Mood Disorders
Announcements:
Moodle Quiz
Read An Unquiet Mind- discussion on Wednesday after break
Lecture Outline
mood
internalizing spectrum
unipolar mood disorders
causal factors of depression
treatment of depression
bipolar mood disorders
causal factors of bipolar
treatment of bipolar
Mood
Mood
Overall emotional experience
Fluctuates over time
Positive vs. negative valence
Associated thoughts
Dimensional model of mood reflects
Broad clustering of the subjective experience of emotions
Intensity
Mood versus Mood Disorder
Mood episode
relatively long-term time period (a few weeks) where moods are abnormal
The building blocks of Mood Disorders
Mood Disorder
Combination of 1 or more Mood Episodes
Can be a combination of Depression and/or Mania
Statistics about Mood Disorders
Depression= 7%
Lifetime women= 10-25%
Lifetime men= 5-12%
Persistent Depressive Disorder (Dysthymia) = 2%
Premenstrual Dysphoric Disorder= 1.3-1.8%
Bipolar Disorders= 1-2%
Signs and Symptoms of Mood Disorder
Mood
Cognitive
Behavioral
Major Depressive Disorder
Once someone has had a manic episode, they can no longer ever be diagnosed with Major Depressive Disorder again, only Bipolar, because some treatments of MDD have a negative impact on those with bipolar
Persistent Depressive Disorder (Dysthymia)
Persistent depressive disorder (in children and adolescents, mood can be irritable and duration must be 1 year or longer)
Depressed mood for most of the day, for more days than not, for 2 years or longer
Presence of 2 or more of the following during the same period
Poor appetite or overeating
insomnia or hypersomnia
low energy or fatigue
Depressive Disorder Specifiers
With Anxious Distress
w mixed features
potential for bipolar diagnosis later on
w catatonia
Individuals not getting out of bed, talking about now feeling sad but feeling empty
w melancholic features
moment to moment emotional reactivity- not responsive
w atypical features
w peripartum onset
w seasonal pattern
w mood-congruent psychotic features
w mood-incongruent psychotic features
Premenstrual Dysphoric Disorder
Mood fluctuations/disturbance
times around menstrual cycle
includes other mood disorder symptoms
severity, but not duration of other mood disorders
Depression
Demographics of Depression
Age
symptoms (and terms) change with age
Gender
1.5 to 3x as likely to be diagnosed in women
Role stress→ “double shift”- gender expectations change in the late 60s early 70s, women were expected to work in the home prior to this, then in the 60s and 70s an increase of women working outside of the home. men and women shared domestic tasks now
Men externalize negative affect, and women internalize
Culture
Symptoms (and terms) change with culture
SES
Low SES associated with higher rates and longer duration of depression
What Causes Depression
Biological factors
Genetic Factors
Higher risk in relatives
5-HTT Gene (Serotonin transporter protein
Neurochemical Factors
Abnormal Neurotransmitter Levels: Norepinephrine (low), serotonin (low)
Monoamine receptors
Hormonal factors: Cortisol
Brain Factors
Prefrontal cortex (left)- less gray matter, reduced activity
Anterior cingulate- dysregulated activity
Hippocampus- smaller, less active
Amygdala- larger, more active
Neuroendocrine Factors
Sleep
Seasons
Psychological Factors
Stressful Life events
Chronic Stress
Major life events
Shifts in rewards or punishments
Learned helplessness
Pessimistic attributional style:
Internal (vs external) attributions
Stable (vs unstable) attributions
Global (vs specific) attributions
Linked to depression, low self esteem, hopelessness/helplessness
Psychological Factors
Cognitive distortions
Negative automatic thoughts
cognitive biases: magnification, overgeneralization, etc
Ruminative Response
Intense focus on feelings/symptoms of depression and causes of those symptoms
Interpersonal problems
E.g. excessive Reassurance
Biological Interventions - Tryclics
Widley used (e.g., Tofranil, Elavil)
Symptom relief in 60% of people with depression
Block reuptake of norepinephrine and other neurotransmitters
Problems
Takes m4 to 8 weeks for the effects to be known
Negative side effects are common
dry mouth, sexual dysfunction, blurry vision, constipation
Low fatal dosage-3-4 times average daily prescription
Biological Interventions- MAOI’s
Monoamine Oxidase (MAO)
Enzyme that breaks down monoamine neurotransmitters
MAO Inhibitors block monoamine oxidase
Similarly effective to tricyclics
Problems
Side effects similar to tricyclics
Plus liver damage, weight gain and low blood pressure
Must avoid foods containing tyramine (beer, red wine, cheese, chocolate)
Bad interactions with many other prescription meds
Biological Interventions- SSRI’s
Specifically block reuptake of serotonin
