test 2
Most Likely Test Terms
Know these definitions word-for-word-ish:
Anaerobic training — high-intensity, intermittent exercise.
Motor unit — motor neuron + all muscle fibers it innervates.
Henneman's Size Principle — motor units are recruited from smaller to larger.
Hypertrophy — increase in muscle size/CSA.
Hyperplasia — increase in number of muscle fibers.
Cross-education — training one limb produces an effect in the opposite untrained limb.
Bilateral deficit — bilateral force is less than the combined unilateral force.
Bilateral facilitation — bilateral force is greater than the combined unilateral force.
Bone mineral density — amount of mineral in a given area of bone.
Wolff's Law — bone adapts to mechanical loading.
Overreaching — short-term excessive training that decreases performance.
Overtraining — excessive training over an extended period causing fatigue, illness, injury, and decreased performance.
Overtraining syndrome — prolonged maladaptation from excessive training/inadequate recovery.
Detraining — loss of adaptations due to stopping or substantially reducing training.
Retraining — adaptations returning after training resumes.
⭐ One thing to memorize above everything else:
ANAEROBIC TRAINING → NEURAL + MUSCLE + BONE + CONNECTIVE TISSUE + CV ADAPTATIONS → MORE STRENGTH, POWER & PERFORMANCE
OVERTRAINING → SYMPATHETIC FIRST → PARASYMPATHETIC LATER
1. What is Anaerobic Training?
Anaerobic training = high-intensity exercise performed in short or intermittent bouts.
Examples:
Resistance/weight training
Plyometrics
Sprinting
Speed and agility training
HIIT
Energy systems
Exercise | Main energy system |
|---|---|
Sprint/plyometrics <10 sec | ATP-PC system |
Resistance training/HIIT ~20–30 sec | Anaerobic glycolysis + ATP-PC |
Remember: Anaerobic = high intensity + short duration.
2. Nervous System Adaptations ⭐
The nervous system is one of the first systems to adapt to anaerobic training.
This is why beginners can become stronger before their muscles actually get much bigger.
Main adaptations
A. Increased motor unit recruitment
Your body becomes better at activating more motor units.
More motor units recruited → more force produced.
B. Better Type II fiber recruitment
The nervous system gets better at recruiting Type II muscle fibers, which are important for high force and power.
C. Increased firing rate
Motor neurons send signals more quickly and frequently.
Faster firing → stronger muscle contraction.
D. Better synchronization
Multiple motor units can fire at the same time.
Better synchronization → faster force production → greater power/RFD.
E. Better coordination
Agonist and synergist muscles work together more effectively.
Better coordination → more efficient movement.
Motor Unit
Motor unit = one motor neuron + all the muscle fibers it controls.
Henneman's Size Principle
Motor units are generally recruited from:
Small → medium → large
Small motor units have a lower recruitment threshold.
Large motor units, including those controlling Type II fibers, require more force to be recruited.
Selective recruitment
Experienced athletes can more effectively recruit higher-threshold motor units when they need to produce a lot of force quickly.
3. Neuromuscular Junction Adaptations
The neuromuscular junction (NMJ) is where the nervous system communicates with the muscle.
Anaerobic training can cause:
Larger NMJ area
Longer nerve-terminal branching
More acetylcholine receptors
Faster action potentials
Faster motor-unit firing
Easy way to remember:
Better nerve → muscle communication = better force production.
4. Reflex Adaptations
Muscle spindle
A muscle spindle detects rapid muscle stretch.
When a muscle is rapidly stretched:
Stretch → muscle spindle → contraction → force
Training can increase muscle-spindle sensitivity.
This can improve:
Explosive strength
Power
Rate of force development (RFD)
Golgi tendon reflex
The inverse myotatic reflex/Golgi tendon reflex normally helps inhibit muscle contraction when tension gets very high.
With anaerobic training:
Decreased inhibitory sensitivity → greater force can be produced before inhibition occurs.
This contributes to increased maximal strength.
5. Cross-Education
Cross-education = training one limb causes strength benefits in the opposite, untrained limb.
Example:
You can't train your injured left leg, so you train your right leg.
Your left leg may still maintain some strength because of central nervous system adaptations.
Strength increases of about 8–22% have been observed in the untrained limb.
Remember:
Train one side → nervous system helps the other side.
