Hunger and Eating
Module 4: Motivation
Chapter 9: Hunger Eating Health
Chapter Overview
A. Digestion, Energy, Storage
B. Theories of Hunger and Eating
C. Factors That Determine What, When, and How Much We Eat
D. Body Weight Regulation
E. Obesity
Chapter Details
A. Digestion, Energy, Storage
Define:
Digestion is the process by which food is broken down into smaller components that can be absorbed into the bloodstream. This process involves both mechanical and chemical breakdown of food.
Steps in Breaking Down Food:
Ingestion: Taking in food via the mouth.
Digestion: Mechanical and enzymatic breakdown into simpler forms (e.g., proteins to amino acids).
Absorption: Nutrients are absorbed through the intestines into the bloodstream.
Excretion: Remaining waste is eliminated from the body.
Food extraction (what we extract from fats, proteins, complex carbs):
Fats: Extracted fatty acids and glycerol.
Proteins: Extracted amino acids.
Complex Carbohydrates: Extracted simple sugars (e.g., glucose).
Forms of Energy Storage:
Glycogen (stored in liver and muscle tissue), triglycerides (stored in adipose tissue).
What two factors control energy metabolism:
Hormones (e.g., insulin and glucagon).
Nutrient availability (availability of glucose and fatty acids).
Three phases of energy metabolism:
Cephalic Phase:
Define: The initial phase of energy metabolism that occurs before food intake, activated by the sight, smell, or taste of food.
Insulin is high during this phase, as the body prepares for incoming energy.
Absorptive Phase:
Define: The phase following food intake where the body absorbs and utilizes nutrients.
Glucagon levels low during this phase.
Cephalic and Absorptive phase promotes:
Moves energy (fats, amino acids, glucose) from blood to storage.
Converts glucose to glycogen and fat.
Converts amino acids to protein.
Stores glycogen in liver and muscle.
Fat is stored in adipose tissue.
Protein is stored in muscle.
Fasting Phase:
Define: The phase during which food intake is absent and the body relies on stored energy.
Glucagon levels high during this phase.
Insulin levels low.
Promotes:
Converts stored fats to energy.
Converts glycogen to glucose as a primary fuel source.
B. Theories of Hunger and Eating
Set-Point Assumption:
What is it?
The idea that the body has a biologically predetermined weight range that it tries to maintain, often referred to as a "set point".
Three Components:
1) Sensory inputs related to hunger and satiety.
2) Biological mechanisms like metabolism.
3) Behavioral aspects of eating.
What is a negative feedback system?
A self-regulating process where deviations from a desired norm (set point) trigger actions that counteract such changes to return to that norm.
Two Examples of Set-Point Theories:
Body weight regulation often involves homeostatic mechanisms of hunger and satiety.
Weight alterations trigger physiological responses aimed to restore the original weight.
Issues with Set-Point Theory:
Variability in individual body weight regulation (some people maintain weight outside of the set-point).
Influence of environmental factors, such as the availability of high-calorie foods.
Changes in metabolism over lifetime and factors like age or health status that can alter the set-point.
Positive Incentive Perspective:
Emphasizes the role of expected pleasure from food (positive incentives) rather than just physiological regulation, accounting for societal and cultural influences on eating behavior.
C. Factors That Determine:
A. What We Eat:
Learned Preferences and Aversion:
People develop specific likes and dislikes for various foods based on experience and exposure.
Learning to Eat Vitamins and Minerals:
i. Design and outcome for thiamine deficient diets in rats:
Studies showing rats can learn to prefer foods containing essential nutrients when deprived.
ii. Implications for humans:
Highlights the importance of nutrient awareness in human dietary habits.
B. When We Eat:
Premeal Hunger:
The sensation of hunger that precedes meals, influencing eating time and amount.
Pavlovian Conditioning:
i. Rat Study:
Research demonstrating that environmental cues can trigger hunger responses in conditioned rats.
C. How Much We Eat:
Satiety Signals:
Three signals that indicate fullness: hormonal signals, sensory cues, and gastric responses.
How the following affect satiety signals:
1. Appetizer Effect:
Initial small quantities of food can increase subsequent hunger and overall food consumption.
2. Serving Size:
Larger portion sizes often lead to increased consumption.
3. Social Influences:
Eating behaviors may adjust based on social contexts (e.g., eating more in groups).
4. Sensory Specific Satiety:
The phenomenon where a person becomes satiated with specific foods while still desiring other types of food.
Cafeteria Diet (Experiment):
Studying dietary choices leads to more consumption when a variety of food is available, reflecting adaptive consequences.
D. Obesity
Reasons for Concern:
Obesity is linked to numerous health issues, including diabetes, heart disease, and overall mortality.
Causes of Obesity:
Evolutionary Factors:
Human evolution has favored fat storage as a survival mechanism.
Cultural Factors:
Societal norms and food availability influence eating behaviors and perceptions of body image.
Why Some but Not Others Become Obese:
Consumption Patterns:
Differences in types, quantities, and frequencies of food consumption.
Energy Expenditure:
a. NEAT (Non-Exercise Activity Thermogenesis):
Daily energy expenditure from non-exercise activities, influencing overall energy balance.
Microbiome:
The gut microbiome composition can influence body weight and metabolic processes.
Genetics:
Genetic predispositions affect metabolism, hunger signals, and fat storage.
Weight Loss Programs:
Dieting Cycle:
Issues with yo-yo dieting and its impacts on metabolism and mental health.
Physical Exercise:
a. NEAT:
Incorporating more physical activity into daily routines to enhance energy expenditure.
Treatments for Obesity:
Gastric Bypass:
A surgical procedure that alters digestion to limit food intake and absorption.
Gastric Band:
A device placed around the upper part of the stomach to restrict food intake.
Gastric Sleeve:
A surgery that removes part of the stomach to reduce its size and capacity.
GLP-1 Agonists:
Medications that mimic incretin hormones to promote insulin secretion and decrease appetite, aiding weight loss.