Exam 1
Complete Health History (Subjective Data)
Biographic Data
Name, address, age, gender, race, occupation, etc.
Reason for Seeking Care
Not a diagnostic statement
Source of History
Judge reliability of informant
Present Health or History of Present Illness (HPI)
Collect all provided data and identify eight critical characteristics
Location, character (quality), quantity (severity), timing, setting, aggravating or reliving factors, associated factors and patient’s perception
Use measurable standards and/or patient’s own wards as qualifiers
Critical Characteristics
Location
Character or quality
Quantity or Severity
Timing— onset duration, and frequency
Setting
Aggravating or relieving factors
Associated factors
Patient’s perception
Past Medical History*
Family History
Review of Systems (ROS)
Evaluate past and present state of each body system
Notes on body system
Evaluate health promotion practices
Record either presence or absence of symptoms (no writing negative)
NO OBJECTIVE data
Focus on health promotion
Functional Assessment
OBJECTIVE measures
Mental Status— Level of Consciousness
Alert
Lethargic- Drifts off to sleep when not stimulated
Obtunded- Sleep most of the time, difficult to arouse
Stupor/Semi-coma— unconscious; responded to persistent vigorous shake or pain
Coma
Delirium
General Survey (Objective Data)
Study of the whole person
Physical Appearance
Age: What age they look like or stated age
Sex
Level of consciousness
Skin color
Facial features
Overall appearance
Stature
Nutrition
Symmetry
Posture
Position
Gait
Behavior
Facial expression
Mood and affect
Speech
Dress
Personal hygiene
Height
Shoeless
BMI
Can indicate protein-calorie malnutrition
Malnutrition
Marasmus - Protein-calorie malnutrition
Decreased anthropometric measurements (i.e. weight loss and subcutaneous fat and muscle wasting
Anorexia, bowel obstruction, cancer cachexia
Waist Circumference
Assess body fat distribution as indicator of health risk
Excess abdominal fat is an independent risk factor for disease, over and above that of BMI
Note measurement at end of normal expiration
Android (upper body obesity):
1.0 or greater in men or 0.8 or greater in women
Risk for obesity-related diseases and early mortality
Metabolic Syndrome
Three or more of the following risk factors are present
Fasting plasma glucose level greater than or equal to 100 mg/dl blood pressure greater than or equal to 100 mg/dl
blood pressure greater than or equal to 130/85mm hg
waist circumference greater than or equal to 40 inches for men and 35 inches for women
HDL cholesterol less than 40 in men and less than 50 in women (high- density lipoproteins- “Good)
triglyceride levels greater than or equal to 150 mg/dl
Nutrition Assessment: Subjective Data
Eating patterns
Usual weight
Changes in appetite, tase, smell, chewing, and swallowing
24hr dietary recall for average day (type, amounts, liquids)
Different Types of Pain (Can originate from the PNS/CNS)
Acute
Chronic
Nociceptive (ex: pain from pinching)
Neuropathic (ex: phantom pain)
Assessment of Pain
Distinguishing sensations
Different responses to analgesics
Structural Plasticity / Reorganization of Pain Pathways: result from long-term uncontrolled pain — cells altered in size and function in dorsal horn
associated with nociceptive hypersensitivity
Nociception four phases
Transduction— Chemicals release
Transmission— Chemical reach CNS and move from spinal cord to brain
Perception— Pain perception
Modulation— Brainstem neurons block pain impulse
Sources of Pain
Referred- Originated at one site but is felt at another (ex: Acute coronary syndrome radiates to left arm or neck
Visceral- “Think visceral organs”
dull, deep, squeezing, craming
ANS associated vomiting, nausea, pallor, and diaphoresis
Somatic- musculoskeletal tissues or body surface
localized / easy to pinpoint
Deep somatic- blood vessels, joints, tendons, muscles and bone-aching/throbbing
Cutaneous- integumentary/subcutaneous
Breakthrough pain- transient spike in pain level with moderate to severe intensity in an otherwise controlled pain syndrome from…
End of dose medication failure
Result of incident or episodic pain
Pain Assessment / Subjective Date and Tools
