NSC 330: Antidepressants
Study Notes for NSC 330: Antidepressants
Depression
Definition: Depressed mood and loss of pleasure/interest in usual activities
Symptoms: Must be present most of the day, nearly every day, for at least 2 weeks
-Hopelessness or sadness
- Lack of interest in activities
- Changes in sleep patterns (loss of sleep or excessive sleep)
- Weight changes (loss/gain)
- Feelings of fear/anxiousness
- Lack of self-worth
- Agitation
- Unexplained physiologic symptoms
- Suicidal ideation
- Behavior changes
- Anxiety
- Panic attacks
- Insomnia
- Irritability
- Hostility
- Impulsivity
- Mania
Pathophysiology of Depression
Complex Factors: Deficiency of neurotransmitters, particularly:Norepinephrine, Serotonin, Dopamine, Acetylcholine, Glutamate
Pharmacologic Treatment
General Note: Antidepressants are not immediately effective.
Mechanism of Action (MOA) of Antidepressants:
1. Prevent reuptake of neurotransmitters, thereby increasing the concentration in CNS synapses.
- Reuptake Inhibitors: Prevent the reuptake process
- Alters the number and sensitivity of receptors
2. Remaining neurotransmitters are metabolized by the enzyme monoamine oxidase (MAO).
- MAOI Inhibitors: Prevent this metabolism, increasing neurotransmitter availability for receptor binding
General Considerations for Antidepressants
Response Times:Initial responses can take 1-3 weeks
- Maximal responses may take up to 12 weeksBoxed Warning: Increased risk of suicidal thoughts and behavior
- Most likely to occur in the first 2 months, especially with dosage increase
- Higher risk in children, adolescents, and young adults (under age 24)
- Be vigilant for an energizing effect that may precede depressive symptom remission which could lead to suicide
Classes of Antidepressants
1. Tricyclic Antidepressants (TCAs)
2. Selective Serotonin Reuptake Inhibitors (SSRIs)
3. Serotonin Norepinephrine Reuptake Inhibitors (SNRIs)
4. Monoamine Oxidase Inhibitors (MAOIs)
5. Atypical Antidepressants
Tricyclic Antidepressants (TCAs)
Examples:
1. Amitriptyline
2. ImipramineMOA: Blocks reuptake of serotonin and norepinephrine; also blocks histamine and acetylcholine (causing increased adverse effects)
Uses:
1. Depression
2. Anxiety disorders
3. OCD
4. Panic disorder
5. Insomnia
6. Neuropathic pain (chronic pain)
Adverse Effects of TCAs
1. Weight gain
2. Sedation (due to histamine receptor blockage in CNS)
3. Anticholinergic effects (blurred vision, urinary retention, dry mouth, constipation, confusion)
4. Tachycardia and potential prolongation of QRS and PR/QT intervals
5. Lowered seizure threshold
6. Orthostatic hypotension
Nursing Implications for TCAs
1. Caution with other CNS depressants or medications with anticholinergic properties
2. Instruct patients that therapeutic effects may not be seen for 3-4 weeks
3. An overdose is potentially fatal (8 times the average dose)
Selective Serotonin Reuptake Inhibitors (SSRIs)
MOA: Inhibit serotonin reuptake, thereby increasing the availability of serotonin and prolonging the stimulatory potential of receptors
Uses:
1. Depression (MDD, bipolar depression)
2. Anxiety, OCD, panic disorder, social phobia, PTSD, PMDD (premenstrual dysphoric disorder)
3. BulimiaExamples:
1. Fluoxetine
2. Paroxetine
3. Sertraline
4. Citalopram
5. Escitalopram
6. Vortioxetine (newer with fewer side effects)
Adverse Effects of SSRIs
1. Headache and/or nausea (initially)
2. Some SSRIs are sedating (not all)
3. Sexual dysfunction (up to 70% with some SSRIs)
4. Early weight loss followed by potential weight gain (varies by individual)
5. Withdrawal symptoms if abruptly discontinued (suggest tapering over 4 weeks)
Advantages of SSRIs
1. No cardiac adverse effects
2. No anticholinergic adverse effects
3. Expect 3-4 weeks to observe full therapeutic effectiveness
4. Monitor for suicidal thinking/behavior during the initial 2-3 weeks
SSRIs Discontinuation Syndrome
1. Do not abruptly discontinue SSRIs
2. Symptoms may persist for 1-3 weeks unless the drug is restarted
Serotonin Syndrome
Definition: A potentially serious side effect characterized by excessive serotonergic activity
Onset: Begins 2-72 hours after dose increase or addition
Resolution: Resolves spontaneously when the drug is stopped
Higher Risk: When used with other serotonergic drugs like MAOIs, TCAs, lithium, linezolid, and St. John's Wort
Serotonin Norepinephrine Reuptake Inhibitors (SNRIs)
MOA: Increase both serotonin and norepinephrine at the synapse
Uses:
1. Major Depressive Disorder
2. Bipolar Disorder
3. Anxiety Disorders
4. PTSD
5. Neuropathic PainExamples:
1. Venlafaxine
2. Duloxetine
SNRIs Considerations
1. Expect a full therapeutic effect in 3-4 weeks
2. Monitor for suicidal thoughts/behavior during the initial 2-3 weeks
Adverse Effects of SNRIs
1. Same as SSRIs but with added hypertension risk
2. More intense withdrawal symptoms requiring slower tapering (over 4 weeks)
Monoamine Oxidase Inhibitors (MAOIs)
MOA: Inhibit monoamine oxidase enzyme, increasing levels of serotonin, norepinephrine, and dopamine
Uses: For drug-resistant or atypical depression; less desirable due to significant drug and food interactions and poor safety profile
Examples:
1. Isocarboxazid
2. Phenelzine
3. Tranylcypromine
4. Selegiline patch
Dietary Considerations with MAOIs
Tyramine: A monoamine precursor of norepinephrine, deactivated by MAO in the GI tract and liver
- Dietary tyramine typically doesn't reach circulation, but MAOIs prevent its metabolismAvoid Foods:
1. Aged cheeses (cheddar, Swiss, bleu)
2. Beer
3. Red wines
4. Smoked meats
5. Fermented sausages (pepperoni, salami, bologna)
6. Soy sauce
7. Sour creamConsequences of Ingestion: May cause hypertensive crisis with symptoms such as severe headache, hypertension, tachycardia, palpitations, nausea/vomiting, confusion, profuse sweating, and potentially lead to stroke or death.
MAOIs General Considerations
Onset: 1-2 weeks
Interactions: Numerous drug-drug interactions
Cautions: Should not be used in impulsive patients, cognitively impaired individuals, or those unable to follow dietary restrictions
Adverse Effects of MAOIs
1. Dizziness
2. Orthostatic hypotension
3. Insomnia
4. CNS stimulation (anxiety, agitation)