Traumatic Brain and Spinal Cord Injury
OBJECTIVES
- Explain pathophysiology, incidence/prevalence, etiology, risk factors, and mechanisms of injury associated with spinal cord injuries (SCI) and traumatic brain injury (TBI).
- Discuss health promotion and prevention of SCI and TBI.
- Describe focused history and assessment for patients with SCI and TBI.
- Describe laboratory tests and diagnostic procedures used in diagnosis and treatment of patients with SCI and TBI.
- Discuss and prioritize nursing care of patients with SCI and TBI.
- Describe common treatments for patients with SCI and TBI including rationales and nursing implications.
- Compare and contrast stabilization techniques and devices for SCI care.
- Describe potential complications and prevention for SCI and TBI.
- Evaluate patient care based on identified priority problems.
- Describe care transitions to rehabilitation or home; identify healthcare resources for SCI and TBI patients.
ANATOMY OF THE BRAIN AND SPINE
Skull and Brain:
- Components include sphenoid, nasal, ethmoid, zygomatic bones, maxilla, frontal bone, parietal bone, occipital bone, and various sutures.
- Major brain areas:
- Frontal Lobe: Involved in thinking, memory, behavior, movement
- Temporal Lobe: Involved in hearing, learning, feelings
- Parietal Lobe: Responsible for language and touch
- Occipital Lobe: Responsible for sight
- Cerebellum: Balance and coordination
- Brainstem: Controls breathing, heart rate, and temperature
Spine Anatomy:
- Composed of 5 sections:
- Cervical: 7 vertebrae (C1-C7)
- Thoracic: 12 vertebrae (T1-T12)
- Lumbar: 5 vertebrae (L1-L5)
- Sacral: 5 fused vertebrae
- Coccygeal: 3-5 vertebrae
- Protects the spinal cord, which communicates signals between the brain and body via 31 pairs of spinal nerves.
- Functions: motor function, sensory function, and autonomic function (reflexes).
TRAUMATIC BRAIN INJURY (TBI)
Pathophysiology:
- Damage to neuronal tissues from external forces varying from mild to severe.
- Injury levels:
- Mild: Glasgow Coma Scale (GCS) score of 13-15
- Moderate: GCS score 9-12
- Severe: GCS score 3-8
Risk Factors:
- Age, falls, motor vehicle crashes (MVCs), drug/alcohol use, occupation, sports, assaults.
Mechanisms of Injury:
- Blunt: Object strikes head, or head strikes an object.
- Penetrating: High-velocity projectiles, low-velocity objects.
- Blast Injuries: Caused by pressure wave from explosions.
Primary vs Secondary Injuries:
- Primary: Occurs at the moment of trauma (e.g., skull fractures, concussions).
- Secondary: Develops as a result of initial trauma, leading to issues like vascular dysfunction and inflammation.
SKULL FRACTURES
Types:
- Closed: Simple fracture with no skin penetration.
- Compound (Open): Skin laceration present, high risk of infection.
- Depressed: Bone pressed inward into brain tissue, may cause serious injury.
- Basilar: Fracture at base of skull, includes signs like raccoon eyes and CSF leaks.
Management:
- Medical: Pain management, possible antibiotics for infection risk.
- Surgical: Required for compound fractures, severe leaks, or persistent fractures.
CONCUSSION AND CONTUSION
- Concussion:
- Damage without skull break; can cause headaches, dizziness, and confusion; symptoms typically resolve within 72 hours but may last longer in some cases.
- Contusion:
- Bruising of brain tissue; may result in severe symptoms like prolonged unconsciousness and focal neurological signs.
INTRACRANIAL HEMATOMAS/HEMORRHAGE
- Types include:
- Epidural Hematoma: Arterial bleeding leading to loss of consciousness and lucid periods.
- Subdural Hematoma: Venous bleed that often causes delayed symptoms.
- Subarachnoid Hemorrhage: Typically presents as a sudden severe headache.
SPINAL CORD INJURY (SCI)
Definition:
- Damage to spinal cord leading to loss of movement and/or sensation.
Etiology:
- Most often caused by MVCs, falls, violence, sports, and medical/surgical factors.
Incidence: Approximately 17,000 new cases annually in the U.S. with a high prevalence in young males.
Pathophysiology:
- Primary SCI: Direct damage to the spinal cord from injury.
- Secondary SCI: Due to complications like ischemia and inflammation following the primary injury.
COMPLICATIONS OF SCI
- Neurogenic Shock:
- Occurs with SCI above T6, resulting in hypotension and bradycardia.
- Autonomic Dysreflexia:
- Characterized by severe hypertension and bradycardia due to strong stimuli below the injury level.
NURSING MANAGEMENT
- Monitor respiratory function, vital signs, bowel, and bladder management, and skin integrity.
- Interventions for Complications:
- Prevent DVT/PE, manage anxiety/depression, and monitor for signs of infection.
- Educate on health promotion and prevention strategies tailored for SCI/TBI patients.
EMERGENT CARE FOR TBI AND SCI
- Conduct a primary and secondary survey, including respiratory support, IV fluids, and thorough neurological assessments.
- Use of diagnostic tools: CT and MRI as indicated for both TBI and SCI assessments.