CHapter 41


I. Key Assessment Points for Cardiovascular Disorders in Children

Health History (Know this cold):

Feeding: Poor feeding, fatigue with feeding, sweating (especially during feeds) – classic signs in infants.

Color Changes: Cyanosis (especially lips, tongue, nail beds); ask “What color does the skin turn when crying?”

Respiratory: Tachypnea, dyspnea, frequent pneumonia.

Growth: Failure to thrive, poor weight gain, delayed motor milestones.

Activity: Squatting (in toddlers – relieves dyspnea by increasing SVR), inability to keep up with peers.

Past Medical: Prematurity, maternal infections, chromosomal anomalies, corticosteroid use.

Exam Tip: They love asking “What is the most concerning history detail?” – Answer: Sweating while feeding, cyanosis that doesn’t improve with O2, frequent respiratory infections.


II. Specific Physical Signs You Must Memorize

Inspection:

Clubbing: Late sign of chronic hypoxia (>1 year old).

Precordial bulge: Often in older children with cardiomegaly.

Color: Look for gray-blue tone = central cyanosis.

Edema: In infants → face; older children → lower extremities.

Palpation:

Femoral Pulse: Weak or absent = suspect coarctation of aorta.

Hepatomegaly: Suggests right-sided heart failure.

Auscultation:

Apical pulse: Listen full minute!

Murmurs:

S3: Pathological if heard when child is upright → suspect heart failure.

S4: Suggests decreased compliance (e.g., cardiomyopathy).

Click at apex: May be mitral or aortic origin.

Bounding pulse: Suspect PDA.

Thread pulse: Aortic stenosis or shock.

Exam Trap: “A mother hears a murmur when the child lies down” – benign? Not always! Check position, grade, and any associated findings (e.g., thrills, symptoms).


III. Cardiac Catheterization – Exact Pre/Post-op Nursing

Before:

NPO: 4–6 hours.

Allergy Check: Shellfish/iodine (contrast dye).

Baseline: Weight (for med dosing), vitals, pedal pulses, O₂ saturation.

Sedation prep: Explain to parents, include child based on age.

After:

Leg straight: For 4–8 hours (vein entry).

Monitor site: Bleeding, hematoma.

Pedal pulse check: Every 15 minutes initially.

Monitor urine output: Contrast can damage kidneys.

S/S infection or poor perfusion: Cool limb, low pulse, discoloration.

If bleeding after apply pressure 1 in above the site to create pressure over area reducing blood flow

Exam Question Clue: “A nurse notes the right foot is cooler and paler than the left after cardiac catheterization.” → Call provider immediately.


IV. Lab & Diagnostics: Match Test with Purpose

Test

Purpose

Key Nursing Action

ECG

Rhythm, conduction

Keep child still; place electrodes properly

Echo

Structure/function, valves

Noninvasive; explain sound waves

Chest X-ray

Heart size, pulmonary status

Remove metal, explain film

Arterial pO₂

Oxygen level

Arterial > venous for accuracy

Holter monitor

24h rhythm eval

Push event button if symptoms

Exercise stress test

Functional tolerance

NPO x4h, baseline vitals


V. Medication Education – Critical Nursing Implications

Digoxin:

Hold if HR <90 (infant), <70 (child).

Monitor for toxicity: Vomiting, bradycardia, anorexia, visual halos.

Therapeutic level: 0.8–2 ng/mL

Missed dose? → Give if within 12 hours, skip if close to next.

Furosemide (Lasix):

Monitor: BP, electrolytes (esp. K+), hearing.

Give with food or milk to reduce GI upset.

Causes photosensitivity.

Alprostadil:

Maintains ductus arteriosus.

Watch for apnea in neonates (20% risk).

Monitor pO₂, temp, and for abdominal distention.

Exam Trap: If a child is on digoxin and furosemide, always be alert for hypokalemia, which increases digoxin toxicity risk.


VI. Nursing Diagnoses with Interventions (Chart-style)

Nursing Dx

Goal

Interventions

↓ Cardiac Output

HR/BP/WNL, warm extremities

O₂ as ordered, monitor VS, semi-Fowler’s, decrease stimuli

Activity Intolerance

Child maintains play tolerance

Cluster care, limit exertion, rest before feeds

Nutrition < body req

Weight gain WNL

High-calorie feeds, weight daily, offer preferred foods

Fluid Overload

Balanced I/O

Daily weight, lung sounds, restrict Na+, diuretics


Growth Delay

Meet milestones

Consult PT/OT, developmental toys, encourage interaction


VII. Red Flag Scenarios

Cyanosis that doesn’t improve with O₂ = Cardiac, not pulmonary cause.

