Metabolism
Metabolism Overview
Definition: Metabolism is the sum of all chemical changes in the body.
Reactions: There are two classes of metabolic reactions:
Anabolism: Requires energy to build complex molecules.
Catabolism: Releases energy by breaking down molecules.
Carbohydrates
Glucose Catabolism:
Most dietary carbohydrates are burned within a few hours of absorption.
Glucose is the primary product of carbohydrate digestion.
Overall reaction: C6H12O6 + 6O2 → 6CO2 + 6H2O, occurring through a series of enzyme-controlled steps aimed at producing ATP.
Major Pathways:
Glycolysis: Splits glucose into two pyruvate molecules; occurs in the cytoplasm.
Anaerobic Fermentation: Processes pyruvate to continue glycolysis without oxygen, also in the cytoplasm.
Aerobic Respiration: Occurs in mitochondria, oxidizing pyruvate to CO2 and water.
Coenzymes
Key Coenzymes:
NAD+: Derived from niacin (vitamin B3).
FAD: Derived from riboflavin (vitamin B2).
Enzymes remove electrons from glucose metabolites and transfer them to coenzymes, acting as temporary energy carriers.
Essential for glucose catabolism; enzymes cannot function without coenzymes.
Glycolysis
Process Summary:
Converts glucose into two pyruvate molecules.
Requires an initial investment of 2 ATP; generates 4 ATP (net yield of 2 ATP).
Requires 2 NAD+ which is converted to NADH during the process.
Results in 2 ATP, 2 NADH, and 2 pyruvate.
Fate of pyruvate depends on the presence of oxygen.
Anaerobic Fermentation
In the absence of oxygen, pyruvate is converted to lactic acid, generating NAD+ essential for recycling in glycolysis.
Produces only 2 ATP; useful only for short-term energy (e.g., during intense exercise).
Aerobic Respiration
Process Overview: Occurs when oxygen is available, with pyruvate entering the mitochondria for further oxidation.
Matrix Reactions: Part of the citric acid cycle; processes each pyruvate to produce 2 ATP, 6 CO2, 8 NADH, and 2 FADH2.
Membrane Reactions: Oxidize NADH and FADH2 via the electron transport chain, where electrons transfer leads to ATP production and water formation.
ATP Production
Summary of Yield:
Glycolysis produces 2 ATP.
Matrix reactions yield 2 ATP.
Membrane reactions yield up to 28 ATP.
Total yield from one glucose molecule = 32 ATP.
Glucose Production and Utilization
Glycogen and Glucose Hydrolysis:
Excess glucose is converted to glycogen or fat.
Processes:
Glycogenesis: Synthesis of glycogen, stimulated by insulin.
Glycogenolysis: Hydrolysis of glycogen, stimulated by glucagon.
Gluconeogenesis: Conversion of non-carbohydrates into glucose.
Lipids
Lipogenesis: Formation of triglycerides from excess glucose and amino acids.
Lipolysis: Breakdown of triglycerides into fatty acids and glycerol, especially when glucose is low.
Fatty acids yield more ATP (129 ATP) compared to glucose due to their oxidation to acetyl groups.
Proteins
Amino acids primarily used for protein synthesis.
Can be converted to glucose or fat; also have the potential to be used directly for energy after deamination (removal of the amine group produces ammonia, which is converted to urea).
Metabolic States
Absorptive (Fed) State:
Lasts about 4 hours post meal; body absorbs nutrients.
Carbohydrates primarily transported to the liver, fats distributed to tissues, and amino acids used for protein synthesis.
Regulation of Absorptive State:
Largely driven by insulin.
Insulin promotes glucose uptake, substrate storage, and protein synthesis.
Postabsorptive (Fasting) State:
Body utilizes stored fuels; involves glycogenolysis and gluconeogenesis to maintain blood glucose levels critical for brain function.
Protein usage increases when glycogen and fat stores deplete.
Metabolic Rate
Definition: Energy use in the body measured in kcal/hour.
Influenced by activity level, mental state, and hormonal levels.
Basal Metabolic Rate (BMR): Energy used at rest; average is ~2000 kcal/day. Total Metabolic Rate (TMR) = BMR + voluntary activities.
Factors Affecting Metabolic Rate
Increase in TMR: Physical activity, pregnancy, environmental temperature change (fever).
Decrease in TMR: Prolonged fasting, depression, weight-loss diets (slow metabolic adaptation).
Appetite Regulation
Energy balance affects weight; determined by energy intake vs. output.
Short-term appetite regulation involves hormones:
Ghrelin: Hunger hormone stimulating appetite.
Peptide YY and CCK: Signals satiety and regulate intake.
Long-term regulation by:
Leptin: Reflects fat stores; inhibits appetite and promotes fat breakdown.
Insulin: Lowers blood glucose and encourages fat storage.
Hypothalamus plays a key role in processing signals from these hormones to regulate appetite effectively.