6. Bilateral Deficit vs. Bilateral Facilitation ⭐
Bilateral Deficit
Bilateral deficit = both limbs together produce LESS force than the combined force of each limb individually.
Example:
Right arm = 60 lb
Left arm = 60 lb
Individually = 120 lb total
Together = less than 120 lb
More common in novice lifters.
To reduce it:
Use more bilateral training.
Bilateral Facilitation
Bilateral facilitation = both limbs together produce MORE force than the combined force of each limb individually.
More common in experienced lifters.
It is associated with increased voluntary agonist activation.
Easy memory:
Deficit = together is worse
Facilitation = together is better
7. Muscular Adaptations ⭐
Hypertrophy
Hypertrophy = increase in muscle size/cross-sectional area (CSA).
Resistance training causes mechanical tension and exercise-induced muscle damage, leading to the addition of contractile proteins within myofibrils.
Type II fibers generally hypertrophy more than Type I fibers.
Hypertrophy results in:
Bigger muscles
Greater force production
Increased strength
Increased power
Hyperplasia
Hyperplasia = increase in the number of muscle fibers.
It can occur through fiber splitting.
However, evidence is strong in animals but inconclusive in humans, and it is thought to have a relatively small effect.
Don't confuse:
Hypertrophy = bigger fibers
Hyperplasia = more fibers
8. Muscle Fiber Type Changes
Muscle fibers exist on a spectrum from less oxidative to more oxidative.
With anaerobic/resistance training:
Type IIx → Type IIa
Type IIa fibers are more fatigue-resistant.
With detraining:
Type IIa → Type IIx
Evidence that Type II fibers completely change into Type I fibers, or vice versa, is not conclusive.
Easy memory:
Training → IIx becomes IIa
Detraining → IIa moves back toward IIx
9. Other Muscle Adaptations
Anaerobic training can cause:
Increased muscle circumference
Increased muscle-fiber size
Increased myofibrillar volume
Increased strength
Increased power
Increased pennation angle
Increased ATP/CP storage
Improved acid-base buffering capacity
Acid-base buffering
High-intensity exercise causes H⁺ accumulation.
Repeated training helps the muscles become better at buffering H⁺.
Better buffering → ability to tolerate high-intensity exercise better.
10. Bone Adaptations ⭐
Bones respond to the mechanical stress placed on them.
Anaerobic training can increase:
Bone diameter
Bone strength
Bone mineral density (BMD)
Bone Mineral Density
BMD = amount of mineral in a given area of bone.
Progressive loading increases BMD.
Inactivity/immobilization decreases BMD.
Wolff's Law
Wolff's Law = bone adapts to the loads placed on it.
More mechanical loading → stronger bones.
Minimal Essential Strain
The minimum amount of strain needed to stimulate bone formation.
It is about 1/10 of the force needed to fracture the bone.
Best exercises for bone adaptation:
Large-muscle, multi-joint, weight-bearing exercises.
Examples:
Squats
Lunges
Deadlifts
11. Connective Tissue Adaptations
Connective tissues include:
Tendons
Ligaments
Fascia
Cartilage
Tendons, ligaments, and fascia
Anaerobic training can increase:
Collagen fibril diameter
Number of collagen fibrils
Density of collagen fibrils
Tendon stiffness
Important:
Connective tissue adapts more slowly than skeletal muscle.
Therefore:
Muscle gets stronger faster than connective tissue can adapt.
Rapidly increasing training loads can cause connective-tissue damage.
12. Cartilage
Training can help maintain cartilage health.
Weight-bearing forces through a full range of motion are important for cartilage viability.
Cartilage doesn't have its own blood supply, so nutrients move through synovial fluid.
Key point:
Moderate-intensity exercise can increase cartilage thickness.
The slides state that strenuous exercise does not appear to cause degenerative joint disease.
13. Cardiovascular Adaptations ⭐
Acute = happens during/immediately after exercise
During resistance/anaerobic exercise:
HR increases
Stroke volume increases
Cardiac output increases
Blood pressure increases
Vascular resistance increases
Muscle contractions can compress blood vessels.
The sympathetic nervous system also causes vasoconstriction in areas that aren't exercising.
Valsalva Maneuver
The Valsalva maneuver increases pressure inside the chest.
It can:
Compress the aorta
Decrease venous return
Increase sympathetic activity/vasoconstriction
This is important during heavy resistance exercise.