Initial Pain Assessment
Onset
Intensity
Quality
Duration
Frequency
Location
Aggravating factors
Relieving factors
Skin
Skin Function (besides the obvious)
Temp reg
Identification
Skin Layers
Epidermis— Basal cell layer forms new skin cells; replaced every 4 weeks
Melanocyte related to melanin
Dermis— Supportive layer consisting of CT or collagen
Subcu— composed of adipose, energy, insulation for temp control, protective soft cushioning effect, mobility over structures underneath
Skin / Hair / Nails
Vellus— fin faint hair covers most of body
Terminal hair— thicker, darker, scalp
Can exist in face / chest in males
Sebaceous (oil) glands— abundant on scalp, forehead, face, and chin
Sweat glands
Eccrine— sweat
Apocrine— open into hair follicles, thick milky secretion, bacteria creates musky body odor
Nails— made of keratin
Physical Examination of Hair and Nails
Separate intertriginous areas (areas with skinfolds) such as under large breasts, obese abdomen, and groin, and inspect them thoroughly
Assess skin as one entity
Summary Checklist
Inspection of the skin, hair, and nails
Includes shape, contour, and consistency
Palpation of skin, hair, and nails
Temp and texture
Edema, mobility, and turgor
Note presence of lesions
Includes configuration / Distribution
Inspection / Palpation of Hair
Color
Texture
Distribution
Lesions
Inspection / Palpation of Skin
Color
Temperature
Take into account the temp of the environment
Moisture
Includes diaphoresis
Texture
Thickness (look for calluses)
Edema
Mobility / Turgor
Vascularity / Bruising
Chronic Arterial Limb Ischemia | Chronic Venous Insufficiency |
Pain— severe at rest; toes, fore-feet, heels | Aching- Relieved by elevation or rest |
Worse at night | Worse later in the day |
Claudication: muscle pain, cramping, or weakness | Cramping- not activity dependent |
Pale feet, rubor red, red to bluish color | Woody, brawny, brown pigmented |
Elevation pallor (pale) & Dependent rubor | Multiple factors |
Skin— thin, scaly, dry; Thick Nails | Veins full if leg slightly dependent |
Hair loss over calf, ankle, foot | Skin- Thick, tough, scarring |
Numbness, Burning, “Toothache” | Premenstrual, salt and water retention |
Pulses diminished to absent | Itching and burning |
Ulcers— Distal, concentric, pale | Pulses intact |
Little or no edema | Ulcers- Distal calf, irregular, pink bed, large yellow drainage |
Edema moderate to severe |
Erythema - intense redness of the skin due to excess blood in dilated superficial capillaries, as in fever or inflammation
Carotenemia - yellow pigmentation of the skin (palm, soles and nasolabial fold secondary to excretion by sebaceous glands in sweat) associated with increased blood carotene (Vit A) levels (carrots, squash, pumpkin, breast milk)
Jaundice due to excess bilirubin (includes yellow sclera)
Skin Lesions
Color
Elevation: flat, raised, or pedunculated
Pattern or Shape: the grouping or distinctness of each lesion
Size in centimeters: use a ruler
Location and distribution: generalized or localized to area of specific irritant
Exudate: color and odor
Inspection / Palpation: Nails
Shape and contour
View index finger at its profile and note angle of nail base; it should be about 160 degrees
Consistency
Observe for smooth, regular, not brittle or splitting, uniform nail thickness
Color
Transulecent nail plate to pink nail bed below
Note ethnic variations
Capillary refill
index or middle finger at heart level
depress for at least 5 sec
Clubbing
nail bed straightens to 180 degrees
pulmonary disease
ABCDEF Skin Assessment
A: asymmetry
B: border irregularity
C: color variations
D: diameter greater than 6 mm
E: elevation or evolution
F: funny looking
Shapes / Configuration of Lesions
Confluent
Lesions run together (ex: hives)
Discrete
Distinct and separate
Grouped
Cluster (ex: eczema)
Linear
Scratch, streak, line, or stripe
Zosteriform
Follow unilateral nerve route (ex: herpes zoster)
Primary Skin Lesions
Tumors
Large in diameter, firm or soft, deeper into dermis, may be benign or malignant
Urticaria (hives)
Wheals (superficial, raised, transient, erythematous, slightly irregular from
edema (mosquito bite, allergic reaction) coalesce to form extensive
pruritic reaction.
