Pharmacodynamics
What is Pharmacodynamics?
Pharmacodynamics is the study of the effects of drugs on the body. Based on the drug response, it can cause a primary and/or secondary physiologic effect. A primary effect is the desired response and the secondary can be desired or undesired.
Drug-Dose Relationship
- When a drug's dose can no longer increase to reach the desired therapeutic response, it is known as maximal efficacy
- The therapeutic dose of a drug (ED50) is the dose that produces a therapeutic response in 50% of the population
- The toxic dose (TD50) is the dose that produces a toxic response in 50% of the population
- The therapeutic index is the difference between the ED 50 and TD 50
- Drugs with a narrow therapeutic index means that they only need a small increase in dosage for a therapeutic dose to be come toxic and the patient requires close monitoring if they are on these drugs
- E.g. Warfarin, Digoxin, Phenytoin
Onset, Peak, and Duration of Action
- Onset is the minimum time it takes for a drug to reach the minimum effective concentration (MEC)
- A peak occurs when the drug reaches its highest concentration in the blood
- Duration of action is the amount of time that a drug exhibits a therapeutic effect
Peak Times
- Oral: 2-3 hours
- IV: 15-60 minutes
- IM: 2-4
- SQ: 15 minutes- several hours
- Sublingual/Buccal: 10-15 minutes
Agonist, Partial Agonists, Antagonists
- Agonists are drugs that activate receptors and produce a desirable response
- Partial agonists produce moderate activity when bound to receptors
- Partial agonists also prevent other drugs from binding and activating responses
- Drugs that prevent receptor activation and block a response are called agonists
Nonspecific and Nonselective Effects
- Many agonistic and antagonistic drugs lack specific and selective effects so they can affect many systems (nonspecific) and receptors (nonselective)
- E.g. Cholinergic receptors are located in the bladder, heart, eyes, stomach, blood vessels, and bronchi
- A cholinergic agonist prescribed for a specific condition with the bladder will also effect the other organs with cholinergic receptors (nonspecific)
- E.g. Epinephrine used to treat anaphylaxis will target alpha1, beta1, and beta 2 receptors (blood vessels, heart, and bronchus) and cause increased blood pressure, increased HR, and dilation and relaxation of the bronchioles (nonselective)
Mechanisms of Action
There are 7 mechanisms of action: stimulation, depression, irritation, replacement, cytotoxic action, antimicrobial action, and modification of immune status
- Drugs that stimulate enhance natural activity
- E.g. Adrenergics
- Depressants decrease neural activity and bodily function
- E.g. Barbiturates and Opiates
- Irritators have a noxious or unpleasant effect
- E.g. Astringents
- Replacement drugs replace essential body compounds
- E.g. Insulin, thyroid drugs, hormones
- Cytotoxic drugs selectively kill parasites and cancers
- Antimicrobials prevent, inhibit, or kill infectious organisms
- Drugs that modify immune status enhance or depress the immune system
- E.g. Interferons and Methotrexate
Side Effects, Adverse Effects, and Toxicity
- Side effects are expected secondary effects of drug therapy
- Chronic illness, sex, weight, and ethnicity play a part in side effects
- The occurrence of side effects is one of the primary reasons patients stop taking prescribed medication
- Adverse drug reactions (ADRs) are unintentional, unexpected reactions to therapy that occur at normal drug dosages
- ADRs are always undesirable and must be reported and documented
- Drug toxicity occurs when drug levels exceed the therapeutic range
- Factors that influence drug toxicity include disease, genetics, and age
- Tachyphylaxis refers to an acute, rapid decrease in response to a drug
- It may occur after the first or several doses of a drug
Drug-Drug Interactions
A drug interaction is defined as an altered or modified action or effect of a drug as a result of interaction with one or multiple drugs. Interactions can be divided into 2 categories: pharmacokinetics and pharmacodynamic.
