L 14 Coronary Heart Disease Study Notes
Coronary Heart Disease
Definition, Epidemiology, and Risk Factors
Definition: Coronary Heart Disease (CHD), also known as Coronary Artery Disease (CAD), is characterized by the narrowing or blockage of coronary arteries due to atherosclerosis.
Epidemiology:
Leading cause of death for both men and women in the United States.
1 in 4 deaths in the US is due to CHD.
One person dies every minute from CHD in the US.
CHD is the number one cause of death worldwide.
Death rates from CHD have declined yearly since 1968 due to risk factor modifications and improvements in medical/surgical therapies.
However, aging of the US population will likely lead to increased prevalence of CHD despite preventive measures.
Risk Factors:
Family History: Premature CHD (myocardial infarction or death from CHD in a first-degree relative before age 55 in males or 65 in females).
Cigarette Smoking.
Dyslipidemia.
Hypertension.
Age: Male ≥ 45, Female ≥ 55.
Gender: Males generally present higher risk; risk increases for females post-menopause.
Inflammation: Elevated high-sensitivity C-reactive protein (hs-CRP): Low risk <1, Intermediate risk 1-3, High risk >3.
Chronic Conditions: Diabetes mellitus, chronic kidney disease.
Lifestyle Factors: Obesity, poor diet, lack of physical exercise.
Psychosocial Factors: Stress, depression, HIV, mediastinal radiation, microalbuminuria.
Course Content Overview
Focus of the course includes:
Coronary heart disease/CAD
Stable angina pectoris
Acute coronary syndrome (ACS): unstable angina, NSTEMI, STEMI
Vasospastic angina
Sudden cardiac death (SCD)/Sudden cardiac arrest (SCA)
Learning Objectives
Recognize risk factors and identify high-risk patients for CHD, stable angina pectoris, ACS, vasospastic angina, and SCD/SCA.
Describe clinical presentations associated with each condition, including symptoms and physical exam findings.
Conduct a focused medical history and targeted physical examination for patients presenting with CHD and related conditions.
Use and interpret the ACC ASCVD and AHA PREVENT risk calculators.
Select and interpret laboratory and diagnostic tests related to diagnosis and management of these conditions.
Apply diagnostic criteria and create management plans for patients.
Understand pharmacologic and nonpharmacologic treatments, including indications, side effects, complications, and contraindications.
Outline methods for primary and secondary prevention of CHD.
Apply screening recommendations for CHD.
Pathophysiology
Atherosclerosis: Characteristics include:
Initiated by endothelial injury/dysfunction, leading to smooth muscle cell proliferation, inflammatory cell recruitment, and lipid deposition.
Fatty streaks in childhood evolve into atherosclerotic plaques, which narrow the coronary artery lumen, affecting myocardial blood flow.
Plaque rupture exposes a thrombogenic core, leading to platelet adherence, aggregation, and potential acute coronary syndromes (ACS).
Mathematical Display:
Coronary Artery Lumen Stenosis:
Low ESS (Endothelial Shear Stress) correlates with
No lumen stenosis: No remodeling, inflammation +, early plaque formation.
Lumen stenosis <30%: Expansive remodeling, inflammation ++, small lipid pool, non-stenotic.
Lumen stenosis <50%: Expansive remodeling, inflammation +++, large lipid pool, thin fibrous cap.
Lumen stenosis >50%: Expansive remodeling, inflammation +++, large lipid pool, thin fibrous cap, plaque rupture leading to acute coronary event.
High-Risk Patients
Risk for major coronary events similar to established CHD patients includes those with:
Noncoronary atherosclerotic diseases (e.g. carotid artery disease, peripheral artery disease, abdominal aortic aneurysm).
Diabetes mellitus and chronic kidney disease.
Symptoms of CHD
Symptoms can be:
Asymptomatic
Chest pain or angina: Heaviness, pressure, squeezing, burning.
Pain radiating to the arm, shoulder, neck, or jaw.
Dyspnea, syncope, weakness, fatigue, palpitations, and signs of heart failure.
Diagnostic Testing
Testing and imaging modalities include:
Laboratory tests: No single test confirms CHD; typical tests include lipid panels, complete metabolic panels, troponin (when ACS suspected).
ECG: Low sensitivity for diagnosing CHD; changes during angina may show ST depression. Assessed abnormalities increase CHD risk.
Imaging: Echocardiogram to evaluate cardiac function and identify wall motion abnormalities.
Coronary Angiography: Considered the gold standard; assesses degree and location of stenosis and complications (risk <1% for serious adverse outcomes).
Management Strategies
Medical Therapies:
Antiplatelet agents (Aspirin, P2Y12 receptor blockers).
Statins for LDL reduction and atherosclerosis regression.
Beta blockers for reducing anginal episodes and improving exercise tolerance.
Calcium channel blockers and nitrates to manage symptoms.
ACE inhibitors and ARBs for patients with hypertension or heart failure.
Ranolazine for chronic angina.
Interventional Treatments:
Percutaneous Coronary Intervention (PCI) with stenting, preferred for stenosis ≥70%.
Coronary Artery Bypass Grafting (CABG) recommended for patients with multiple lesions or complex anatomy.
Screening Recommendations
Begin screening at age 20:
Baseline lipid panel and ASCVD risk calculation.
General consensus suggests no routine screening in asymptomatic patients; individual risk assessments should be factored in.
Conclusion
Emphasize lifestyle modifications and ongoing management for patients with CHD for secondary prevention and improving outcomes.