1.4 Adrenal Glands: Zona Fasciculata
Zona Fasciculata Overview
Composed of cells arranged in bundles resembling fascicles.
Cells here are target cells for ACTH and produce glucocorticoids.
Glucocorticoids
Definition: Hormones produced from the adrenal cortex that have significant effects on glucose levels.
Cortisol: The primary human glucocorticoid.
Functions of Cortisol
Regulation of Metabolism during Long-term Stress
Ensures cells receive necessary nutrients to manage prolonged stress conditions (from hours to even decades).
Increases Protein Metabolism & Fat Breakdown
Enhances lipolysis (fat breakdown) predominantly through adipocytes.
Encourages protein catabolism, especially in muscle tissue, leading to muscle wasting.
Promotes Gluconeogenesis
Stimulates the synthesis of glucose from non-carbohydrate sources (e.g., amino acids).
Resistance to Stress
Provides the body with nutrients during stress by mobilizing resources into the bloodstream.
Additional Functions of Glucocorticoids
Increased Blood Pressure
Enhances blood vessel sensitivity to hormones like angiotensin II, promoting vasoconstriction.
Anti-Inflammatory & Immunosuppressive Effects
Reduces histamine release from mast cells, decreases capillary permeability, and inhibits phagocytosis, reducing inflammation.
Example usage: Cortisone shots for inflammation reduction.
Regulation of Glucocorticoid Release
Governed by a negative feedback loop involving hypothalamus and anterior pituitary.
Increased glucocorticoid levels suppress CRH and ACTH production, reducing further glucocorticoid secretion.
Clinical Implications of Glucocorticoid Administration
Example: A heart transplant patient receiving prednisone (a modified glucocorticoid) will have low ACTH and CRH levels due to negative feedback suppression.
Endocrine Pathologies
Cushing's Syndrome
Caused by hypersecretion of glucocorticoids (especially cortisol).
Symptoms:
Redistribution of fat (e.g., moon face, buffalo hump).
Muscle atrophy due to protein catabolism.
Poor wound healing and increased bruising due to immune suppression.
Addison's Disease
Caused by hyposecretion of glucocorticoids and possibly aldosterone.
Symptoms:
Hypoglycemia from low cortisol levels.
Muscle weakness and low blood pressure from low aldosterone (due to low sodium retention).
Dehydration and potential cardiac arrest from high potassium levels due to low aldosterone.
Skin darkening due to high ACTH levels as a compensatory response (due to low glucocorticoid production).
Notable Example
JFK and Addison's Disease: History of JFK exhibiting symptoms consistent with Addison's, including skin pigmentation changes and autoimmune destruction of adrenal cortex.