S2W4 Pacing Q& A MT
ADD IMAGES
Questions and Answers Overview
Question 1: Patient Device Requirements
Atrial Only Device:
Indicated for young patients with sino-atrial disease that have good AV node conduction.
RV Only Device:
Recommended for pacing in patients who are in permanent Atrial Fibrillation (AF).
Dual Device:
Suitable for all other patient populations.
Question 2: Advantages of Single Chamber Device
Benefits:
Simpler implantation process.
Lower initial cost of device.
Important Consideration:
Dual devices provide additional protection against future AV node involvement.
Current medical practice has shifted towards utilizing the dual system.
Clinical Observations
Clinical evidence shows increased left atrial diameter, decrease in left ventricular (LV) fractional shortening, and increased AF incidence in patients with DDD/R pacing compared to single chamber atrial devices (Reference: "Atrial pacing, the forgotten pacing mode" by M. EL Gamal).
Question 3: Mode Switch in Pacemakers
Functionality:
Mode switch prevents the pacemaker from tracking rapid atrial tachycardias or other rapid signals (which can be artifact).
Reduces adverse hemodynamic effects and patient symptoms.
Historical Context:
Originally, paroxysmal atrial tachycardias were contraindications for DDD devices.
Question 4: ECG Interpretation
Atrial Pacing:
Observations from ECG lead II.
Question 4 (Continued): Additional ECG Interpretation
DDD System:
Identifying both atrial and ventricular pacing in ECG leads (aVR, aVL, V₁).
Question 5: Sensing Issues in Pacemakers
Undersensing:
Occurs when pacemaker fails to detect the intrinsic rhythm, causing asynchronous pacing.
ECG finding: Pacing spikes seen within QRS complexes, or QRS complexes present without clock reset.
Oversensing:
Occurs when non-cardiac signals (like large P/T waves) are incorrectly recognized as intrinsic rhythms, leading to inhibited pacing.
Question 6: Pacing Problems
Output Failure:
Occurs when expected paced stimulus is not generated, leading to reduced or absent pacing function.
Causes include oversensing, wire fracture, lead displacement, and external interference.
Failure to Capture:
Occurs when the pacing stimulus does not depolarize cardiac tissue (spike without QRS complex or P wave).
Causes include electrode displacement, wire fracture, electrolyte disturbances, myocardial infarction.
Question 7: Issues Caused by Pacemakers
Pacemaker-Mediated Tachycardia (PMT):
A re-entry tachycardia involving antegrade conduction via the pacemaker with retrograde conduction through the AV node.
Results from retrograde P waves being detected as intrinsic activity leading to paced ventricular contractions, causing a continuous cycle of tachycardia.
Pacemaker Syndrome:
Arises from poor timing between atrial and ventricular contractions, leading to AV dyssynchrony.
Symptoms include fatigue, dizziness, palpitations, pre-syncope, and significant BP drops during rhythm changes.
Lead Displacement Dysrhythmia:
Occurs when a pacing lead dislodges, causing irregular stimulation of the myocardium (ventricular ectopics or VT).
Diagnosis confirmed via chest x-ray.
Question 8: ECG Analysis
Observation:
A ventricular paced rhythm with intermittent failure to capture is noted.
Atrial sensing appears operational (spikes follow each P wave) but there is a complete heart block or high-grade 2nd degree AV block as intrinsic P waves don’t capture ventricles.