Corticosteroids
Corticosteroids are for maintenance, and control therapy
Inhaled agents can be used with systemic corticosteroids in severe asthma and may allow for reduction or elimination of systemic corticosteroids for asthma control
Inhaled corticosteroids in combination with other agents are recommended by the ATS and the GOLD for COPD
Intranasal aerosol agents: Management of seasonal and perennial allergic and non allergic rhinitis
Step 2 asthma:
> 2 days/week, not daily
Night awakenings 3-4 times per month
Aerosolized corticosteroids:
Beclomethasone dipropiomate HFA
QVAR
Flunisolide
AeroSpan
Fluticasone propionate/salmeteror
Advair diskus, advair HFA
Used to treat asthma and COPD, 1 puff BID
Fluticasone furoate
Arnuity ellipta
Budesonide
Pulmicort flexhaler
Pulmicort respules
Mometasone furoate
Asmanex twist halter
Asmanex HFA
Ciclesonide
Alvesco
Aerosolized corticosteroids (Combinations):
Budesonide/formoterol, fumarate HFA
Symbicort
Mometasone furoate/formoterol, fumarate HFA
Duelera
Fluticasone furoate/umeclidinium/vilanterol
Trelegy elipta
Budesonide/glycopyrrolate/formoterol
Brezti aerosphere
Brezti
Corticosteroids come from two primary sources:
Naturally occurring hormones produced within the body
Synthetic medications developed in labs
The body naturally produces corticosteroids in the adrenal cortex, the outer part of the adrenal glands located on the top of each kidney
3 types produced by the adrenal cortex:
Glucocorticoids/glucocrticosteroids
Cortisol (Stress, regulates metabolism, controls inflammation, helps blood pressure)
Mineralocorticoids/aldosterone
Regulate body water
Sex hormones
Androgens and estrogens:
Androgen: Male sex hormone (Testosterone) (Can be seen in both males and females)
Estrogen: Female sex hormones (Can be seen in both males and females)
The production and release of these hormones are regulated by the hypothalamic-pituitary-adrenal (HPA) axis, a feedback system involving the hypothalamus, pituitary gland, and adrenal glands.
One of the most significant side effects of treatment with glucocorticoid drugs (exogenous corticosteroids) is adrenal suppression or HPA suppression
HPA suppression with steroid use:
Body cannot distinguish between endogenous and exogenous
Administration of glucocorticoid drugs raises body’s level
Inhibits hypothalamus and pituitary glands
Referred to as HPA suppression or adrenal suppression
It is seen with systemic administration of corticosteroids, it begins after 1 day of treatment, and is significant after 1 week of oral therapy at usual doses
A primary reason for using aerosolized glucocorticoids is to minimize adrenal or HPA suppression by minimizing the dosage and localizing the site of treatment
Diurnal steroid cycle:
Production of body’s own glucocorticoids follows rhythmic cycle: Circadian rhythm or diurnal
Cortisol levels are highest in the morning at 8 am
Jet lag and working night shifts would interfere with the diurnal steroid cycle
Inflammation of airway:
Asthma and Chronic Bronchitis are the most common
Treatment: Anti inflammatory agents such as glucocorticoids is important to reduce basal level of airway inflammation
Reduces airway hyper responsiveness and predisposition of acute episodes of obstruction
Asthmatic reactions are biphasic
Early phase and late phase
Early phase is caused by immunoglobulin E (IgE)
Response peaks at 15 minutes
Mast cells mediate and release of cytokines recruit other inflammatory cells to cause late phase reaction
Response occurs after 6-8 hours, can last up to 24 hours
Corticosteroids effects:
Steroids cause leukocytosis through several mechanisms that affect how white blood cells circulate in the body
Steroids cause neutrophils that are typically stuck to the walls of blood vessels to detach and enter the main circulation
They inhibit the movement of white bloods cells from the bloodstream into the tissues where they would normally fight infection or inflammation
Steroids prolong the lifespan of neutrophils, meaning fewer are removed from circulation
They stimulate the bone marrow to release more white blood cells into the blood stream
Effects on B receptors:
Restore responsiveness to B adrenergic stimulation
Enhance B receptor stimulation
Hazards and side effects of systemic administration:
Suppression of the HPA axis
Immunosuppression
Psychiatric reactions
Cataract formation
Myopathy of striated skeletal muscle
Peptic ulcer
Fluid retention
Hypertension
Increased WBCs
Dermatological changes
Slowing of growth (Children)
Hyperglycemia
Hazards and side effects of aerosol administration:
Major:
HPA suppression
Loss of bone density
Growth restriction (Children)
Potential:
Adrenal insufficiency
Allergic inflammation after cessation
HPA suppression
Growth reduction (Children)
Topical (Local) side effects with aerosol administration:
Most common:
Oropharyngeal candidiasis (Thrush)
Dysphonia (Voice alteration)
What to do?:
Use minimal dose
Use reservoir
Rinse mouth
Aerosol steroids in:
Asthma:
Early use in asthma
Acute and sever
COPD:
Relieve symptoms
Little to no effect on FEV1
Nonsteroidal Antiasthma Agents:
Cromolyn-like-drugs (Mast cell stabilizers)
Anti-leukotrienes
Monoclonal antibodies
Management of mild persistent asthma
General indications:
Cromolyn and Anti-LT as alternatives to ICS in asthma requiring step 2 care
Cromolyn is often used with infants and young children as alternatives to ICS in asthma requiring step 2 care because of safety profiles of ICS
Anti-leukotrienes can be useful in combinations with ICS to reduce the steroid dose
Cromolyn-like-drugs:
Cromolyn (Generic only)
Anti-leukotrienes:
Zafirlukast (Accolate)
Montelukast (Singulair)
5-LO inhibitor: Zileuton (Zyflo; Zyflo CR)
Monoclonal antibodies:
Omalizumab (Xolair)
Benrailzumab (Faserna)
Mepolizumab (Nucala)
Reslizumab (Cinqair)
Dupilumab (Dupixent)