Unit 04 Pt3
Overview
Focus on vesicle fusion with target compartments.
Key compartments: Endoplasmic Reticulum (ER) and Golgi Apparatus.
Vesicle Fusion Mechanism
SNAREs in Vesicle Fusion
SNAREs (Soluble NSF Attachment Protein Receptors) facilitate vesicle fusion.
V-SNARE (vesicular SNARE) on transport vesicle and T-SNARE (target SNARE) on target membrane.
T-SNARE depicted in blue; V-SNARE in red.
They intertwine creating a helical structure to facilitate fusion.
Proximity Requirement
Requires 1 to 1.5 nm distance between membranes for fusion.
Close proximity allows lipid flow and excludes water, facilitating fusion.
Fusion Process Steps
Trans SNAREs utilize energy to bring membranes closer together.
Experimental observations show slow fusion when V-SNAREs mix with T-SNAREs in liposomes.
Inner membranes form a stalk facilitating lipid flow between them.
SNAREs interact with RAB effector proteins to promote fusion.
Delayed Fusion & Regulated Exocytosis
Delayed fusion noted in regulated exocytosis (e.g., neurotransmitter release).
Neurotransmitter release at synaptic junctions is crucial and regulated by SNAREs.
Pathological Implications of SNARE Proteins
Botulinum and Tetanus Toxins
Clostridium botulinum toxin destroys SNAREs, inhibiting neurotransmitter release.
Clostridium tetani also targets SNAREs affecting motor reflexes.
Resetting SNARE Proteins
Post-fusion: SNARE proteins must be reset for new transport cycles.
Requires ATP hydrolysis and proteins like NSF (N-ethylmaleimide-sensitive factor).
Biological Processes Requiring Membrane Fusion
Membrane fusion is crucial for various biological processes such as:
Fertilization
Muscle fiber formation
Viral entry into cells.
Viral entry involves virus fusing with cell membranes, and new virus particles budding off.
Golgi Apparatus Overview
Important site for carbohydrate synthesis and protein sorting.
Proteins transition from ER to Golgi via COPII-coated vesicles.
Proteins sorted and directed outwards via secretory vesicles.
Cargo Transport Mechanisms
Selectivity and Exit Signals
Research evolved understanding of selectivity via exit signals.
Proteins vary in packaging and exit rates based on folding efficiencies.
Chaperone Proteins
Chaperones like BiP help in protein folding and masking exit signals.
Misfolded proteins are degraded via proteasome.
Transport Vesicles and Retrograde Transport
Homotypic membrane fusion can occur allowing for vesicular tubular clusters (VTC).
These clusters transport cargo either to the plasma membrane or back to ER via COPI-coated vesicles (retrieval).
KDEL Retrieval Signal
KDEL sequence on soluble proteins aids in their retention in the ER.
KDEL receptors on the Golgi membranes facilitate retrieval of soluble ER proteins.
C-terminal KKXX signals characterize ER membrane resident proteins.
Structure of the Golgi Apparatus
Composed of flattened stacks of cisternae (10-20 stacks).
Has distinct cis face (near ER) and trans face (near plasma membrane).
Post-Translational Modifications
Cargo undergoes modifications while transitioning through the Golgi.
Enzymatic activities localized at different regions of the Golgi ensure proper processing.
Sorting of proteins is vital for their function and localization.