Molecular Basis of Inheritance
Molecular Basis of Inheritance
Introduction
Presenter: Bethanie-Michelle Statler
Griffith Experiments
Key Discoveries: Frederick Griffith discovered that a molecule within cells provides genetic material.
Types of Bacteria:
Pathogenic bacteria (S cells), capable of causing disease.
Harmless bacteria (R cells), non-pathogenic.
Experimental Results: Mixing heat-killed S cells with R cells resulted in the death of mice.
Transformation: The process where a cell incorporates foreign DNA into its genome, leading to genetic changes.
Viral DNA and Transforming Substance
Key Contributors: Oswald Avery, Maclyn McCarty, and Colin MacLeod identified the transforming substance as DNA.
Bacteriophages (Phages): Viruses that specifically infect bacteria, composed of either DNA or RNA enclosed by a protein coat.
DNA as Genetic Material
1952 Experiment by Alfred Hershey and Martha Chase: Demonstrated that DNA is the genetic material of phage T2.
Experiment Details - Batch 1:
Labeled phages with radioactive sulfur ($^{35}S$) in phage protein.
Phages infect cells; agitation frees outside phage parts from cells.
Cells subjected to centrifugation; radioactivity (phage protein) found in liquid.
Experiment Details - Batch 2:
Labeled phages with radioactive phosphorus ($^{32}P$) in phage DNA.
Similar procedures followed, resulting in radioactivity (phage DNA) being found in the pellet.
Structure of DNA
Composition of DNA: DNA is a polymer of nucleotides, each nucleotide consists of:
A nitrogenous base
A sugar
A phosphate group
Nitrogenous Bases:
Adenine (A)
Thymine (T)
Guanine (G)
Cytosine (C)
Chargaff’s Rules (1950):
DNA composition varies among species.
In any species, the quantity of A equals T (A=T), and quantity of G equals C (G=C).
DNA Double-Strand Structure
Covalent Bonds: Nucleotides linked via covalent phosphodiester bonds in one strand.
Hydrogen Bonds: Nitrogenous bases form hydrogen bonds with a complementary second strand.
Antiparallel Nature: DNA strands run in opposite directions (3' to 5' and 5' to 3').
X-ray Crystallography and DNA Discoveries
Contributors: Maurice Wilkins and Rosalind Franklin employed X-ray crystallography to study DNA.
Franklin's Contribution: Produced a critical picture of DNA, aiding in structural understanding.
Watson and Crick’s Model: Deduced the structure of DNA as a double helix using Franklin's data. Recognized for their contribution with the Nobel Prize in 1962.
Key Features of DNA Structure
Dimensions:
Distance between base pairs: 0.34 nm
Width of DNA double helix: 2 nm
Complete turn of the helix: 3.4 nm
Base Pairing:
A pairs with T
G pairs with C
DNA Replication - Semiconservative Model
Replication Process: Watson and Crick’s model predicts each daughter molecule consists of one old strand and one new strand.
Competing Models:
Conservative model: Parent strands rejoin post-replication.
Dispersive model: Each strand is a mix of old and new DNA.
Experimental Support: Matthew Meselson and Franklin Stahl's experiments supported the semiconservative model.
Mechanism of DNA Replication
Origins of Replication: Sites where DNA strands are separated, forming a replication “bubble”.
Directionality of Replication: Replication occurs bidirectionally from each origin until the entire molecule is copied.
Replication Fork: The Y-shaped region where new DNA strands elongate.
Key Enzymes:
Helicases: Untwist the DNA double helix.
Single-strand Binding Proteins: Stabilize single-stranded DNA.
Topoisomerases: Correct the overwinding ahead of replication forks by breaking, swiveling, and rejoining DNA strands.
DNA Polymerization Process
Synthesis Direction: DNA polymerase can only synthesize DNA in the 5' to 3' direction.
Role of Primase: Synthesizes a small RNA primer to initiate DNA synthesis, generally 5-10 nucleotides long.
DNA Polymerase Activity: DNA polymerase (DNA pol III) begins elongating DNA by adding nucleotides to the 3' end of an RNA primer. DNA pol I replaces RNA primers with DNA.
Antiparallel Elongation Details
Leading Strand: Synthesized continuously towards the replication fork.
Lagging Strand: Synthesized discontinuously in fragments called Okazaki fragments, starting closest to the origin of replication.
Joining of Fragments: DNA ligase joins Okazaki fragments together after synthesis.
DNA Replication Checkpoint Questions
Untwisting the Double Helix: Which enzyme? (Helicase)
Adding Nucleotides: Which enzyme adds DNA nucleotides to the 3’ end? (DNA pol III)
Preventing Overwinding: Which enzyme prevents overwinding of the DNA double helix? (Topoisomerase)
Adding RNA Primers: Which enzyme adds an RNA primer? (Primase)
DNA Proofreading and Repair Mechanisms
Proofreading by DNA Polymerases: Corrects errors by replacing incorrect nucleotides during DNA synthesis.
Mismatch Repair: Repair enzymes replace incorrectly paired nucleotides that evade proofreading mechanisms.
Normal Mutation Rates: A low rate of mutation contributes to genetic variation.
Causes of DNA Damage: Harmful chemical agents (e.g., cigarette smoke) and physical agents (e.g., X-rays).
Nucleotide Excision Repair: A nuclease removes damaged DNA sections and replaces them with proper nucleotides.
Challenges with Ends of Linear DNA
Limitations of Replication Machinery: Cannot complete the 5' ends of daughter DNA strands, leading to progressively shorter strands.
Prokaryotic vs Eukaryotic DNA: Circular chromosomes in prokaryotes avoid this issue.
Telomeres in Eukaryotic Cells
Function of Telomeres: Special nucleotide sequences at the ends of eukaryotic chromosomes.
Conservation of Genes: Telomeres do not prevent DNA shortening but postpone erosion of essential genes.
Telomerase Activity: Enzyme found in germ cells (egg and sperm) catalyzing telomere lengthening. Shortening of telomeres may help prevent cancer by limiting cell division.
Cancer Cells: Some exhibit telomerase activity, which causes continuous division and survival.
Chromosomal Structure and Organization
Bacterial Chromosomes: Double-stranded, circular DNA associated with minimal protein, supercoiled within a nucleoid region of the cell.
Eukaryotic Chromosomes: Contain linear DNA associated with extensive proteins; the DNA forms chromatin that is compacted to fit into the nucleus.
Histones: Proteins essential for chromatin folding and organization.
Nucleosome and DNA Packing
Nucleosome Structure: DNA wound around a core of eight histones, forming a 10-nm fiber.
Chromatin Types:
Euchromatin: Loosely packed, accessible for transcription.
Heterochromatin: Highly condensed, transcriptionally inactive regions.
Chromatin Organization:
30-nm fiber: Compacted structure formed by histones.
Looped domains: Further compaction, forming metaphase chromosomes, enabling the orderly segregation of genetic material during cell division.