Hesi 10: Ch. 31
Drugs Acting on the Autonomic Nervous System
Chapter 31: Adrenergic Antagonists
Adrenergic Antagonists are classified as follows:
Nonselective Adrenergic Blocking Agents
Nonselective Alpha-adrenergic Blocking Agents
Alpha 1 Selective Adrenergic Blocking Agents
Nonselective Beta-Adrenergic Blocking Agents
Beta 1 Selective Adrenergic Blocking Agents
Alpha-Receptors
Alpha-1 Receptors:
Responsible for processes such as:
Vasoconstriction leading to increased peripheral resistance and increased blood pressure.
Contraction of piloerection muscles, pupil dilation, thickened salivary secretions.
Closure of the urinary bladder sphincter and maintenance of male sexual emissions.
Alpha-2 Receptors:
Involvement in the negative feedback control of norepinephrine release from the presynaptic neuron and moderation of insulin release from the pancreas.
Beta-Receptors
Beta-1 Receptors:
Functions include:
Increased heart rate and conduction through the atrioventricular node.
Enhanced myocardial contraction.
Induction of lipolysis in peripheral tissues.
Beta-2 Receptors:
Actions entail:
Vasodilation, bronchial dilation, increased breakdown of muscle and liver glycogen.
Release of glucagon from the pancreas, relaxation of uterine smooth muscle.
Decreased gastrointestinal (GI) muscle tone and activity, reduced GI secretions, and relaxation of the urinary bladder detrusor muscle.
Non-Selective Adrenergic Blocking Agents (Antagonists)
Examples:
Amiodarone, Carvedilol, Labetalol.
Mechanism:
Blocks both alpha and beta receptor sites.
Notably, Labetalol is often mislabeled as a beta blocker as it also blocks alpha-1 receptors.
Actions:
Decreases heart rate (HR), lowers blood pressure (BP), and increases renal perfusion.
Uses:
Labetalol is used for hypertension (HTN).
Amiodarone is utilized in emergencies as part of ACLS drugs and as an antiarrhythmic (discussed in Pharm 3).
Carvedilol is used for HTN and heart failure; it possesses beneficial alpha-blocking activity despite most drugs in this class being contraindicated in heart failure.
Side Effects of Non-Selective Adrenergic Blocking Agents
CNS Effects:
Dizziness, insomnia, depression, fatigue.
GI Effects:
Nausea and vomiting (N&V), diarrhea, anorexia, flatulence.
Cardiac Effects:
Arrhythmias, hypotension, heart failure (HF), and stroke.
Respiratory Effects:
Bronchospasm, cough, rhinitis, bronchial obstruction.
Other Effects:
Decreased exercise tolerance, hypoglycemia, rash, and potential for hepatic failure with carvedilol and labetalol.
Amiodarone may lead to hepatic injury, pulmonary fibrosis, loss of vision, and thyroid issues.
Teaching/Nursing Considerations
Abrupt discontinuation should be avoided; taper dose slowly over 2 weeks to minimize risks of myocardial infarction (MI), stroke, or arrhythmias.
Consult a physician regarding any withdrawal drugs before surgery.
Patient monitoring includes:
Regular monitoring of heart rate and blood pressure.
Monitoring of GI system, particularly increased need for access to the bathroom due to diarrhea.
Observation for signs and symptoms (s/s) of liver failure.
Safety Precautions:
Given the aforementioned side effects and interactions, nurses and patients should be well informed.
Advisable to avoid herbal and alternative therapies.
Nonselective Alpha-Adrenergic Blocking Agents
Phentolamine (Regitine):
Currently, the only drug in this class still in use.
Exhibits specific affinity to alpha receptors.
Mechanism:
Blocking alpha-1 lowers blood pressure.
Blocking alpha-2 causes reflex tachycardia.
Uses:
Most often used for management of extravasation to prevent cell death and tissue sloughing; causes local vasodilation and restoration of blood flow to the area.
Alpha-1 Selective Adrenergic Blocking Agents
Examples: Alfuzosin, doxazosin (Cardura), prazosin (Minipress), silodosin, tamsulosin (Flomax), terazosin.
Characteristics:
Possess specific affinity to alpha-1 receptors.
Induce a decrease in blood pressure without eliciting reflex tachycardia; relax the bladder and prostate.
Uses:
Effective in managing benign prostatic hypertrophy (BPH) as described in Chapter 52.
Side Effects:
Related to alpha-1 blocking activity:
Common issues include hypotension, dizziness, tachycardia, and priapism.
Risk of orthostatic hypotension with the first dose.
Teaching/Nursing Considerations:
Close monitoring of vital signs (VS); implement safety precautions where necessary.
Nonselective Beta-Adrenergic Blocking Agents
Examples: Carteolol, levobunolol, nadolol, nebivolol, propranolol (Inderal), sotalol, timolol.
Mechanism:
These agents block both beta-1 and beta-2 receptors.
Uses:
Primarily employed in treating cardiovascular issues such as hypertension, angina, and preventing reinfarction after myocardial infarction. Also applied for migraine headaches.
Main Therapeutic Effects:
Cardiac Blockade:
Leads to decreased heart rate, contractility, and excitability, contributing to reduced arrhythmias, workload, and oxygen consumption.
Nephron Blockade:
Decreases renin release, subsequently lowering blood pressure.
Propranolol
Characteristics:
Highly effective in blocking all beta receptors. Other medications may offer more selectivity and fewer adverse effects.
Food increases the availability of propranolol while decreasing the absorption of some other drugs within the class.
Propranolol crosses the blood-brain barrier (BBB) while other drugs may not.
Cautions/Contraindications:
Propranolol is contraindicated in patients with bradycardia, heart failure, shock, heart block, bronchospasms, or acute asthma. Caution needed in diabetic, COPD/asthmatic, renal/hepatic dysfunction.
Side Effects:
Related to blockade of beta receptors:
CNS side effects: dizziness, fatigue, sleep disturbances.
Cardiac issues: bradycardia, hypotension.
Respiratory challenges: difficulty breathing, coughing, bronchospasm.
Gastrointestinal effects include nausea, vomiting, diarrhea, and colitis.
Genitourinary effects: decreased libido, impotence.
Other issues such as decreased exercise tolerance and variations in blood sugar levels (hypo/hyperglycemia).
Beta-1 Selective Adrenergic Blocking Agents
Examples: Acebutolol, atenolol (Tenormin), betaxolol, bisoprolol, esmolol, metoprolol (Lopressor).
Characteristics:
Typically do not block beta-2 receptors, thus preserving bronchodilation, making them preferred in patients who smoke or have asthma.
Becomes less selective when doses exceed therapeutic levels.
Uses:
Employed in treating hypertension, angina, and managing some cardiac arrhythmias. They also aid in preventing reinfarction after myocardial infarction.
Teaching/Nursing Considerations:
Abrupt cessation is discouraged; taper doses over a two-week period.
Oral administration of metoprolol with food enhances drug absorption.
Consider the possibility of increased CNS side effects with metoprolol due to its ability to cross the BBB.