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Lipid Metabolism Overview
Lipid metabolism encompasses the digestion, absorption, transportation, storage, and utilization of lipids in the body.
Digestion and Absorption of Lipids
Lipid Digestion - An Introduction
Dietary lipids: 98% triacylglycerols (TAGs), including fats and oils.
Salivary enzymes: ineffective on lipids (TAGs) as they are water insoluble.
Stomach processing: TAGs physically change into chyme (small globules) through a physical process, not chemical.
Lipid Digestion - The Stomach
Gastric lipase: hydrolyzes TAG ester bonds; about 10% of TAGs hydrolyzed.
High-fat meals prolong gastric retention, increasing satiety.
Lipid Digestion - The Intestinal Cells
Chyme enters the small intestine: emulsified by bile salts.
Pancreatic lipase: hydrolyzes TAGs; normally two out of three fatty acids are released.
Micelles formation: Fatty acids and monoacylglycerols combine with bile salts, allowing for absorption.
Repackaging: Monoacylglycerols and free fatty acids are converted back to TAGs in intestinal cells and packed into chylomicrons.
Lipid Digestion - Bloodstream
Hydrolysis in bloodstream: TAGs are fully hydrolyzed by lipase enzymes.
Fatty acids and glycerol: absorbed as energy (acetyl CoA) or stored as lipids.
Triacylglycerol Storage and Mobilization
Adipose Tissue Overview
Specialized storage: TAGs stored in adipocytes within adipose tissue.
Location: beneath the skin, abdominal region, around vital organs.
Function: insulation, protection, and energy storage.
Hydrolysis of TAGs
Hormonal triggers: cAMP activates hormone-sensitive lipase (HSL) leading to TAG breakdown.
Daily TAG replacement: Average replacement of 10%.
TAGs and Energy Reserves
Energy source: TAGs are the primary stored energy form, surpassing proteins and glycogen.
Glycerol Metabolism
Glycerol in Bloodstream
Conversion: Glycerol to dihydroxyacetone phosphate in liver/kidney via:
Phosphorylation of glycerol.
Oxidation to dihydroxyacetone phosphate.
Oxidation of Fatty Acids
Fatty Acid Breakdown
Three-step process:
Activation by coenzyme A.
Transport to mitochondrial matrix.
Repeated oxidation to form acetyl CoA, FADH2, NADH.
Fatty Acid Activation
Occurs at the outer mitochondrial membrane; involves ATP conversion to AMP.
Fatty Acid Transport
Acyl CoA transported to mitochondrial matrix through a shuttle mechanism.
β-Oxidation Pathway Reactions
Four key reactions repeated to cleave two-carbon units:
Dehydrogenation: Creates a double bond using FAD.
Hydration: Forms a secondary alcohol.
Second dehydrogenation: Oxidizes to a keto group using NAD+.
Thiolysis: Cleaves the fatty acid chain to produce acetyl CoA.
Unsaturated Fatty Acids
Oxidation Process
Requires two extra steps compared to saturated fatty acids (epimerase and cis-trans isomerase).
ATP Production from Fatty Acid Oxidation
Energy Yield Overview
120 net ATP synthesized from complete oxidation of C18 fatty acid (stearic acid), after accounting for activation costs.
Stearic acid produces 2.5 times more energy than glucose.
Ketone Bodies and Ketogenesis
Basic Concepts
Formation: Excess acetyl CoA from β-oxidation converted into ketone bodies under carbohydrate scarcity.
Conditions leading to ketogenesis: high-fat/low-carb diets, diabetes, prolonged fasting.
Key Ketone Bodies
Three main types produced: Acetoacetate, β-hydroxybutyrate, acetone.
Steps in Ketogenesis
Acetyl CoA condenses to form acetoacetyl CoA.
Condensation with a third acetyl CoA produces HMG-CoA.
HMG-CoA is cleaved to acetyl CoA and acetoacetate.
Acetoacetate can be reduced to β-hydroxybutyrate.
Lipogenesis
Overview of Fatty Acid Biosynthesis
Occurs in the cytosol, starting from Acetyl CoA transported via the citrate-malate shuttle.
ACP complex formation: Fatty acid synthesis intermediates linked to carrier proteins.
Chain Elongation Reactions
Involves condensation, reduction, dehydration, and re-addition of hydrogen.
Unsaturated Fatty Acid Production
Requires molecular O2; humans can introduce double bonds only at specific locations, necessitating dietary intake for some essential fatty acids.
Relationship Between Lipogenesis and the Citric Acid Cycle
Contrasts: Diacids in citric acid cycle vs. monoacids in lipogenesis; reverse order of reaction intermediates.
Fate of Acetyl CoA and Cholesterol Synthesis
Routes for Acetyl CoA
Acetyl CoA used for:
ATP production
Formation of ketone bodies
Storage as TAGs
Lipid and fatty acid production.
Role of Cholesterol
Critical for cell membranes & precursor for bile salts, sex hormones, adrenal hormones.
B Vitamins and Lipid Metabolism
Importance of B Vitamins
Act as coenzymes; important B vitamins include:
Niacin (NAD+/NADH/NADPH)
Riboflavin (FAD)
Pantothenic acid (CoA)
Biotin
End of Notes