Vesicles
Elevated (up to 1 cm)
Containing fluid (clear serum)
i.e. herpes; blister
Cysts
Encapsulated fluid filled cavity (bigger than vesicles)
Pustules
Macule
Just a color change
Flat and circumscribed (less than 1 cm)
ex: Freckle
Papule
Solid, elevated, circumscribed less than 1 cm diameter
Mole or wart
Secondary Skin Lesions
Debris
Crust— Thickened dried out exudate
Scale— Compact flakes (shedding)
Break
Fissures— Linear crack with abrupt edges extending into dermis (“look like cracks)
Erosions— Shallow depression (scooped out)
Ulcers— Deeper depression extending into dermis with irregular shape, may bleed, leaves scar
Excoriations— Self-inflicted abrasion that is superficial
Atrophic scars— Depressed scars
Lichenifications— From intense scratching → thickened skin → tightly packed papules
Keloids— Benign excess of scar tissue beyond original injury
Pressure Injuries (PI)
Pressure ulcer
Decubitus ulcer
Deep depression extending into dermis
Stages
Non-blanchable erytheme
Partial-thickness skin loss
Full-thickness skin loss
Full-thickness skin/tissue loss
Vascular Lesions
Caused by blood flowing out of breaks in the vessels
Petechiae: tiny punctate hemorrhages indicating
abnormal clotting factors (check mouth, buccal mucosa,
conjunctivae)
Purpura: > 3mm, flat, red to purple macular hemorrhage
seen with thrombocytopenia and old age as blood leaks
from capillaries in response to minor trauma
Contusion: (Bruise) red-blue or purple immediately then
blue-green, yellow and brown from trauma
Non Cancerous Findings
Seborrheic Keratosis: dark, greasy and “stuck on” Develop
mostly on trunk, hands and face on both unexposed and sun
exposed areas. Do not become cancerous.
Actinic keratosis: red-tan scaly plaques, raised roughened.
May have silvery white scale adherent to plaque. Occurs on
sun-exposed surfaces and are directly related to sun
exposure. Premalignant and may develop into squamous
cell carcinoma.
Senile lentigines (lenltigo): common variation of
hyperpigmentation, liver spots, flat brown macules, extensive
sun exposure and are not malignant and do not require
treatment.
