Biotoxins_-_Zootoxins-1__2_
Zootoxins Overview
Zootoxins are chemical means of defense and/or food acquisition found in various animals.
Present in all phyla of the animal kingdom; species produce poisons or venoms.
Poisons: Compounds in nonspecialized tissues, requiring usually oral contact for toxicity.
Venoms: Produced in specialized tissues or glands, delivered through mechanisms like stingers or fangs.
Types of Zootoxins
Poisons
Characteristics:
Produced as secondary metabolites without active delivery systems.
Exposure typically requires direct contact (oral or rarely dermal).
Venoms
Characteristics:
Produced in specialized organs; involves mechanisms like envenomation.
Venomous animals have apparatuses to inject venom into prey.
Mixtures of chemical compounds that can synergistically enhance toxicity.
Clinical Considerations
Envenoming or poisoning events are rare in domestic animals due to responsible ownership.
Snakes: Venomous Species
Approximately 400 species of venomous snakes globally, absent in Hawaii, Ireland, and New Zealand.
Crotalidae and Elapidae are prevalent in the Western Hemisphere.
Crotalids (Pit Vipers)
Characteristics
Heat-sensing pits between nostrils and eyes.
Features include elliptical pupils, triangular-shaped heads, retractable fangs.
Rattlesnakes possess keratin rattles on their tails.
North American Crotalids
Rattlesnakes: Includes genera Crotalus and Sistrurus (29 subspecies).
Cottonmouths (Agkistrodon piscivorus) and Copperheads (Agkistrodon contortrix) are also present.
Clinical Impact
Copperheads are responsible for most snakebites in the U.S.
Rattlesnakes are linked to most human deaths due to more potent venom.
Annually, 150,000 to 300,000 animals bitten by pit vipers in the U.S.
Venom Composition of Crotalids
Complex mixtures of:
Enzymes
Cytotoxins
Neurotoxins
Cardiotoxins
Hemolysins
Coagulants/anticoagulants
Other components (e.g., lipids, nucleotides).
Types of Rattlesnake Venom
Classic Diamondback Venom: Severe tissue destruction.
Mojave A Venom: Severe neurotoxic effects, little tissue damage.
Mixed Venom: Contains elements of both classic neurotoxic venoms.
Impact of Venom Components
Hyaluronidase: Increases venom spread through tissue breakdown.
Myotoxins: Cause muscle damage and necrosis.
Cardiotoxic Agents: Lead to hypotension unresponsive to treatment.
Clinical Signs of Crotalid Bite
Local pain, swelling, discoloration.
Systemic signs can include shock, tachycardia, nausea, and lethargy.
Neurological signs in neurotoxic bites (e.g., ataxia, paralysis).
Treatment Protocols
First Aid
Keep the animal calm, keep bite location below heart level.
Monitor swelling and mark bite site.
Medical Interventions
Intravenous therapy: Address hypotension.
Blood transfusions: Manage coagulopathy.
Antivenom: Main treatment for moderate to severe envenomation.
Antivenoms
Polyvalent Crotalid Antivenom: Effective for multiple pit viper species.
Fab antivenoms: Newer options with higher potency but higher costs.
Vaccination
Rattlesnake vaccine protects against major rattlesnake venoms in dogs and horses.
Does not protect against Mojave rattlesnake venom.
Elapid Snakes
Two venomous species in North America: Sonoran Coral Snake and various subspecies of Eastern Coral Snake.
Coral Snake Characteristics: Non-aggressive, nocturnal, bright-colored.
Venom and Effects
Coral snake venom causes neuromuscular blockade and local tissue necrosis.
Clinical signs develop slowly; CNS effects like quadriplegia may follow.
Widow Spiders (Latrodectus spp.)
Worldwide distribution with distinctive markings.
Widows are shy and typically bite only when threatened.
Widow Venom
Contains neuroactive proteins, primarily α-latrotoxin.
Clinical signs include severe muscle pain, hypertension, tachycardia.
Treatment Options for Widow Spider Bites
Symptomatic treatment: Opioids, muscle relaxants, and specific antivenom for cats.
Recluse Spiders (Loxosceles spp.)
Medical importance primarily from Brown Recluse distribution in the U.S.
Produces necrotizing enzymes leading to severe tissue damage.
Clinical Signs & Treatment
Characterized by a “bull’s-eye” lesion around the bite site.
Treatment includes wound care, pain management, and possible antibiotics.
Scorpions
Mainly Bark Scorpions in North America (e.g., Arizona bark scorpion).
Venom consists of polypeptides, causing pain and neurotoxic symptoms.
Treatment
Focuses on symptom control and managing systemic reactions after envenomation.
Tick Paralysis
Common in livestock and dogs from specific ticks; presents as paralysis similar to botulinum.
Supportive care and tick removal are crucial for treatment.
Blister Beetles
Toxic principle: Cantharidin, found in hemolymph and excretions.
Potentially lethal if ingested in hay contaminated by crushed beetles.
Clinical Signs and Treatment
Causes severe gastrointestinal and urinary tract signs.
Diagnosis confirmed through cantharidin detection; treatment is primarily supportive.