pt 2
Overview of Viruses and Cytotoxic T Cells
Viruses invade host cells, utilizing cellular mechanisms to replicate and produce viral proteins.
The viral proteins are presented on the cell surface, alerting the immune system to infection.
The replication of viruses can lead to the destruction (lysis) of host cells, promoting the spread of infection.
The virus functions like a "come and kill me" signal, indicating the need for immune response activation to prevent further viral spread.
Cytotoxic T Cells and Activation
Cytotoxic T (T c) cells play a crucial role in identifying and destroying infected cells.
Inactive T c cells recognize specific shapes known as antigens, which are displayed by body cells.
Each T cell expresses a unique T cell receptor (TCR) that has specificity for a single antigen shape.
T cell activation process:
Recognition of Antigen:
Activated by locking onto the antigen presented on the MHC Class I molecule of an infected cell.
Co-stimulation:
Requires interaction with CD8 (cluster differentiation protein 8) to confirm activation.
Upon successful binding and co-stimulation, the T cell proliferates in a process known as clonal expansion.
Clonal Expansion and Memory Cells
Clonal expansion results in the replication of T c cells that recognize the same antigen, significantly increasing their numbers (e.g., from 1000 to 3000).
Some T c cells become memory T cells, which remain inactive but are primed for future encounters with the same pathogen.
Mechanism of Action of Cytotoxic T Cells
Upon activation, T c cells execute infected cells via:
Cytokine Release:
Stimulates apoptosis (programmed cell death) in the infected cells.
Perforins:
Proteins that perforate the infected cell membrane, leading to a loss of cellular integrity and eventual cell death.
Lymphotoxins:
Toxic proteins that disrupt metabolic functions within the infected cell, further promoting cell death.
Role of Helper T Cells
Helper T (T h) cells are essential for coordinating the immune response.
T h cells recognize antigens presented by MHC Class II molecules on antigen presenting cells (APCs).
T h cells also express CD4, distinctive from the CD8 of T c cells.
Activation of T h cells leads to:
Clonal expansion and differentiation into various effector cells, including cytokine-secreting cells that help B cells.
Activation of B Cells
B cells are produced and mature in the bone marrow.
Each B cell expresses a unique antibody on its surface, which serves as a receptor for antigens.
B cell activation involves:
Antigen Recognition:
B cells bind to specific antigens via their antibodies and endocytose them, processing the antigen and presenting it on MHC Class II molecules.
Helper T Cell Activation:
B cells require help from activated T h cells, which provide signals through cytokine release and direct interaction to promote B cell proliferation and differentiation.
Upon activation, B cells undergo clonal expansion and can develop into:
Plasma Cells:
Specialized to produce and secrete antibodies into circulation, effectively neutralizing pathogens.
Memory B Cells:
Remain in a dormant state for quicker responses upon re-exposure to the same antigen.
Antibody Structure and Function
Antibodies (also known as immunoglobulins) consist of:
Two heavy chains and two light chains, forming a Y-shaped structure.
Antigens are bound at the tips, determining specificity through variable regions.
Antibodies facilitate immune response by:
Marking pathogens for destruction by phagocytes.
Binding to antigens to inhibit their function.
Activating complement pathways to lyse pathogens directly.
Memory Cells and Immune Response Regulation
Memory T and B cells are crucial for rapid responses during subsequent infections by the same pathogen.
Regulatory T (T reg) cells function to prevent overactivation of the immune system, secrete cytokines that help slow down immune responses after an infection is cleared.
Conclusions on Immune Mechanisms
Understanding the detailed interactions between various immune cells (e.g., T c, T h, B cells) enhances comprehension of effective immune responses and the management of infections.
The immune system's complexity underscores the necessity of rigorous regulatory mechanisms to maintain balance and prevent pathological conditions like autoimmune diseases.