imunology lesson

Terminology

  • Anaphylaxis: A severe and possibly fatal allergic response.
  • Antigen: Cells, organisms, or structures on a cell membrane that are foreign to the body. In a healthy animal, the presence of an antigen will stimulate an immune response.
  • Antibody: Also known as immunoglobulins; proteins produced by plasma cells in response to antigens. Antibodies are specific to a certain antigen.
  • Autoimmune Disease: An abnormal disease process where the body identifies its own cells as foreign and starts an immune response.
  • Immunization: The process of stimulating an immune response by administering (or inoculating) an animal with a vaccine. The immune response is similar to the response that would occur through natural disease.

Overview

  • Immunology: The study of the immune system.
  • Main Function of the Immune System: To protect the body from damage or disease by identifying self and non-self. Once a non-self or foreign invader is identified, the body can utilize various methods to remove or destroy the invader, including:
    • Phagocytosis and destruction of foreign cells.
    • Lysis of foreign cell membranes.
    • Inactivation of pathogenic organisms or chemical substances.
    • Precipitation or clumping of cells or molecules.
  • Excessive Reaction: If the reaction to a foreign invader is excessive, it can injure the body's cells as well as the invader, resulting in anaphylaxis.
  • Autoimmune Diseases: Occur when the body mistakenly identifies self as non-self.

Innate (Non-specific) Immunity

  • Definition: Strives to eliminate all threats to the body and is not specific to any one type of threat.
  • **Examples of Innate Immunity:
    • Skin: Physical barrier against microbes.
    • Saliva and Gastric Acid: Destroys/inhibits bacteria and viruses.
    • Tears: Rear alike its function in destroying/inhibiting bacteria and viruses.
    • Turbinates/Mucociliary Escalator: Cells lining the respiratory tree are covered with cilia and mucus; these trap microorganisms and prevent them from invading lung tissue.
    • Bodily Functions: Sneezing, urination, and coughing work to expel pathogens.
    • Inflammation:
    • Inflammatory mediators are proteins produced by damaged cells, white blood cells, and complement.
    • They cause blood vessel dilation, increase blood flow to affected areas, and make vessel walls "leaky" to allow plasma and white blood cells into tissues.
    • Induce fever to inhibit pathogen growth.
    • Phagocytosis: Cells such as neutrophils, macrophages, monocytes, and dendritic cells engulf and digest pathogens.
    • Natural Killer (NK) Cells: Circulate through the body, seeking abnormal proteins that may indicate sick or cancerous cells for destruction.
    • Interferon: Inhibits virus growth and modulates the immune system.
    • Complement Cascade: A group of proteins that circulate in inactive forms and are activated upon encountering a pathogen. Functions include:
    • Opsonization: Coats pathogens to facilitate phagocytosis by macrophages.
    • Mast Cell Activation: Induces release of histamine and amplifies inflammation.
    • Membrane Attack Complex (MAC): Causes rupture of pathogen cell membranes.

Adaptive (Specific) Immunity

  • Definition: Immunity designed to target specific antigens and can be referred to as acquired immunity.
  • Interaction with Innate Immunity: Specific immunity collaborates with innate immunity, relying on both systems for optimal function.
  • Components of Specific Immunity: Two main responses:
    • Cell-Mediated Immunity:
    • Involves T-cells (lymphocytes) that react to antigens presented by Antigen Presenting Cells (APCs), which often are macrophages and dendritic cells.
    • Functions include:
      • Cytotoxic T-cells: Destroy cells displaying the antigen.
      • Helper T-cells:
      • Release cytokines to stimulate migration of white cells and enhance macrophage function.
      • Assist B-cells in activating to produce antibodies.
      • Guide NK cells.
      • Suppressor T-cells: Reduce antibody formation and immune response to prevent autoimmune diseases and assist in immune memory by preventing a rapid response that could overwhelm the immune system.
      • Memory Cells: Long-lasting T cells that respond more rapidly to subsequent exposures to antigens.
    • Humoral Immunity:
    • Involves B-cells that bind to antigens either directly or with assistance from Helper T-cells.
    • Upon activation, B-cells can:
      • Differentiate into plasma cells generating antibodies.
      • Phagocytize pathogens and present antigens to Helper T-cells.
    • Memory cells from B-cells also persist in circulation for rapid response upon re-exposure to the antigen.