Sweating with feeds = Strong indicator of infant heart failure.

Bounding upper extremity pulse but weak femoral = Coarctation of the aorta.

Squatting = Classic in Tetralogy of Fallot; increases SVR to improve oxygenation.




Got it! Let’s break this down into hyper-specific, exam-focused nursing care and education points based on what your textbook covered. This will help you confidently answer scenario-based, priority-focused, and trick questions on your exam.


I. Key Assessment Points for Cardiovascular Disorders in Children

Health History (Know this cold):

Feeding: Poor feeding, fatigue with feeding, sweating (especially during feeds) – classic signs in infants.

Color Changes: Cyanosis (especially lips, tongue, nail beds); ask “What color does the skin turn when crying?”

Respiratory: Tachypnea, dyspnea, frequent pneumonia.

Growth: Failure to thrive, poor weight gain, delayed motor milestones.

Activity: Squatting (in toddlers – relieves dyspnea by increasing SVR), inability to keep up with peers.

Past Medical: Prematurity, maternal infections, chromosomal anomalies, corticosteroid use.

Exam Tip: They love asking “What is the most concerning history detail?” – Answer: Sweating while feeding, cyanosis that doesn’t improve with O2, frequent respiratory infections.


II. Specific Physical Signs You Must Memorize

Inspection:

Clubbing: Late sign of chronic hypoxia (>1 year old).

Precordial bulge: Often in older children with cardiomegaly.

Color: Look for gray-blue tone = central cyanosis.

Edema: In infants → face; older children → lower extremities.

Palpation:

Femoral Pulse: Weak or absent = suspect coarctation of aorta.

Hepatomegaly: Suggests right-sided heart failure.

Auscultation:

Apical pulse: Listen full minute!

Murmurs:

S3: Pathological if heard when child is upright → suspect heart failure.

S4: Suggests decreased compliance (e.g., cardiomyopathy).

Click at apex: May be mitral or aortic origin.

Bounding pulse: Suspect PDA.

Thread pulse: Aortic stenosis or shock.

Exam Trap: “A mother hears a murmur when the child lies down” – benign? Not always! Check position, grade, and any associated findings (e.g., thrills, symptoms).


III. Cardiac Catheterization – Exact Pre/Post-op Nursing

Before:

NPO: 4–6 hours.

Allergy Check: Shellfish/iodine (contrast dye).

Baseline: Weight (for med dosing), vitals, pedal pulses, O₂ saturation.

Sedation prep: Explain to parents, include child based on age.

After:

Leg straight: For 4–8 hours (vein entry).

Monitor site: Bleeding, hematoma.

Pedal pulse check: Every 15 minutes initially.

Monitor urine output: Contrast can damage kidneys.

S/S infection or poor perfusion: Cool limb, low pulse, discoloration.

Exam Question Clue: “A nurse notes the right foot is cooler and paler than the left after cardiac catheterization.” → Call provider immediately.


IV. Lab & Diagnostics: Match Test with Purpose

Test

Purpose

Key Nursing Action

ECG

Rhythm, conduction

Keep child still; place electrodes properly

Echo

Structure/function, valves

Noninvasive; explain sound waves

Chest X-ray

Heart size, pulmonary status

Remove metal, explain film

Arterial pO₂

Oxygen level

Arterial > venous for accuracy

Holter monitor

24h rhythm eval

Push event button if symptoms

Exercise stress test

Functional tolerance

NPO x4h, baseline vitals


V. Medication Education – Critical Nursing Implications

Digoxin:

Hold if HR <90 (infant), <70 (child).

Monitor for toxicity: Vomiting, bradycardia, anorexia, visual halos.

Therapeutic level: 0.8–2 ng/mL

Missed dose? → Give if within 12 hours, skip if close to next.

Furosemide (Lasix):

Monitor: BP, electrolytes (esp. K+), hearing.

Give with food or milk to reduce GI upset.

Causes photosensitivity.

Alprostadil:

Maintains ductus arteriosus.

Watch for apnea in neonates (20% risk).

Monitor pO₂, temp, and for abdominal distention.

