Cytoskeleton: Actin and Microtubules Overview
Key Concepts in Cytoskeleton
Types + Function of the Cytoskeleton
Actin Filaments
Microvilli: Projections that increase surface area and are primarily made of actin.
Functionality:
Force generation for cellular movement.
Controlling cell shape.
Cellular adhesion, influencing how cells stick together.
Different types of Actin Filaments:
single
microvilli
stress fibers
striated muscles
Globular + Filamentous Actin
G-actin are ATPases that bind + hydrolyze H2O
are also asymmetrical (has plus and minus ends
F-actin are made up of 2 G-actin strands
Tubules
GTPases (can exist in GTP or GDP bound states)
Microtubules
made up of alpha-tubulin + beta-tubulin (tubulin dimers)
creates a polarized filament where 13 of them can form a microtubule
Functions:
cell division
exocytosis
cell/organelle movement
Processes of the Cytoskeleton
Processes:
Dynamic Process
fast-changing process
e.g., cell division, where actin forms the contractile ring to split the cell during cytokinesis and microtubules form the mitotic spindle to separate the chromosomes during mitosis
Stable Process
extremely slow changes
e.g., in epithelial tissues:
actin forms microvilli
microtubules forms transport tracks
intermediate filaments stabilizes desmosomes (cell connections)
Regulation of the Cytoskeleton:
Assembly + Disassembly
g-actin subunits are held together by non-cova bonds to create f-actin polymers
for protofilaments, the more polymers are tgt = stronger + more stable
less polymers = energy is more evenly spread out = easier to break bonds
Nucleation (Lag Phase)
nucleation: process of forming new cytoskeleton (beginning the cytoskeleton is rate limiting)
3 phases: lag phase, elongation phase, steady state
Cytoskeleton Kinetics
Treadmilling:
A dynamic process in which actin filaments grow at one end (plus end) while shrinking at the other end (minus end).
Occurs at a critical concentration of actin monomers.
Treadmilling is influenced by ATP hydrolysis (the conversion of ATP to ADP), which stabilizes the polymer.
Cc(T) < C < Cc(D)
Rates
polymerization = add polymers
rate constant: Kon
rate: Rpol = kdepolarization C Kon
C = [free monomer]
depolymerization = remove polymers
rate constant = koff
rate: Rdepol = koff
Critical Concentration (Cc):
when rate of polymerization = rate of depolymerization
results in no net change in filament length (equilibrium)
Polymerization
+ end = more dynamic —> can (de)polymerize faster than - end
monomers + polymers
G-actin = monomer —> can change shape to attach to - end of polymer
G-actin monomer can be added to +/- end
+ end is added more quickly bc the shape is more favourable
- end is added more slowly bc the shape is less favourable
nucleotide binding
NTP-subunits > NDP-subunits (better affinity)
ADP actin has low affinitiy
ATP actin has high affinity and will change shape + become D when connected
T-subunit looks like G-actin subunit (before conformation change) —> tubulin GTP, actin ATP
T-subunit polymerizes into D-subunit —> actin ADP, tubulin GTP
at equilibrium, Cc(T) < Cc(D)