Psychology 20 - Sedative Hypnotics Lecture Notes
Sedative Hypnotics Overview
Sedative hypnotics are a class of drugs primarily used for:
Treatment of anxiety (anxiolytics).
Managing insomnia (historically more common).
Controlling epilepsy by suppressing neuronal excitability.
Historical Context
High prevalence of anxiety in the population has led to a need for anxiolytic drugs.
Alcohol was one of the earliest substances used to reduce anxiety.
Alcohol is not a suitable anxiolytic due to significant side effects, including:
Behavioral impairment
Strong physical dependence and withdrawal symptoms.
Early sedative hypnotics include chloral hydrate, synthesized in the early 1800s.
First extensively used, now associated with recreational abuse and physical dependence.
Historical references include:
"Quarterly Journal of Inebriety"
Usage as "knockout drops" or Mickey Finn, leading to inappropriate drugging.
Drug Classes
Two main classes of anxiolytics and sedatives:
Barbiturates
Oldest class
Parent compound synthesized by Adolphe von Baeyer in 1864 through:
Combining malonic acid (found in apples) with urea (found in urine).
Initial compound: barbituric acid, not effective until modified.
First effective barbiturate: Diethylbarbituric acid (trade name: Veranol).
Derived from barbituric acid with two diethyl groups added.
Named after the peaceful city of Verona.
Benzodiazepines
Emerged in the 1960s.
Classification of Barbiturates
Classified into categories based on onset and duration of action:
Long-acting: e.g., Phenobarbital
Onset in about 1 hour, duration ~6 hours.
Intermediate-acting: e.g., Amobarbital (trade name Amitol)
Onset ~30 minutes, duration ~6 hours.
Street names include blue angels, blue dolls.
Historically used as a truth serum, but does not guarantee truthful responses.
Short-acting: e.g., Pentobarbital (trade name Nimbutal)
Commonly abused; known street names include yellow jackets.
Ultra short-acting: e.g., Thiopental and Hexobarbital.
Used as presurgical anesthetics. Not commonly abused except for propofol (Milk of Amnesia, involved in high-profile cases like Michael Jackson's death).
Effects and Risks of Barbiturates
All barbiturates produce:
Sedation
Anxiety reduction
Reduced euphoria at lower doses
Interference with REM sleep and potential for REM rebound after discontinuation.
Risks include:
Low therapeutic index (risk of overdose and death).
Severe withdrawal symptoms similar to alcohol:
Increased neuronal excitability leading to convulsions and seizures.
Alcohol and barbiturates act synergistically, heightening the risk of fatal reactions.
Neurochemical Mechanism
Barbiturates enhance GABAergic activity (GABA is an inhibitory neurotransmitter):
Occupy a satellite receptor site on the GABA receptor, prolonging the opening of ion channels.
Reduce glutamate transmission (excitatory neurotransmitter), causing profound amnesia.
Development of Tolerance and Dependence
Tolerance develops rapidly to the reinforcing effects of barbiturates relative to lethal effects, closing the gap between effective and lethal doses:
Cross-tolerance with alcohol exists.
Withdrawal symptoms mirror the drug effects and can lead to severe complications without medical supervision.
Other Anxiolytic Options Developed
Other drugs developed for anxiety treatment in the past during barbiturate popularity included:
Mefenacin Carbonate (not effective as an antibacterial but noted for calming effects).
Meprobamate (trade names Milltown, Equanil) - marketed as a tranquilizer in 1955:
Similar issue with physical dependence as barbiturates but remains in use for anxiety treatment today.
Methaqualone (trade name Quaalude): originally an antimalarial, became popular for its calming effects, linked to significant abuse and notable deaths (including Jimi Hendrix, Elvis Presley).
Conclusion
The barbiturates had their peak use in the 1950s and 60s but declined due to the introduction of benzodiazepines, which had a better safety profile.
Sedative hypnotics continue to be problematic due to their potential for dependence, abuse, and dangerous interactions with other substances (especially alcohol).
The lecture affirms the importance of understanding both historical implications and current medical practices surrounding these medications.