Exam Trap: If a child is on digoxin and furosemide, always be alert for hypokalemia, which increases digoxin toxicity risk.


VI. Nursing Diagnoses with Interventions (Chart-style)

Nursing Dx

Goal

Interventions

↓ Cardiac Output

HR/BP/WNL, warm extremities

O₂ as ordered, monitor VS, semi-Fowler’s, decrease stimuli


Activity Intolerance

Child maintains play tolerance

Cluster care, limit exertion, rest before feeds

Nutrition < body req

Weight gain WNL

High-calorie feeds, weight daily, offer preferred foods

Fluid Overload

Balanced I/O

Daily weight, lung sounds, restrict Na+, diuretics

Growth Delay

Meet milestones

Consult PT/OT, developmental toys, encourage interaction


VII. Red Flag Scenarios

Cyanosis that doesn’t improve with O₂ = Cardiac, not pulmonary cause.

Sweating with feeds = Strong indicator of infant heart failure.

Bounding upper extremity pulse but weak femoral = Coarctation of the aorta.

Squatting = Classic in Tetralogy of Fallot; increases SVR to improve oxygenation.









1. Atrioventricular Canal Defect (AVSD / AV Canal)

Seen in 35–40% of children with Down syndrome

Pathophysiology:

Failure of endocardial cushion → ASD + VSD + common AV valve

Results in left-to-right shunting of oxygenated blood back to lungs

Causes increased pulmonary blood flow, pulmonary edema, and heart failure

Timing of Symptoms:

Usually appears at 4 to 8 weeks of age

Initially asymptomatic due to high pulmonary resistance in neonates

Assessment / Signs:

Tachypnea, diaphoresis with feeding, poor weight gain

Frequent infections

Characteristic harsh holosystolic murmur (left sternal border)

Adventitious lung sounds (rales if heart failure is present)

Thrill may be palpable if severe

Diagnostics:

Echocardiogram or MRI = definitive

Cardiac cath = pressure/flow eval

CXR: cardiomegaly, pulmonary congestion

Parent Education:

Monitor for sweating, fatigue, poor feeding

Feed every 3 hours or less, limit feedings to <30 minutes

Monitor daily weight

Explain possible need for surgical repair and frequent follow-ups


2. Patent Ductus Arteriosus (PDA)

Common in preterm infants, high altitude births

Pathophysiology:

Ductus arteriosus remains open → aorta shunts blood to pulmonary artery

Causes volume overload on lungs → pulmonary hypertension

Timing of Symptoms:

Detected within first 2 weeks of life

May be asymptomatic if small

Assessment / Signs:

Tachypnea, fatigue, poor growth

Bounding peripheral pulses

Wide pulse pressure (↑ systolic, ↓ diastolic)

Harsh, continuous “machine-like” murmur under left clavicle

Diagnostics:

Echo confirms size/severity

ECG: may show LV hypertrophy if large PDA

CXR: pulmonary congestion

Parent Education:

If small, may close on its own

Medical closure = indomethacin or ibuprofen

Surgical closure if not resolved

Teach signs of heart failure: breathing faster, sweating, not eating well


3. Coarctation of the Aorta

Accounts for ~10% of CHDs

Pathophysiology:

Narrowing of aorta → ↑ pressure before narrowing, ↓ after

Causes upper extremity hypertension, lower extremity hypoperfusion

May lead to LV hypertrophy, collateral circulation

Timing of Symptoms:

Often detected in infancy or school-age child during BP screening

Assessment / Signs:

BP in upper > lower extremities by ≥20 mmHg

Bounding radial/brachial pulses, weak or absent femoral pulses

Leg pain with activity, headaches, dizziness

Murmur on back or left axilla

Diagnostics:

Echo = confirms narrowing

CXR: rib notching (collateral circulation)

ECG: LV hypertrophy

Parent Education:

Monitor BP in all 4 extremities

Educate on importance of early surgical repair

Watch for leg cramps, poor endurance

May require lifelong follow-up for re-coarctation or hypertension


4. Aortic Stenosis

Accounts for ~5% of CHDs

Pathophysiology:

Obstruction at or below the aortic valve

Causes LV to work harder, leading to LV hypertrophy

↓ cardiac output → poor systemic perfusion

Timing of Symptoms:

Often asymptomatic early

Can cause exertional chest pain, dizziness, fainting

Assessment / Signs:

