Neuroscience 2610: Chapter 4.3: Generation and Propagation of the Action Potential
action potential
large, brief reversal in the polarity of an axon membrane
occurs when a large concentration of Na+ first and the K+ crosses the membrane rapidly
depolarization phase of the action potential is due to Na+ influx
hyperpolarization phase of the action potential is due to K+ efflux
threshold potential
action potential is triggered by the opening of Na+ and K+ voltage activated channels
membrane charge undergoes a remarkable change with no additional stimulation
if an axon memebrane is stimulated electrically while the solution surrounding the axon contains the chemical TEA
the result is a smaller than normal ion flow due to an Na+ influx
if an axon’s membrane is stimulated electrically while the solution surrounding the axon contains TTX
the result is a slightly different ion flow due to the efflux of K+
voltage activated channels
gated potential channel that opens or closes only at specific membrane voltages
closed when an axon’s membrane is at its resting potential, ions can’t pass through them
when the membrane reaches the threshold voltage, they open briefly which enable ions to pass through then close again to restrict the flow
dendrites and dendritic branches do not have many voltage activated channels and do not produce action potentials
inputs close to the initial segment are much more influential than those occurring some distance away and inhibitory as well, creating IPSPs
giant depolarizing potentials
some cells in the developing hippocampus can produce addition action potentials when the cell would ordinarily be refractory
back propagation
reverse movement of an action potential into the soma and dendritic field of a neuron
plays a role in the plastic changes that underlie learning
optogenetic
transgenic technique that combines genetics and light to excite or inhibit targeted cells in living tissue
ChR2
light activated channel that absorbs blue light
opens briefly to allow the passage of Na+ and K+
the resulting depolarization excites the cell enough to generate action potentials
NpHR
light driven ion pump fr Cl- ions
found in archaea or halobacteria
when illuminated with green-yellow light, the NpHR pumps Cl- anions into the cell, hyper polarizing if and inhibiting its activity
absolutely refractory
state of an axon in repolarizing period, during which a new action potential cannot be elicited because gate 2 of Na+ channels is closed
unable to produce an action potential while its repolarizing
relatively refractory
state of an axon in the later phase of an action potential, during which higher intensity electrical current is required to produce another action potential
a phase during which K+ channels are still open
nerve impulse
propagation(to give birth) of an action potential on the membrane of an axon
total voltage change during an action potential is 100 mV
membrane’s resting state is -70mV
action potential threshold levels is -50mV
an action potential does not dissipate
either generated completely or not generated at all
refractory periods
determined by the positions of the gates that mediate ion flow in the voltage activated channels
the action potential’s refractory phase has 2 uses for nerves that are conducting information
the maximum rate at which action potentials can occur is about 200 per second, the sensitivity of voltage activated channels affects firing frequency
refractory periods prevent the action potential from reversing direction and returning to its point of origin
action potential cannot occur where myelin is wrapped around an axon because the myelin is an insulating barrier that blocks ionic current flow
glial cells play a role in speeding nerve impulses in the vertebrate nervous system
nodes of ranvier
tiny gaps in the myelin sheath that is not covered in myelin
myelin has 2 important consequences for propagating action potentials
propagation becomes energetically cheaper, since action potentials are regenerated only at the nodes of ranvier and not along the axon’s entire length
myelin improves the action potential’s conduction speed
multiple sclerosis(MS)
disorder resulting from the loss of myelin sheath produced by oligodendroglia around axons in the CNS
initial symptoms
loss of sensation in the face, limbs or body
loss of control over movements
or both
autoimmune diseases
results from the loss of the immune system’s ability to discriminate between foreign pathogens in the body and the body itself
the cell body does not contain voltage activated ion channels but if summed inputs excite the initial segment to a threshold level, action potentials are triggered and then propagated as they travel a long the axon as a nerve impulse
nerve impulses travel more rapidly on myelinated axons because of saltatory conductions
saltatory conduction
action potentials leap between the nodes separating the glial cells that form the axon’s myelin sheath