Locomotion and functions of the cerebellum
Introduction to Locomotion and the Cerebellum
Presented by Professor Vincent CK Cheung, School of Biomedical Sciences, The Chinese University of Hong Kong.
Date: 7 January 2026, Wednesday.
Overview of cyclic nature of locomotion.
Historical Background on Motion Studies
Eadweard Muybridge (1830 – 1904)
Key contributions to the understanding of movement through photography.
Documented various modes of locomotion:
Walking
Running
Crawling
Flying
Swimming
Example: A running bison activates muscle groups in a cyclical form.
Visual Representation of Movement
Key works from artists and photographers:
"The Horse in Motion" by Eadweard Muybridge (1878)
"The Barb" by Richard Wingfield Stuart (1886)
"Erlkoenig" by Moritz von Schwind (1860)
Gait Cycle Overview
Each gait cycle comprises two primary phases:
Stance Phase: When the foot is in contact with the ground.
Swing Phase: When the foot is in the air.
Example: In walking cats, both flexors and extensors engage in complex patterns during these phases:
Hip: Flexion and extension
Knee: Flexion and extension
Ankle: Controlled extension and flexion.
Neurological Basis of Locomotion
The spinal cord’s sufficiency in generating locomotor patterns once locomotion is initiated.
In experiments with decerebrate cats (forebrain severed), locomotor rhythm maintained without brain involvement.
Sensory feedback from limbs to the spinal cord is critical for coordinating locomotion.
Graham Brown's Experiment (1911)
Conducted a deafferentation experiment establishing that:
Locomotor rhythmic activities can persist without sensory input to the central nervous system (CNS) once locomotion is triggered.
Isolation of the spinal cord from brain control demonstrated sustained locomotor rhythm.
Findings indicate that alternating muscle contractions are independent of peripheral sensory feedback.
Central Pattern Generators (CPG)
Definition: Neuronal networks within the CNS that generate rhythmic motor activities autonomously, without sensory input coordination.
Components include:
Half Center Model: Two mutually inhibitory centers that regulate alternate muscle activation.
Inhibitory Output: Fatigues over time, triggering activation of the opposite center to create rhythmic motion.
Functions facilitated by:
Descending modulation
Sensory modulation
Temporal and spatial specifications.
Modulation of CPG Activities
Inputs from the brainstem that can alter CPG activity:
Midbrain Locomotor Region (MLR)
Subthalamic locomotor region
Pontine reticular formation
These areas influence speed and facilitate modifications for muscle recruitment patterns.
Effects of Stretching on Locomotion
Stretching of hip flexors (iliopsoas) simulates end-of-stance phase conditions.
Stretching can induce early initiation of the swing phase.
Muscle spindle afferents provide real-time feedback altering phase lengths or timing during locomotion.
Visual and Motor Cortex Interaction
Motor Cortex Activity: Engages during locomotion, specifically for obstacle navigation.
Requires sensory inputs to guide walking trajectories and obstacle avoidance.
Feedback from visual stimuli is integral to modulating locomotor responses.
Role of Neurons in Obstacle Navigation
The posterior parietal cortex is critical for processing spatial memory relevant to steady walking on uneven surfaces.
Involves working memory to adapt limb movements based on obstacle heights:
Demonstrated through comparisons of pause durations before and after lesions impacting this cortical area.
Key Takeaways on Locomotor Patterns
Spinal locomotor patterns exhibit adaptability influenced by:
Sensory inputs (proprioception and cutaneous signals)
Constant regulation by descending commands from higher brain areas.
Adjustments in muscle patterns and locomotion speed necessary for various locomotor tasks.
Understanding Human Locomotion
Research by Capogrosso et al. (2016) shows that:
Direct cortical inputs to the spinal cord are essential for initiating or maintaining CPG activities, especially after spinal cord lesions.
Articulating the connection between brain, spinal cord, and sensory feedback during locomotion.
Overview of Cerebellar Function
Cerebellum: Located in the hindbrain, plays a critical role in motor output modification, facilitating flexibility and precision.
Composed of three layers with specific roles:
Purkinje Cells: Receive converging inputs from granule cells, integrating sensory and motor signals for movement coordination.
Cerebellar Structure and Connectivity
Inputs:
Mossy Fibers: Convey sensory information to granule cells.
Climbing Fibers: Provide error signals necessary for refining coordination.
Outputs:
Purkinje Cells: Each can influence approximately 500 other neurons, creating extensive neural networks for motor regulation.
Motor Control Strategies
Two fundamental strategies:
Feedforward Motor Control: Involves pre-planned commands that do not adapt to feedback.
Feedback Motor Control: Adjusts movements based on real-time sensory input, enhancing accuracy.
Spinocerebellum Functions
Acts as a neural substrate for feedback motor control:
Generates motor commands for muscle activations based on sensory feedback from ongoing movements.
Cerebrocerebellum Role
Responsible for motor planning and anticipation of movement execution:
Binds closely with feedforward control alongside the motor cortex.
Effects of Cerebellar Lesions
Motor impairments following cerebellar damage include:
Inability to coordinate spatially and temporally across multi-joint movements.
Dysfunction in motor planning and feedback adjustments that typically ensure accurate motion.
Final Thoughts on Cerebellar and Locomotor Networks
The cerebellum modulates and refines activities across the spinal cord and cortex through feedback loops.
Highlighting the intricate connections between the cerebellum, cortex, and spinal network for optimal motor control.