Fatigue, syncope, angina with activity

Thrill at base of heart

Systolic murmur at left sternal border, may radiate to neck

Infant-specific Signs:

Poor feeding, weak pulses, pallor

May present as shock in severe cases

Diagnostics:

Echo: visualizes stenosis

ECG: LV hypertrophy

Exercise testing may be done in older children

Parent Education:

Teach activity restrictions based on severity

Instruct to report chest pain, fainting, or fatigue

May require balloon valvuloplasty or valve replacement


5. Pulmonary Stenosis

Pathophysiology:

Narrow pulmonary valve → RV can’t eject blood → RV hypertrophy

Can cause reopening of foramen ovale → cyanosis

Timing of Symptoms:

Mild = asymptomatic

Severe: early onset cyanosis, dyspnea with exertion

Assessment / Signs:

Systolic ejection murmur at left upper sternal border

Click after S2

Cyanosis if foramen ovale reopens

RV heave, fatigue, exercise intolerance

Diagnostics:

Echo: valve thickening or narrowing

ECG: RV hypertrophy

Parent Education:

Monitor for exertional dyspnea or cyanosis

Educate on the possibility of needing a balloon valvuloplasty

Teach that symptoms may worsen with growth


6. Transposition of the Great Vessels (TGV)

Medical emergency! Requires early surgery

Pathophysiology:

Aorta and pulmonary artery are switched

Creates parallel circuits: deoxygenated blood goes to body; oxygenated blood circulates back to lungs

Not compatible with life without mixing

Timing of Symptoms:

Presents in first few hours to days of life when PDA closes

Rapid progression of cyanosis

Assessment / Signs:

Cyanosis unresponsive to oxygen

Tachypnea, retractions

Single loud S2; murmur only if VSD or PDA also present

Diagnostics:

Echo = confirms diagnosis

Pulse ox: severe hypoxemia

CXR: “egg on a string” heart

Parent Education:

Immediate intervention needed

Prostaglandin E1 (alprostadil) keeps PDA open until surgery

Explain corrective surgery (arterial switch) done at 4–7 days old

Teach signs of hypoxia, poor feeding, or cyanotic spells


What They’ll Test – Exam-Specific Watchpoints

SATA Likely Topics:

Signs of coarctation = bounding arm pulses, weak leg pulses, BP difference

Teaching for PDA = indomethacin, signs of HF

Signs of AV canal = diaphoresis, poor feeding, harsh murmur

Signs of TGV = cyanosis not relieved by oxygen, emergency prostaglandin

High-Risk Select Options:

“What would you expect in a child with aortic stenosis?” → Fatigue, weak pulses, systolic murmur

“What needs immediate intervention?” → O₂ sat 78% in newborn with known TGV

“Which findings suggest worsening pulmonary stenosis?” → Cyanosis with exertion, fatigue, murmur intensifying




Pages 1485–1493 | Congenital & Acquired Heart Defects: Clinical & Test-Critical Notes


1. Total Anomalous Pulmonary Venous Connection (TAPVC)

Key Point: Pulmonary veins drain into right atrium, not left

Requires ASD or PFO for survival

Cyanosis without murmur in newborn

Murmur appears later if septal defect is present

Signs: Prominent RV impulse, hepatomegaly, retractions

Echo + cardiac cath confirm diagnosis

High-Yield Tip: Mixing of oxygenated and deoxygenated blood → severe hypoxia if no ASD


2. Truncus Arteriosus

One major artery (instead of separate aorta/pulmonary) leaves heart

Mixing of blood from both ventricles

VSD always present

↑ pulmonary flow → pulmonary hypertension

Signs: Cyanosis, feeding difficulty, grunting, retractions

Murmur: From VSD

Test Focus: Look for single great vessel, mixing, and progressive heart failure


3. Hypoplastic Left Heart Syndrome (HLHS)

Most critical & fatal defect if untreated

Left ventricle underdeveloped or absent

PDA-dependent circulation

As ductus closes → shock & death without surgery

Signs: Rapid cyanosis, gray/blue skin, poor perfusion, gallop rhythm, weak pulses

Key Interventions:

Prostaglandin E1 to keep PDA open

Emergent surgery (3-stage Norwood or transplant)

NGN Danger Clue: “Infant begins to crash as ductus closes” = HLHS or TGV emergency


4. BOX 41.1 – Relieving Hypercyanotic (Tet) Spells

They will ask you this.