14. Reactive Hyperemia
After finishing a resistance-training set:
Muscles relax → blood vessels open → blood flow/venous return increases
This surge in venous return is called reactive hyperemia.
After a set:
HR can increase
BP may decrease
Sympathetic activity decreases
Vasodilation increases
15. Chronic Cardiovascular Adaptations
After consistent training:
Resting blood pressure decreases
Approximately 2–4% for SBP and DBP.
The decrease is greater in people who started with elevated BP.
Resting heart rate decreases
Approximately 4–13%.
Also:
Same workload → lower cardiovascular response after training.
In other words, your body becomes more efficient.
16. Left Ventricular Adaptation
Resistance training creates pressure overload.
This can cause:
Concentric cardiac hypertrophy
Meaning:
Left ventricular wall gets thicker
Little/no change in LV volume
Easy comparison:
Resistance training → pressure overload → thicker wall
Aerobic training → volume overload → larger chamber
17. Pulmonary Adaptations
Acute
During anaerobic exercise:
Respiratory rate increases
Tidal volume increases
These increase significantly after completing a set.
Chronic
With training:
Max exercise:
Increased respiratory rate
Increased tidal volume
Submax exercise:
Increased tidal volume
Decreased respiratory rate
This can make breathing more efficient.
18. Anaerobic Overreaching vs. Overtraining ⭐
Overreaching
Short-term excessive training → temporary decrease in performance.
It isn't necessarily bad.
It can sometimes be intentionally used to "shock" the system and help with adaptation or peaking.
Overtraining
Too much training for too long + not enough recovery → extreme fatigue, illness, injury, and decreased performance.
Common causes:
Too much training
Not enough rest
Not enough recovery
Possibly inadequate nutrition
19. Overtraining Syndrome (OTS) ⭐⭐⭐
Overtraining syndrome = excessive training/recovery imbalance lasting weeks to months that results in prolonged maladaptation.
The key phrase to remember is:
PROLONGED MALADAPTATION
20. Stages of Overtraining Syndrome
This is VERY important for Question #2.
Stage 1: Sympathetic OTS
Occurs earlier.
The sympathetic nervous system becomes overly active.
Think:
OVER-RESPONSIVE
Signs:
↑ Resting HR
↓ HRV
↓ HR recovery
Possible ↑ BP
Stage 2: Parasympathetic OTS
Occurs later.
The parasympathetic nervous system becomes overly active.
Think:
UNDER-RESPONSIVE
Signs:
Lethargy
↓ Resting HR
Blunted HR response during exercise
21. Volume-Related vs. Intensity-Related Overtraining
Volume-related overtraining
Think:
TOO MUCH WORK
Caused by:
Too many sets
Too many exercises
Too many training sessions
Too much overall workload
Not enough recovery
This is a metabolic stressor.
Intensity-related overtraining
Think:
TOO MUCH NEURAL STRESS
Caused by:
Loads that are too heavy
Peaking too often
Too much neural demand
Not enough recovery between heavy sessions
This is a neural stressor.
You can also have a combination of both.
22. Sympathetic vs. Parasympathetic OTS ⭐⭐⭐
Sympathetic OTS | Parasympathetic OTS |
|---|---|
Earlier | Later |
Over-responsive | Under-responsive |
↑ Resting HR | ↓ Resting HR |
↓ HRV | Lethargy |
↓ HR recovery | Blunted exercise HR |
Possible ↑ BP | Reduced HR response |
Super easy memory:
Sympathetic = SPEED UP
Parasympathetic = SLOW DOWN
During OTS:
Sympathetic first → Parasympathetic later.
23. Detraining ⭐
Detraining = loss of adaptations/performance because training is stopped or substantially reduced.
Causes:
Injury
Decreased frequency
Decreased volume
Decreased intensity
Principle of Reversibility
If you stop training, you gradually lose the adaptations you gained.
Important points:
Power decreases relatively quickly.
Muscle CSA decreases quickly without a stimulus.
Type II fibers decrease more.
Fiber-type changes occur more with longer periods of detraining.
Strength is maintained longer in experienced lifters than beginners.
24. Retraining
Retraining = returning to training after a period of detraining.
The good news:
Adaptations usually return quickly once training resumes.
Strength and power can return especially quickly because of neural adaptations.
This supports the idea of muscle memory.