SCHIZOPHRENIA
Foundations
Schizophrenia causes distorted and bizarre __________.
Public perception has historically viewed schizophrenia as __________.
Schizophrenia is now recognized as an illness that can be controlled with __________.
Peak incidence for males occurs in the __________.
Peak incidence for females occurs in the __________.
Schizophrenia affects about __________ of the population.
Symptoms
Positive symptoms include delusions, hallucinations, and disorganized __________.
Negative symptoms include flat affect, lack of volition, and __________.
Medication may control __________ symptoms.
Negative symptoms often __________.
Negative symptoms present barriers to __________.
Onset & Early Signs
Onset of schizophrenia can be abrupt or __________.
Most clients develop symptoms __________.
Early symptoms include social withdrawal, loss of interest, and neglected __________.
Diagnosis is made when __________ symptoms appear.
Course & Prognosis
Earlier onset of schizophrenia leads to __________ outcomes.
Younger clients show poorer premorbid adjustment and greater __________ impairment.
Gradual onset correlates with poorer __________ outcomes.
Relapse is common and linked to nonadherence, substance use, and poor __________.
Neurologic soft signs include sensory and __________ deficits.
One clinical pattern involves ongoing psychosis without full __________.
Another pattern involves alternating episodes of psychosis and __________.
Intensity of psychosis can __________ with age.
About __________ of clients recover completely.
Long‑term impairment affects social and __________ functioning.
Schizophrenia becomes less disruptive over time, allowing some clients to live __________.
Persistent negative symptoms and impaired cognition hinder full __________.
Stigma and social withdrawal are not improved by __________.
Early detection and aggressive treatment improve __________.
Continued medication and psychosocial interventions reduce __________ rates.
Related Disorders
Schizoaffective disorder combines schizophrenia and __________ symptoms.
Schizophreniform disorder lasts less than __________ months.
Diagnosis changes to schizophrenia if symptoms persist beyond __________ months.
Catatonia involves marked __________ disturbance.
Delusional disorder involves one or more __________ delusions.
Delusions & Hallucinations
Persecutory delusions involve beliefs of being __________.
Erotomanic delusions involve false beliefs of __________.
Grandiose delusions involve inflated sense of __________.
Somatic delusions involve false beliefs about __________.
Psychosis includes delusions and __________.
Hallucinations are false __________ perceptions.
Auditory hallucinations may include __________ hallucinations.
Command hallucinations may instruct the person to perform __________ actions.
Etiology
Etiology of schizophrenia is __________.
Genetic vulnerability interacts with __________ factors.
Environmental risk factors include pregnancy complications, substance misuse, and __________.
Immediate family members have increased __________ risk.
Identical twins have a __________% risk.
Fraternal twins have a __________% risk.
Children have a __________% risk if one parent has schizophrenia.
Children have a __________% risk if both parents have schizophrenia.
Biologic Factors
Brain structure differences include enlarged __________.
People with schizophrenia have less brain tissue and __________.
They also have diminished glucose metabolism in __________ structures.
Neurochemical theories involve dopamine, serotonin, norepinephrine, and __________.
Excess dopamine induces __________ reactions.
Serotonin modulates and controls excess __________.
Immunovirologic theories involve exposure to __________.
Higher rates occur after __________ epidemics.
Prodromal phase may last __________ before full psychosis.
Communication Guidelines
Medication response varies due to genetic differences in __________.
When communicating with delusional clients, be sincere and __________.
Avoid vague or __________ remarks.
Do not make promises you cannot __________.
Encourage clients to talk without __________.
Do not argue or try to __________ the client about delusions.
Focus on __________ things, not delusional material.
Gradually introduce clients to __________ activities.
Show empathy and reassure __________.
Never convey acceptance of delusions as __________.
Interject doubt only when the client is __________.
Help clients differentiate between holding a belief and __________ on it.
Treatment
Antipsychotics decrease __________ symptoms.
Antipsychotics do not __________ schizophrenia.
Typical antipsychotics target __________ symptoms.
Atypical antipsychotics may lessen __________ symptoms.
Examples of typical antipsychotics include chlorpromazine and __________.
Examples of atypical antipsychotics include clozapine and __________.
Side effects of antipsychotics are a major cause of __________.
Side Effects & Management
EPS include dystonia, akathisia, and __________.
Tardive dyskinesia is __________.
Neuroleptic malignant syndrome is rare but __________.
NMS symptoms include muscle rigidity, high fever, and elevated __________.
Benztropine requires increased __________ intake.
Diphenhydramine may cause __________.
Propranolol may cause dizziness and cold __________.
Psychosocial Interventions
Psychosocial treatment includes individual and __________ therapy.
Cognitive adaptation training uses environmental __________.
Cognitive enhancement therapy combines computer training with __________ sessions.
Family education reduces __________ rates.
ACT programs reduce hospital admissions by managing __________.
Case managers help with housing, transportation, and __________ management.
Schizophrenia affects all aspects of life including social interactions and __________ health.
Positive symptoms include delusions, hallucinations, and disordered __________.
Negative symptoms include social isolation, apathy, and lack of __________.
Each client must be individually assessed to determine appropriate __________.
SCHIZOPHRENIA — ANSWER KEY
Thoughts, perceptions, emotions, movements, and behavior
Dangerous and uncontrollable
Medication
Late teens to early 20s
20s to early 30s
1%
Thinking, speech, and behavior
Social withdrawal or discomfort
Positive
Remain and persist
Recovery
Insidious
Gradually
Hygiene
Positive
Worse
Cognitive
Immediate and long‑term
Social relationships
Motor
Recovery
Recovery
Diminish
10% to 15%
Occupational
Independently or in structured settings
Independence
Medication
Outcomes
Relapse rates
Mood disorders
6
6
Psychomotor
Nonbizarre
Followed or harmed
Love
Power or importance
Bodily function
Hallucinations
Sensory
Command
Dangerous or life‑threatening
Multifactorial
Environmental
Poverty or social isolation
Genetic risk
50%
15%
One (16%)
Both (46%)
Ventricles
Cerebrospinal fluid
Frontal cortical
Glutamate
Psychotic reactions
Dopamine
Viruses
Influenza
Months or years
Metabolism
Honest
Evasive
Keep
Prying
Convince
Real
Group
Presence and acceptance
Reality
Ready
Acting
Psychotic
Cure
Positive
Negative
Fluphenazine
Risperidone
Nonadherence
Parkinsonism
Irreversible
Fatal
Creatine phosphokinase
Fluid and fiber
Sedation
Hands and feet
Group
Supports
Group
Relapse
Symptoms and medications
Money
Emotional
Thought processes
Motivation or volition
Nursing actions