Fluoxetine (Prozac) is the most popular SSRI
No more effective than other meds, but:
Faster symptom relief (2-6)
less severe side effects
overdose is typically not fatal
also relieves anxiety, reduces binge eating, reduces obsessions and compulsions
Problems
side effects- agitation, jitteriness, nausea, decreased sex drive
increased suicidality in some people (especially children and adolescents)
Biological Interventions- Newer Meds
Other medications have been developed for treating depression
SSNRIs
Block reuptake of serotonin and norepinehprine
e.g. Cymbalta, Effexor
Side effects may be more varied than SSRIs
Wellbutrin/Zyban
Blocks reuptake norepinephrine and dopamine
Can help with cognitive and psychomotor symptoms of depression
Sometimes used to treat low sex drive caused by other meds
Biological Interventions- Electroconvulsive Therapy
Electroconvulsive Therapy (ECT)
Involves applying brief electrical current to brain
Results in temporary seizures
Usually 6 to 10 treatments are required
Side effects: short-term memory loss
Uncertain Why ECT works
Relapse is common
Biological Interventions- Other treatments
Light therapy
Helps with seasonal affective disorder
Brain/nerve stimulation
rTMS
Deep Brain Stimulation
Vagal Nerve stimulation
Psychological Interventions- Psychodynamic Therapy
Depression results form unconscious grief over real or imagined losses, compounded by excessive dependence on other people
Therapy uses same basic procedures for all psychological disorders:
Free association
Therapist interpretation
Research has mixed findings
Psychological Interventions- Behavioral Therapy
Depressed mood is result of lack of positive reinforcemnt in dat-to-day life
Use behavioral principles to change this
Reintroduce pleasurable activities
Appropriately reinforce/punish behavior
Teach social skills
Teach “mood management” skills for stress
Only limited effectiveness when 1 technique is applied
Combine 2 or 3= better results
Psychological Interventions- Cognitive Therapy
Depression cause by a pattern of negative thinking
Maladaptive attitudes→ “cognitive triad”
Negatively viewing oneself, the world, and the future
These biased views combine with illogical thinking to produce automatic thoughts
Therapy=Identity and Change negative thinking
4 Phases (usually 6-16 sessions):
Increase activates and elevate mood (behavioral)
Identify negative thinking and biases
Challenge automatic thoughts
Change primary attitudes/beliefs
Psychological Intervention-Interpersonal Therapy
Interpersonal Therapy (IPT)
This model holds that four interpersonal problems may lead to depression and must be addressed
Interpersonal loss
Interpersonal role dispute
interpersonal role transition
Interpersonal deficits
Studies suggest that IPT is as effective as cognitive therapy treating depression
Psychological Interventions- Couples therapy
The main type of couples therapy is behavioral marital therapy (BMT)
Focus is on developing specific communication and problem-solving skills
If marriage is conflictual, BMT is as effective as other therapies for reducing depression
Bipolar Mood Disorder- Manic Episode
Must have manic or hypomanic mood episodes
Manic episodes include at least 1 week of abnormal and persistent
Elevated,
Expansive, or
Irritable Mood
Hypomanic Episode
DSM Criteria same as manic episode except:
A period of persistently elevated, expansive, or irritable mood, lasting at least 4 days
Not severe enough to cause marked impairment in functioning or require hospitalization. No psychotic features
Bipolar 1 Disorder
Major depressive episode + manic episode
Bipolar 2 Disorder
Major depressive episode+ hypomanic episode
Cyclothymic Disorder
Hypomania+ mild depression
Contributing Factors
Biological factors
strong genetic link
Dysregulation of monoamines
Increase during mania
Decrease during depression
Abnormal brain structure
Amygdala and Prefrontal cortex
Abnormal brain activation
Striatum
Part of reinforcement system (ex: scrolling through tiktok)
What Causes Mania?
Psychological Factors
Reward sensitivity
Stress
Rhythm/routine change
Treatments for Bipolar Disorder
Biological Interventions
Lithium is a common salt
Primary drug of choice for bipolar disorders?
Acts as a “mood stabilizer”
Reduces suicidality
Side effects may be severe
Dosage must be carefully monitored
Why lithium works remains unclear
Anticonvulsants are also effective
Tegretal, Depakote
Atypical antipsychotics also used
Zyprexa, Geodon
Psychotherapy
Therapy focuses
Education
medication management
Social skills
relationship issues
depressive symptoms
maintaining daily stability
Research suggests it helps
reduce hospitilization
improves social functioning
increases clients’ ability to obtain and hold a job