- Patient factors that increase risk for drug interactions:
- Chronic illnesses
- Use of multiple medications
- See more than one provider
- Use multiple pharmacies
- Older age
- 20% of older adults take 5 or more medications
- The most common symptoms of drug-drug interactions are nausea, vomiting, heartburn, and lightheadedness
- The most feared interactions would cause a dramatic drop in BP or cause a rapid or irregular heartbeat
- Interactions that produce toxins that may damage vital organs need to be closely monitored
Pharmacokinetic Interactions
Pharmacokinetic interactions are changes in absorption, distribution, metabolism, or excretion.
Absorption
- A drug can block, decrease, or increase the absorption of another drug
- This can be accomplished by altering gastric emptying time, changing gastric pH, or by forming drug complexes
- Most drugs are absorbed in the small intestine with exceptions being barbiturates, salicylates, and theophylline, which undergo gastric absorption
- Opioids and respiratory anticholinergics decrease gastric emptying time and GI motility, which increases absorption rate
- When gastric pH is lowered, weakly acidic drugs are less ionized and are absorbed
- Drugs that raise gastric pH decrease the absorption of weak acid drugs
- Alterations in gut bacterial fauna can affect pharmacokinetics
Metabolism
- Drugs that inhibit or stimulate hepatic metabolic enzymes can alter the metabolism of drugs
Drug Induction
- Drugs that promote the activity of liver enzymes are known as enzyme inducers
- The use of enzyme inducers promote drug elimination and decreased drug concentrations in the blood
- Onset and termination of inducers are slow (approximately 1 week)
- Dosages may need to be increased with the use of an inducer
- Monitor serum drug levels if the drug has a narrow therapeutic range
- Chronic cigarette smoking and alcohol consumption lead to an increase in hepatic enzyme activity
Drug Inhibition
- Drugs that inhibit liver metabolic enzymes are known as enzyme inhibitors
- Enzyme inhibitors decrease metabolism of the drug and increase its blood concentration
- To avoid toxicity due to decreased metabolism, the dosage used must be reduced
- Inhibitor drug onset is rapid increases the half-life of the target drug which can prolong drug effect
- Monitor serum drug levels if the drug has a narrow therapeutic range
- Acute alcoholism and flavonoids inhibit metabolism
- Flavonoids inhibit certain drugs
- Carbamazepine, Calcium Channel Blockers, and drugs for ED
- Flavonoids are found in the juice and pulp of citrus fruits
#### Excretion
- Drugs that affect renal function can increase or decrease renal excretion of drugs
- The use of diuretics cause an increase in urinary frequency, which can lead to increased excretion
- Some diuretics decrease the reabsorption of potassium and can cause [[Hypokalemia]]
- Hypokalemia can alter the action of some drugs
- E.g. Hypokalemia enhances the action of digoxin and lead to toxicity
- 2 or more drugs that undergo the same route of excretion may compete with one another for elimination from the body
- Alkaline urine promotes the excretion of weak-acid drugs and acidic urine promotes the excretion of weak-base drugs
- Ionized drugs have a lower rate of reabsorption by the kidneys
- With patients with decreased hepatic or renal function, there is usually an increased in free drug levels and need close monitoring when taking multiple drugs
- Therapeutic drug monitoring (TDM) is important for drugs that have narrow therapeutic ranges
- E.g. Digoxin and Phenytoin
Pharmacodynamic Interactions
Pharmacodynamic interactions are those that result in additive, synergistic, or antagonistic drug effects
Additive
- When 2 drugs are administered in combination, the response is increased beyond what either could produce alone
- An additive interaction is the sum of the effects of the 2 drugs
- Additive effects can be desirable or undesirable
Synergistic
- When 2 or more drugs are administered in combination, one drug can have a synergistic effect on another
- The response is substantially greater than either drug alone
- Different than an additive effect in that 1 drug would increase the abilities of the other rather than simply adding itself to the combination
Antagonistic
- When drugs with antagonistic effects are added together one drug reduces or blocks the effects of the other drug
- Opposite effect of a synergistic interaction