SATA:

Knee-to-chest position

Oxygen

Morphine

IV fluids

Propranolol


5. Nutritional Needs in CHD Infants

↑ calorie needs: up to 150 cal/kg/day

Infant formulas: 24–28 cal/oz

Gavage feed if too tired

Feed <20 min to conserve energy

Small, frequent feedings

Monitor weight daily

Note: Breastfeeding boosts immunity, may be hard for sick infants


6. Preoperative & Postoperative Care for Cardiac Surgery

Pre-op Priorities:

Full history, including med/surgical, infections, and parent understanding

Review recent lab/echo data

Baseline vitals + weight

NPO + fluid restrictions

Educate on: ICU stay, equipment (pacing wires, chest tubes), incisions

Let parents accompany child to OR if policy allows

Post-op Priorities:

Vital signs q1h, cap refill, skin color

Assess: heart rate/rhythm, edema, breath sounds

Watch for:

Cardiac tamponade = muffled heart sounds + ↑ HR + ↓ BP

Chest tube output drop + ↑ HR = EMERGENCY

Pain control, early ambulation, lung expansion exercises

NGN Trap Tip: Choose all that reflect monitoring perfusion, respiratory function, and bleeding


7. BOX 41.2 – Possible Post-Op Complications

You’ll see a SATA here.

Cardiac tamponade

Arrhythmias

Hemorrhage

Pneumonia

Seizures

Wound infection

Postpericardiotomy syndrome

Atelectasis

Endocarditis


8. Teaching Guidelines 41.2 – Parent Teaching: CHD Child

SATA/NCLEX-Style Must-Know:

Give meds as ordered

Weigh daily same time, same scale, same clothes

Allow child to rest between activities

Provide high-calorie, nutritious food

Teach infection prevention

Track signs of worsening heart failure:

↓ wet diapers

SOB

Color changes

Irritability


9. Heart Failure in Children

Most common cause = CHD

Up to 20% of CHD kids develop HF

Systolic Dysfunction Signs:

Pulmonary congestion, ↑ work of breathing, feeding fatigue

Rales, wheezing, retractions

Diastolic Dysfunction Signs:

Hepatomegaly

Jugular distention

Periorbital/facial edema

Red Flags to Ask Parents:

“Baby stops drinking and resumes later”

“Sweats while feeding”

“Sleeps better upright”

“Rapid breathing during feeds”


10. Therapeutic Management & Meds

Digoxin: Hold if HR < 90 (infants), <70 (kids), watch for vomiting/bradycardia

ACE inhibitors: Watch for hypotension, monitor K+

Diuretics: Daily weight, K+ loss

Beta-blockers: For cyanotic spells




Perfect — here is a hyper-specific, exam-focused summary of pediatric cardiac disorders from pages 1480–1492, aligned to NGN/NCLEX and textbook-level questions. I’ve included must-know teaching points, SATA bait, red flag signs, and lab/assessment priorities. This covers exactly pages 1480 through 1492 — which includes AV Canal, PDA, Coarctation, Stenosis types, TGV, TAPVC, HLHS, CHF, and Cardiac Surgery Nursing Care.


I. Atrioventricular Canal Defect (AV Canal)

Hallmark Teaching/Exam Tips:

Strongly associated with Down syndrome (35–40% of affected kids).

Left-to-right shunt → increased pulmonary blood flow → heart failure

Key Signs: sweating with feeds, poor feeding, failure to thrive

Patho Tip: Oxygenated blood enters L atrium → re-circulates to lungs again via septum.

Murmur: Characteristic holosystolic harsh murmur at L sternal border

Parent Education:

Signs of heart failure (tachypnea, diaphoresis)

Expect murmur early (4–8 weeks old)

Early intervention often needed


II. Patent Ductus Arteriosus (PDA)

Clinical Highlights:

Very common in preemies

Allows oxygenated blood from aorta → pulmonary artery

Causes left-to-right shunt

SATA Red Flags:

Bounding pulses

Wide pulse pressure

Diastolic BP may be low

“Machinery-like murmur” (continuous, loudest at 1st–2nd ICS)

Exam Scenario Trap: PDA can keep cyanotic infants oxygenated briefly → watch for worsening symptoms when PDA closes


III. Coarctation of the Aorta

Key Signs You MUST Know:

Upper extremity HTN; lower extremity hypotension

Weak or absent femoral pulses

BP in arms >20 mmHg higher than legs

Systolic murmur (back or left axilla)

SATA Traps:

Look for leg pain with activity

Headaches

Epistaxis (nosebleeds)

RIB NOTCHING on CXR

Murmur most audible at base of heart or left axilla


IV. Aortic Stenosis

Obstruction between left ventricle and aorta

Most common: narrowing at the valve

Signs: chest pain, dizziness with standing, fatigue

May be asymptomatic or have a systolic ejection murmur

Pulse may be faint, thrill at base of heart

Exam Angle: if infant – look for poor feeding + faint pulses. With exertion → syncope, angina.


