Review of Fluids and Electrolytes
Lecture Objectives
Describe factors that influence fluid movement between intracellular and extracellular fluid compartments
Calculate maintenance fluid requirements based on patient-specific information
Recognize common IV fluids utilized in various patients care scenarios
Identify common electrolyte abnormalities related to medication administration
Recommend appropriate treatment for common fluid and electrolyte abnormalities
Body Fluid Compartments
Intracellular Fluid (ICF) - 67%
Transcellular Fluid - 3%
Extracellular Fluid - 33%
Intravascular Space - 25%
Interstitial Space - 75%
Fluid Movement Between Extracellular and Intracellular Compartments
Osmotic pressure determines water distribution
Sodium → Extracellular osmole
Holds water in extracellular space
Potassium → Intracellular osmole
Holds water within the cell
Regulation of cell volume maintained by Na+/K+/ATPase
Movement of Fluid Between Plasma and Interstitial Fluid
Determined by plasma oncotic and hydrostatic pressure
Disruption leads to third spacing/edema
Third spacing = accumulation of excess fluid in interstitium (edema) or in fluid spaces (effusion)
Due to heart, kidney, or liver failure
Excess sodium retention
Anasarca = massive amount of generalized edema
Fluid Deficit
Causes:
Diarrhea, vomiting
NG aspiration
Excessive, unreplenished urinary losses
Excessive insensible losses
Fever, burns, fasting
Sources of loss
Dehydration < 3 days
ECF - 80%
ICF - 20%
Loss from outside cells
Dehydration > 3 days
ECF - 60%
ICF - 40%
Loss from inside cell, must save plasma volume
IV Hydration Solutions

Emergent Needs
Isotonic fluids without dextrose
Normal saline (NS)
Lactated ringers (LR)
Bolus infusion
Adults - 1-2 liters
Pediatrics - 20 mL/kg
Never more than 2 liters
Appropriate IV Hydration Solutions
Adults/pediatric patients - D5NS with or without 20 mEq/L of KCl
Neonates - D10 0.2% NaCl or D10 1/4NS
Fluid Overload
Causes:
Limited renal function, errors in estimating, SIADH
Consequences:
Cardiovascular and pulmonary
Treatment:
Fluid restrictions, diuretics, dialysis/hemofiltration
Maintenance IV Fluids (MIVFs)
Replaces daily urine, fecal, insensible losses
Prevents dehydration, electrolyte disorders, ketoacidosis, protein degradation
Holliday-Segar Method:
Premature infants: 75-120 mL/kg
Term infants: 60-120 mL/kg
3-10 kg: 100 mL/kg
10-20 kg: 1000 mL + 50 mL/kg (for every kg between 10-20)
> 20 kg: 1500 mL + 20 mL/kg (for every kg > 20 kg)
4-2-1 Method:
< 10 kg: 4 mL/kg/hr
10-20 kg: 40 mL/hr + 2 mL/kg/hr (for every kg between 10-20)
> 20 kg: 60 mL/hr + 1 mL/kg/hr (for every kg > 20 kg)
Normal Electrolyte Concentrations
Sodium
Newborn: 133-145 mEq/L
Pediatric: 135-145 mEq/L
Adult: 135-145 mEq/L
Maintenance of extracellular fluid volume + tonicity
Regulation of osmolarity and cell membrane potential
Potassium
Newborn: 4-6.2 mEq/L
Pediatric: 3.4-4.7 mEq/L
Adult: 3.5-5.0 mEq/L
Determination of resting membrane potential
Phosphorous
Newborn: 4.2-9 mEq/L
Pediatric: 4.5-5.5 mEq/L
Adult: 2.5-4.5 mEq/L
Hypokalemia
Increased entry into cells → Elevation of extracellular pH → Increased insulin
Increased GI losses → Vomiting, NG output, diarrhea, ostomy output
Increase urinary losses → Diuretics, metabolic acidosis, hypomagnesemia, medications, polyuria
Medication induced:
Albuterol, loop and thiazide diuretics, corticosteroids, anti-pseudomonal penicillins, insulin, amphotericin B, caspofungin, antivirals
Hyperkalemia
Renal dysfunction
Excessive intake
Cell lysis
Medication-induced:
K+ sparing diuretics
Heparin
Trimethoprim/sulfamethoxazole
Potassium penicillin
ACEi/ARBs
Pseudo-Hyperkalemia
Hemolysis of lab specimen
Hemolysis of blood products given
Improper blood draw → Contaminated sample
Magnesium
Cofactor for many enzyme systems
Muscle contractility
Nerve conduction
Hypermagnesemia
Caused by: excess magnesium intake, renal failure
Symptoms: respiratory paralysis, AV block, PVC, hypotension
Hypomagnesemia
Caused by: GI/renal losses
Symptoms: nausea, dizziness, depression, weakness, convulsions
Medication induced: amphoteracin B, aminoglycosides, loop/thiazide diuretics, cisplatin, cyclosporine, tacrolimus
Calcium
Essential for neuromuscular activity, regulation of endocrine secretory activities, blood coagulation, activation of complement system, bone metabolism
Hypercalcemia
Caused by: cancer, hyperparathyroidism, adrenal insufficiency, immobilization
Symptoms: fatigue, weakness, anorexia, cognitive dysfunction, constipation, kidney stones
Hypocalcemia
Caused by: vitamin D deficiency, hypoparathyroidism, magnesium deficiency
Symptoms: osteoporosis, tetany, muscle cramps, memory loss, confusion, prolonged QT interval
Phosphorous
Main intracellular anion
Important for bone/cell membrane composition, maintenance of normal pH, provision of energy through ATP
Hyperphosphatemia
Caused by: renal dysfunction
Hypophosphatemia
Caused by: starvation, alcoholism, burns, DKA, hyperparathyroidism, diarrhea, long term diuretics, long term aluminum antacids, dialysis
Calcium-Phosphorous Relationship

Calcium Deficiency
Osteoporosis
Severe deficiency
Cardiovascular = hypotension, decreased myocardial contractility, prolonged QT interval
Neuromuscular = distal extremity paresthesias, muscle cramps, tetany, seizures
Phosphorus Deficiency
Neuro: ataxia, confusion, paresthesias
Neuromuscular: weakness, myalgia, rhabdomyolysis
Cardio/Pulmonary: cardiac/ventilatory failure
Hematologic: reduced 2,3-diphosphoglycerate concentration, hemolysis
Consequences of refeeding syndrome
Serum Calcium
Measures bound/unbound calcium → Dependent on albumin status
Corrected Ca2+ = Ca2+ + ((4 - albumin) x 0.8))
Biologically active form = ionized Ca2+ - free calcium