NOT HAVE TO MEMORIZE THE CELLULAR LEVELS OF DAMAGE
Most common lesion of cartilage is Degeneration
Chronic cartilage damage
Is the result of continual wear and tear
Usually more widespread and located in different areas
The cause of the wear and tear on the cartilage differes from indiv. to indiv.
Grading Outerbridge Classification (ON TEST)
Stages of Knee Osteoarthritis
Grade 1 - softening of the cartilage in combination with swelling
Grade 2 - is where there is fibrillation where there is a partial-thickness defect; the diameter does NOT exceed 1.5 cm or where it fails to reach the subchondral bone
Grade 3 - is where the partial-thickness DOES exceed 1.5cm in diameter and/or reaches the subchondral bone
Grade 4 - is the most serious occurs where the subchondral bone becomes fully exposed (knee replacement)
Articular Cartilage: Degeneration
Clinical Manifestations
Boney Enlargement
Crepitus with movement
Jt. deformity/malalignment
Loss of ROM
Tenderness
Articular gelling
Management
Education
May need assistive devices
Activity modification
Jt. protection
Proper weight
Exercise
Intra-articular Fibrocartilage
Involves intra-articular fibrocartilaginous discs and menisci
Usually tearing from traumatic injury or repeated stress
ID with MRI, CT scan
Minor lesion - will have clicking of the joint, with specific movements; may have instability, locking
Major lesion - usually have greater interference of jt. mechanics with decrease in joint motion in non-capsular pattern
If result of trauma, may also cause strain on jt. capsule so will get a restriction of movement in capsular pattern
Medial meniscus is often torn over lateral meniscus
Surgical options:
Palliative -- Include simple arthroscopic debridement and lavage
Reparative - this approach consists of marrow stimulation techniques that result in the formation of fibrocartilage.
Restorative - This includes osteochondral or astrocyte transplantation and involved inducing newly attracted or transplanted chondrocytes to become mature chondrocytes using growth factors
LOOK AT DIAGNOSTIC WORKSHEET ON MOODLE KNOW GUIDELINES
Articular Cartilage: Loose Body “Locked knee”
Fragment of cartilage is free in jt.
Occurs in late stages of degeneration as result of avascular necrosis
May also be result of traumatic injury
Symptomatic when it changes mechanics of jt.
Management
Return the displaced structure
Manipulation or traction
Positioning
Removal
PT management depends on impairments and functional limitations
ROM
Strengthening
Functionals
Balance/proprio
Gait
Physical agents; to address pain/swelling
Joint Capsule: Fibrosis
Typically occurs with
Prolonged immobilization of joint
Low grade inflammatory process of DJD
With resolution of acute inflammation of synovium
Adhesive Capsulitis
Risk Factors
Female
DB or thyroid
Age 40-65
Previous episode of adhesive capsulitis
Findings
Multiregional synovitis; inflammation
Synovial angiogenesis (increased capillary growth)
New nerve growth in capsuloligamentous complex which may explain heightened pain response
Capsuloligamentous complex fibrosis
Adhesive Capsulitis: 4 stages
Stage 1
Sharp pain at end of ROM
Achy pain at rest
Sleep disturbance
There is synovial inflammation but without adhesions
Minimal or no loss of ROM
Early loss of ER with an intact rotator cuff is hallmark sign
May last up to 3 months
Stage 2 “Freezing stage”
This is the painful stage. Any movement of the shoulder causes pain
Gradual loss of motion in all directions; both AROM and PROM. Especially elevation and ER
This stage lasts from 3 to 9 months from onset of symptoms
Stage 3 “Frozen stage”
Pain might lessen because of less synovitis but still painful
The shoulder becomes stiffer and greater loss of motion
Using it becomes more difficult
This stage lasts from 9 to 15 months from the onset of symptoms
Stage 4 “Thawing stage”
Pain lessens significantly or resolves
The shoulder’s ability to move begins to improve
This stage lasts from 15 to 24 months from the onset of symptoms
Adhesive Capsulitis: Shoulder
Joint motion is limited
ROM loss >25% in at least 2 planes and ER loss >50% or ER <30 degrees
ER loss with arm at side, loss abd, loss IR
Capsular end-feel: leathery
Pain complains of stiffness and loss of motion
Pain varies depending on stage
Loss of function: dressing, reaching overhead, reaching behind back or out to the side
May have weakness IR, ER, elevators
Adhesive Capsulitis: Intervention
Need to determine the irritability; the intervention depends on this
If in irritable stage need to reduce the irritability: suggest talk with MD regarding steroid
Joint Capsule: Fribrosis