Overview of Autonomic Nervous System (ANS) Innervation
The autonomic nervous system innervates organs such as the heart, providing motor functions to various muscle types and glands.
It consists of sympathetic and parasympathetic branches, which regulate bodily functions unconsciously.
Heart as a Focus Example
The heart is innervated by autonomic pathways that influence cardiac muscle function.
Key components include:
SA Node: Sinoatrial Node, primary pacemaker of the heart.
AV Node: Atrioventricular Node, secondary pacemaker.
Central Regulation of Heart Function
Brain and Spinal Cord Involvement:
Motor functions initiated from the brain stem and spinal cord.
Cardioacceleratory Center: Located in the brain stem, regulates heart rate and myocardial contractility.
Sympathetic Nervous System Activation
Sympathetic activation is referred to as the 'fight or flight' response.
It leads to:
Increased heart rate to deliver more oxygen to tissues.
Increased myocardial contractility to pump more blood effectively.
To activate the sympathetic pathway:
Information is sent from the brain stem down to the spinal segments T1 to T2.
Pathway from CNS to Heart
At spinal levels T1 to T3:
Gray Matter: Contains autonomic cell bodies in the ventrolateral horn.
The autonomic signal leaves via the ventral root and links with the dorsal root to form a typical spinal nerve.
Descending fibers can target ganglia:
Sympathetic Chain Ganglia: Includes inferior, middle, and superior cervical ganglia.
Preganglionic neurons synapse at varying levels within the sympathetic chain.
Characteristics of Neurons in Sympathetic Pathway
Preganglionic Neurons:
Myelinated and synapse at sympathetic chain.
Release Acetylcholine (ACh) onto postsynaptic cells at nicotinic receptors.
Postganglionic Neurons:
Longer neurons that exit the spinal nerve and travel via the vagus nerve and other pathways to the heart.
Release Norepinephrine (NE) onto target receptors in the heart.
Receptor Binding and Physiological Effects
Postganglionic Action: Norepinephrine acts on adrenergic receptors:
Alpha-1 Receptors: Primarily found in vascular smooth muscle.
Beta-1 Receptors: Specifically located in the heart.
Upon binding to Beta-1 receptors:
Norepinephrine activates a second messenger cascade, increasing intracellular calcium levels.
Result: Increased contractile force in cardiac tissue, enhancing the heart's pumping effectiveness.
Chronotropic and Ionotropic Effects:
Heart rate is increased (chronotropic effect).
Contractility is also enhanced (ionotropic effect).
Summary of Fight or Flight Response in Heart Function
The entire process facilitates an increased cardiac output, primarily during stress and physical exertion to support the body's immediate energy needs.