Unit 45 Bio 1201
Chapter 45: Hormones and the Endocrine System
Endocrine Signaling Mechanisms
Endocrine signaling: Hormones are secreted into the bloodstream and affect distant target cells throughout the body.
Paracrine signaling: Hormones are released into the interstitial fluid and affect nearby target cells.
Autocrine signaling: A cell releases hormones that bind to receptors on its own surface, causing direct effects on itself.
Synaptic signaling: Neurons release neurotransmitters into synapses that affect adjacent cells.
Neuroendocrine signaling: Neurosecretory cells release hormones into the blood, affecting distant cells.
Endocrine Glands
Key endocrine glands include:
Thyroid gland
Pineal gland
Hypothalamus
Pituitary gland
Parathyroid glands
Adrenal glands
Ovaries (in females)
Testes (in males)
Pancreas
Hormone Classification
Water-soluble hormones (hydrophilic):
Examples: Polypeptides like insulin, amines like epinephrine.
Mechanism: Bind to receptors on the plasma membrane of target cells.
Lipid-soluble hormones (hydrophobic):
Examples: Steroids like cortisol, amines like thyroxine.
Mechanism: Pass through the plasma membrane and bind to intracellular receptors in the nucleus or cytoplasm.
Action of Water-soluble vs Lipid-soluble Hormones
Water-soluble hormones:
Receptor location: Plasma membrane.
Result: Activation of second messengers inside the target cell leading to a rapid response.
Lipid-soluble hormones:
Receptor location: Nucleus or cytoplasm.
Result: Directly influence gene regulation, leading to long-term changes in cellular function.
Signal Transduction Pathways
Epinephrine (Adrenaline) signaling pathway:
Binding of epinephrine to its receptor activates a G protein.
G protein activates Adenylyl cyclase, converting ATP to cAMP (cyclic AMP), which acts as a second messenger.
cAMP activates Protein Kinase A (PKA), which then phosphorylates target proteins.
Different effects on different target cells:
Liver cells: Glycogen breaks down to glucose, releasing it into the bloodstream.
Smooth muscle cells in blood vessels: Varying responses to epinephrine binding (relaxation vs contraction).
Local Regulators
Types of local regulators include:
Cytokines and growth factors
Nitric oxide (NO)
Prostaglandins
Endocrine Pathways
Simple endocrine pathway example:
Stimulus: low pH in the duodenum.
Secretin released by: Endocrine S cells of the duodenum into circulation.
Target cells: Pancreatic cells.
Response: Release of bicarbonate.
Simple neuroendocrine pathway example:
Stimulus: Suckling.
Neurosecretory cell: Hypothalamus releases oxytocin into the posterior pituitary.
Target cells: Smooth muscle in mammary glands.
Response: Milk release.
Blood Glucose Regulation
Homeostasis Pathway:
STIMULUS: Blood glucose level rises (e.g., after a meal).
Insulin released by: Beta cells of the pancreas.
Body cells take up more glucose.
Liver takes up glucose and stores it as glycogen.
Response: Blood glucose level declines.
Opposite response to low blood glucose levels (e.g., between meals):
Alpha cells of pancreas release glucagon to raise blood glucose levels.
Neurohormonal Coordination
Hypothalamus and pituitary gland interactions:
Neurosecretory cells in the hypothalamus secrete neurohormones:
Posterior pituitary releases ADH (Antidiuretic Hormone) and Oxytocin.
Anterior pituitary hormones and their effects:
Tropic effects: FSH (Follicle Stimulating Hormone), LH (Luteinizing Hormone), TSH (Thyroid Stimulating Hormone), ACTH (Adrenocorticotropic Hormone).
Nontropic effects: Prolactin, MSH (Melanocyte-Stimulating Hormone).
Both: GH (Growth Hormone).
Stress Response
Short-term stress response:
Adrenal medulla releases epinephrine and norepinephrine.
Effects:
Glycogen breaks down to glucose, blood glucose levels increase.
Increased blood pressure, heart rate, and metabolic rates; changes in blood flow.
Long-term stress response:
Adrenal cortex secretes mineralocorticoids and glucocorticoids.
Effects:
Retention of sodium and water by kidneys; increased blood volume and pressure.
Breakdown of proteins and fats for glucose conversion; suppression of immune system activity.