Antibodies

  • Definition: Proteins produced by B-cells, acting in immune response by binding to specific antigens.
  • Classes of Antibodies:
    • IgG: Most prevalent antibody; crosses the placenta and forms ~75% of circulating immunoglobulins. Produced after IgM, plays a crucial role in colostrum.
    • IgM: First antibody formed in response to infection and by newborns; constitutes about 5% of immunoglobulins.
    • IgA: Predominantly in mucosal areas (saliva, tears, mucus); represents ~20% of immunoglobulins, important for mucosal barriers.
    • IgE: Found mainly in basophils and mast cells; involved in allergic and parasitic responses, present in small amounts.
    • IgD: Function poorly understood; present on B-cell surfaces in trace amounts.
  • B-cells' Flexibility: B-cells can produce different types of antibodies and even switch types during infection.

T and B Cell Origins and Locations

  • Naming Origin:
    • T-cells: Named for their maturation in the thymus.
    • B-cells: Named after the Bursa of Fabricius in birds where they were first identified; in mammals, they originate from the bone marrow.
  • Location in Body:
    • T-cells: Predominantly circulate in blood.
    • B-cells: Mostly reside in lymphatic organs (lymph nodes, spleen, tonsils).

Immune System Function Overview

  • The immune system ensures proper distinction between self and non-self, crucial in avoiding autoimmunity.

Hypothetical Pathogen Interaction

  • Analogy: A scoop of ice cream covered with various toppings represents a pathogen.
    • Antigen Examples:
    • Sprinkles, peanut pieces, and cherries may serve as antigens.
    • Epitopes: Different variations on these toppings represent epitopes.

White Blood Cell Interaction with Pathogens

  • Recognition of Self vs Non-Self: White blood cells must identify self to prevent autoimmunity.
  • Epitope Specificity of T-cells and B-cells: Each T-cell and B-cell is specific to a single epitope, activated only upon recognizing that epitope.

Immune Response Scenario: "Darby and the Ice Cream Sundae"

  • Initial Exposure: "Darby", a puppy, encounters a vanilla ice cream sundae (pathogen).
  • Innate Immune Response:
    • Mucus traps sundae particles; irritation promotes inflammation.
    • Macrophages phagocytize the sundae and present its antigens.
    • Neutrophils and T-cells respond to chemical signals from inflamed tissues.
  • Specific Immune System Activation:
    • Activated T-cells differentiate into various functional types (helper, cytotoxic, memory, suppressor).
    • Helper T-cells attract B-cells that recognize the same antigens, leading to antibody production, primarily IgM first and then IgG.
    • Antibodies attach to pathogens, aiding in their destruction and creating easier targets for phagocytes.
  • Regulation by Suppressor Cells: Once the infection is controlled, suppressor cells help regulate and reduce the immune response.
  • Memory Cell Formation: T-memory and B-memory cells circulate and provide faster responses upon future encounters with the same pathogen.

Passive Immunity

  • Definition: Immunity obtained from an external source, such as antibodies from the mother.
  • Colostrum: Vital early nursing liquid for newborns, providing necessary antibodies against known pathogens, requiring timely consumption post-birth (1-18 hours).
  • Hyperimmunized Serum: Treatment using serum rich in antibodies against a specific illness can enhance recovery (e.g., parvovirus).
  • Antitoxins and Antivenoms: Provide antibodies against specific toxins or venoms, used in emergencies.

Active Immunity

  • Definition: Immunity developed after exposure to pathogens, through natural infection or vaccination.
  • Vaccine-Induced Immunity: Simulated responses to non-pathogenic versions of organisms promote memory development for future encounters.

Vaccine Types

  • Core vs Non-Core Vaccines:
    • Core vaccines are recommended universally based on species and age.
    • Non-core vaccines are tailored to individual animal needs.
  • Infective Vaccinations: Mimic real pathogens but do not cause disease; stimulate a robust immune response but carry a small risk of reverting.
  • Non-infective Vaccinations: Do not invade host cells; often require adjuvants to bolster immune response, but may be linked to inflammatory issues.

Vaccine-Related Terms

  • Adjuvanted: Vaccines with added adjuvants.
  • Non-adjuvanted: Vaccines without adjuvants.
  • Attenuated: Modified organisms that do not cause disease, could be fully or partially killed.
  • Recombinant: Vaccines modified at the DNA level to enhance immune responses.
  • Sub-unit: Composed of purified segments of pathogens rather than whole cells.
  • Vectored: Utilize harmless viruses carrying genetic material from pathogenic organisms to stimulate an immune response.