Hypothalamus/pituitary physiology
HUF2-102 Hypothalamus/Pituitary Physiology Notes
Background on the Embryonic Origin of the Pituitary Gland
The pituitary gland (hypophysis) originates embryonically from two distinct structures:
The anterior pituitary (adenohypophysis) develops from an upward evagination known as Rathke’s pouch from the primitive foregut (epithelial tissue).
The posterior pituitary (neurohypophysis) forms as a downward extension of the floor of the diencephalon (neurohypophysial diverticulum) (neural tissue).
Anatomical Position and Neighbouring Structures
The pituitary gland is located in the sella turcica, a bony cavity of the sphenoid bone.
Nearby anatomical structures include:
Optic chiasm
Cranial nerves III (oculomotor), IV (trochlear), VI (abducens)
Structural and Functional Relationship to the Hypothalamus
The functional relationship between the pituitary gland and hypothalamus is critical for hormone secretion control:
Anterior pituitary:
Regulated by the hypothalamic-hypophyseal portal system, which facilitates the delivery of hypothalamic hormones to the anterior pituitary.
Posterior pituitary:
Connected via a fiber tract down the pituitary stalk and contains axonal projections from neuroendocrine cells.
Neuroendocrine Control/Integration Significance
The hypothalamus integrates neural and endocrine signals to achieve regulatory control, with examples including:
Hypothalamus-anterior pituitary interactions
Hypothalamus-posterior pituitary interactions
Influence on the adrenal medulla
Regulation of endocrine pancreas functions
Hypothalamic Hormones and Anterior Pituitary Control
Hypothalamic hormones play essential roles in regulating anterior pituitary hormone secretion:
Example of hypothalamic hormones:
Gonadotrophin Releasing Hormone (GnRH)
Thyrotrophin Releasing Hormone (TRH)
Corticotrophin Releasing Hormone (CRH)
Growth Hormone Releasing Hormone (GHRH)
Somatostatin (GH inhibiting hormone)
Anterior pituitary hormones regulate target glands:
Thyroid Stimulating Hormone (TSH) - targets the thyroid gland
Adrenocorticotropic Hormone (ACTH) - targets the adrenal gland
Feedback Mechanisms in Hypothalamic-Pituitary-Target Gland Axis
The feedback mechanisms include:
Negative feedback: Predominant form of hormonal control;
Involved in the hypothalamic-pituitary-target gland regulation, where target gland hormones inhibit hypothalamic and pituitary secretion (e.g., cortisol inhibiting ACTH).
Positive feedback: Seen in specific conditions, such as the gonadotropin surge in females during the menstrual cycle.
Example:
Estrogen surge leading to positive feedback on GnRH and resulting LH/FSH surge.
Types of Anterior Pituitary Hormones
Glycoprotein Hormone Family:
Includes hormones like TSH, LH, FSH; characterized by α and β subunits, with biological activity determined by the β subunit.
Prolactin-Growth Hormone Family:
Includes Prolactin and Growth Hormone; shares structural similarities in peptide chains and functional activity.
Conditions of Hyperpituitarism and Hypopituitarism
Hyperpituitarism: Overproduction of functions resulting from:
Pituitary tumors (adenomas) which can cause compression effects (mass effect) leading to symptoms such as headaches and visual disturbances (e.g., bitemporal hemianopia, diplopia).
Hypopituitarism: Caused by various factors:
Pituitary destruction: Leads to decreased hormone production.
Sheehan’s syndrome: Postpartum hemorrhage causing pituitary necrosis.
Tumors causing compression on the pituitary stalk resulting in selective loss (e.g., raising prolactin).
Prolactin Regulation
Control of secretion: Regulated by:
Neuroendocrine reflex linked to suckling during lactation
Inhibition by dopamine (PIH) and stimulation by TRH, VIP.
Effects of prolactin:
Supports mammogenesis, lactogenesis, and influences reproductive functions.
Antidiuretic Hormone (ADH) Secretion Control
Control Mechanisms:
Stimulated by increases in plasma osmolality (via hypothalamic osmoreceptors)
Stimulated by decreases in blood volume (detected by baroreceptors)
Inhibition by alcohol and atrial natriuretic peptide (ANP).
Disorders of ADH secretion:
Syndrome of Inappropriate Antidiuretic Hormone Secretion (SIADH): High levels of ADH lead to water retention and hyponatremia.
Diabetes Insipidus (DI): Results in polyuria; centrally or nephrogenically caused.
Oxytocin Actions and Control
Oxytocin regulates:
Uterine contractions during labor through a positive feedback mechanism initiated by cervical stretch.
Milk ejection reflex during breastfeeding triggered by suckling (neuroendocrine reflex).
Control Mechanism: Similar to ADH, oxytocin release can be influenced by sensory inputs from suckling and conditioned reflexes.
Clinical Assessment and Treatment Approaches
Diagnostic Tests for pituitary function:
Hormonal assays to measure anterior pituitary hormone levels in response to hypothalamic stimulation.
Imaging (MRI) for anatomical assessment (tumors, structural anomalies).
Treatment Options for disorders include:
Prolactinomas are treated with dopamine agonists (e.g., cabergoline, bromocriptine).
Diabetes Insipidus requires hormonal replacement or vasopressin analogues, depending on the type.
Management of hyperprolactinemia involves treating underlying causes and normalizing prolactin levels to restore physiological functions.