Study Notes on Pulmonary Embolism
Overview of Pulmonary Embolism
Discusses signs, symptoms, risk factors, pathophysiology, investigations, diagnosis, treatment, and management.
Signs and Symptoms of Pulmonary Embolism
Common indicators include:
Dyspnea (difficulty breathing)
Chest pain (may vary in intensity)
Tachycardia (elevated heart rate)
Hypotension (low blood pressure)
Signs of Deep Vein Thrombosis (DVT), including:
Swollen legs
Pain in lower legs
Causes of Pulmonary Embolism
Majority of cases (90-95%) are due to thrombosis from deep veins (DVT).
Deep Vein Thrombosis mainly develops in lower limb veins, particularly:
External iliac vein
Femoral vein
Deep femoral vein
Popliteal vein
Posterior tibial vein
Less common sites include:
Right-sided heart chambers
Uterine veins
Great saphenous vein
Risk Factors for Pulmonary Embolism
Surgical procedures:
Major abdominal and pelvic surgeries
Orthopedic surgeries
Physiological changes:
Pregnancy (obstetric risk factor)
Cardiorespiratory issues (e.g., COPD, congestive heart failure)
Vascular conditions:
Varicose veins
Fractures
Malignant diseases
Age and lifestyle concerns:
Increasing age
Immobility (prolonged inactivity)
Thrombotic disorders:
Conditions that predispose to clot formation.
Pathophysiology of Pulmonary Embolism
Thrombus Definition:
A thrombus is a mass composed of clumped red blood cells, platelets, and fibrin.
Virchow's Triad:
Three components that lead to thrombus formation:
Abnormal blood flow (Venous stasis)
Hypercoagulability (thrombophilia)
Altered vessel wall (vessel wall injury): endothelial injury
Fates of a thrombus:
Possible outcomes include:
Resolution (disappearance of thrombus)
Propagation (growth of thrombus)
Embolism (thrombus detachment leading to obstruction)
Recanalization (formation of channels in the thrombus)
Organization (integration of thrombus into vessel walls)
Effects of Embolism on the Cardiovascular System
Impact on blood pressure and heart output:
Increased pulmonary vascular pressure, causing:
Backflow of blood to the right side of the heart
Elevated right ventricular pressure
Dilation of right ventricle leading to right-sided heart failure
Consequences:
Decreased stroke volume
Decreased cardiac output
Resulting in hypotension or low blood pressure
Compensation Mechanisms:
Detection of decreased cardiac output stimulates:
Sympathetic nervous system response
Increasing heart rate (tachycardia)
Attempted vasoconstriction to raise blood pressure, which may be ineffective.
Effects of Embolism on the Respiratory System
Ventilation vs. Perfusion (VQ Mismatch):
Obstruction leads to abnormal gas exchange.
Causes:
Inflammation and release of cytokines.
Bronchoconstriction, decreasing oxygen intake.
Results in:
Hypoxemia (low oxygen levels)
Hypocapnia (low carbon dioxide levels)
May lead to respiratory alkalosis, detectable via arterial blood gas test.
Investigations for Pulmonary Embolism
X-Ray:
Usually normal but can help exclude differential diagnoses (e.g., pneumonia, pneumothorax).
Possible findings in PE include:
Enlarged pulmonary artery
Wedge-shaped opacity (indicative of infarction)
Elevated hemidiaphragm
Pleural effusion
ECG:
Often normal but used to differentiate from myocardial infarction:
Common findings:
Sinus tachycardia (present in 50% of cases)
Right ventricular strain (inverted T-waves in leads V1 to V4)
S1Q3T3 pattern (deep S-wave in lead I, deep Q and T-waves in lead III)
CT Pulmonary Angiogram:
Considered the gold standard for PE detection.
VQ Perfusion Scan:
Less definitive than CT but provides additional information.
Bedside Echocardiogram:
Assess heart function and presence of thrombi.
D-Dimer Assay:
Used for assessing the propensity for thrombus formation and ruling out PE.