V. Pulmonary Stenosis

Obstructs blood flow from RV to pulmonary artery

Often tied to Tetralogy of Fallot

RV hypertrophy develops

Key Murmur: systolic ejection murmur with click

SATA Signs:

Dyspnea on exertion

Cyanosis if severe

Right-sided heart failure signs if prolonged


VI. Transposition of the Great Vessels (TGV)

Critical Exam Red Flags:

Severe cyanosis that doesn’t improve with oxygen

Aorta arises from RV and PA from LV → deoxygenated blood goes to body

Often detected in first few days of life

PDA may initially compensate

Must-Know: Surgery required within 4–7 days


VII. Total Anomalous Pulmonary Venous Return (TAPVC)

Pulmonary veins connect to RA instead of LA

Mixing of oxygenated & deoxygenated blood

Causes volume overload of RA and RV → pulmonary edema

Assessment:

Cyanosis

Tachypnea

Edema

Fixed splitting of S2

Palpable liver


VIII. Truncus Arteriosus

One large artery instead of aorta + pulmonary artery

Mixed blood enters systemic + pulmonary circulation

High risk of pulmonary over-circulation → CHF

May have a murmur; may be associated with DiGeorge syndrome


IX. Hypoplastic Left Heart Syndrome (HLHS)

L side of heart is nonfunctional

Often fatal unless surgically corrected

Blood shunted through PDA and ASD until closed

Early Signs:

Cyanosis

Shock after PDA closes

Gallop rhythm, poor feeding, weak pulses

Critical Teaching:

Surgery needed in 3 stages OR heart transplant

May need PGE (prostaglandin) to keep PDA open


X. Signs of Pediatric Heart Failure – Exam Goldmine

SATA/Exam Tip:

Sweating during feeds

Poor feeding, failure to thrive

Tachypnea at rest

Hepatomegaly

Irritability

Periorbital edema

Frequent infections

Pulmonary congestion signs:

Nasal flaring

Retractions

Crackles, grunting

Apical HR may be rapid; assess for gallop rhythm, S3/S4


XI. Management of Congenital Heart Disease (1478–1492)

Nutrition Teaching Points:

Use high-calorie formula (24–28 kcal/oz)

Feed <30 min per session

Frequent, small feedings

NG/gavage if energy too low

Daily weights, strict I/Os

Improving Oxygenation:

Semi-Fowler’s for lung expansion

Monitor SaO₂, respiratory effort, nasal flaring

Use O2 cautiously with L→R shunts

Preventing Infection:

Strict hand hygiene

Endocarditis prophylaxis before dental/valve procedures


XII. Cardiac Surgery Care

Preop Care:

Establish baselines

Prepare child/family with developmentally appropriate language

NPO before surgery

Teach about expected equipment: chest tubes, O2 sat probes, pacing wires, dressings

Postop Care:

Vital signs Q1H

Cardiac monitor

Check pulses, cap refill

Assess for:

Tamponade (↑HR, ↓BP, ↓output)

Arrhythmias

Poor perfusion

Weight gain, crackles (fluid overload)

Key Concept Alert:

Abrupt cessation of chest tube drainage + ↑HR + increased filling pressure = CARDIAC TAMPONADE.


XIII. BOX 41.2 – Post-Cardiac Surgery Complications

Tamponade

Seizures

Endocarditis

Pneumothorax/pleural effusion

Postperfusion syndrome

HF

Wound infection

Arrhythmias


XIV. Medication-Specific Pearls (From Digoxin, Diuretics)

Digoxin Dosing Tip (Box 41.1):

Usual safe dose: 10–15 mcg/kg/day in 2 doses

Hold if HR <90 in infants, <70 in children

Toxicity = vomiting, bradycardia, lethargy