Malignant Skin lesions (From UV Sunlight / Tanning Bed Radiation)
Basal Cell Carcinoma: small pink or red papule with pearly
translucent top and broken blood vessel at center then
develops rounded, pearly boarders with central red ulcer or
large open pore with central yellowing- slow growing
Squamous Cell Carcinoma: arises from actinic keratoses or
denovo. Erythematous scaly patch with sharp margins, 1 cm
or more, develops central ulcer and surrounding erythema-
fast growing
Malignant Melanoma: Brown, tan, black, pink-read, purple or
mixed pigmentation. Irregular boarders, scaling, flaking ,
oozing
Wounds:
Abrasion: scrapes from friction on rough surface
Incision: clean cut from sharp object (ex: scalpel)
Laceration: irregular tear
Puncture
Avulsion: characterized by flap
Assess:
1. approximation of edges
2. color,
3. edema,
4. drainage
5. exudate
6. odor
7. Pain
Thorax and Lungs
Anterior Thoracic Landmarks
Suprasternal notch: U-shaped in between clavicles
Sternum: “breastbone” / manubrium
Manubriosternal angle: Angle of Louis; Crack
Angle of Louis marks site of tracheal bifurcation into right and left main bronchi
Costal Angle: right and left costal margins form an angle (^)
Lobes of the Lung
Posterior Chest: almost all lower lobes
Lateral chest: lung tissue goes from apex of axilla down to seventh / eighth rib
Left lung: no middle lobe / very little lower lobe (mostly upper and middle)
Thorax and Lungs Examination
Inspection
Thoracic cage, respirations, skin color, and condition
Palpation
Symmetry and tactile fremitus (“99" / “Blue moon”)
Detection of any lumps, masses, or tenderness
Percussion
Resonance
Assess breath sounds (presence/quality), normal and note any abnormal/adventitious sounds
Inspection of Anterior Chest
Shape/configuration of chest wall
Note respiratory effort
Palpation of Anterior Chest
Tactile Fremitus (“99”)
Decreased fremitus with pneumothorax
Increased fremitus with severe pneumonia
Note tenderness/lumps
Note skin mobility, turgor, temp, and moisture
Hyperinflation of lungs = chronic emphysema and asthma
Percussion of Anterior Chest
Bilateral comparison
Do not percuss directly over female breast; displace breast
Borders of cardiac dullness normally found on anterior chest
Right hemithorax = liver dullness located in fifth intercostal space, right midclavicular line
Left hemithorax = over gastric space
Resonance is normal lung sound in all fields
Breath Sounds
Three types of breath sounds heard normally in adults and older child
Bronchial- trachea and larynx
Bronchovesicular- over major bronchi
Vesicular: over peripheral lung fields, bronchioles and alveoli
Note description of characteristics
Auscultation Breath Sounds
Crackels (Rales)
Fine
Popping (Inspiration)
Obstructive disease
Chronic bronchitis, emphysema, NOT cleared by coughing
Coarse
Velcro (Inspiration / Expiration)
Pulmonary edema, pneumonia, terminally ill with depressed cough reflex
Reduced by coughing
Wheezes (Ronchi)
High pitched sibilant
Squeaking, musically (Expiration, Inspiration secondary)
Acute asthma or chronic emphysema
Low Pitched
Snoring (cleared somewhat with coughing)
Airflow obstruction, bronchitis, bronchus obstruction
Stridor Sound: Rooster
Upper air way obstructions or swelling, croup or foreign body inhalation
Posterior Chest
Inspection
Shape and configuration of chest wall
Position the person take to breathe
Lesions; inquire about changes
Palpation
Symmetric
Tactile fremitus (“99” or “blue moon”)
Percussion
Resonance is low-pitched, clear, hollow sound that predominates in healthy lung tissue in adult
Auscultation
Tactile Fremitus
Increased:
Occurs with conditions that increase lung tissue density (better conduction medium for vibrations)
Pneumonia
Decreased:
Obstructs transmission of vibrations
Obstructed bronchus, pleural effusion, thickening pneumothorax, emphysema
Voice Sounds Auscultation
Normal voice transmission is soft, muffled, and indistinct
Increased lung density / transmission of voice sounds = Disease
Respiratory Patterns
Tachypnea- >24 per min; fever, fear, exercise, pneumonia, and respiratory insufficiency
Bradypenea- <10 per min; drug-induced depression or increasd intracranial pressure and diabetic coma
Chronic Obstructive breathing- normal inspiration and prolonged expiration; chronic obstructive lung disease
Cheyne-Stokes Respiration: cycle respirations gradual wax and wane in pattern with increasing rate and depth and then decreasing
Periods of apnea
Severe heart failure
Drug overdose
Increased intracranial pressure
May be normal for infants / older adults
Abdomen
Internal Anatomy
Peritoneum lines abdominal wall (parietal)
Covers surface (visceral) of most organs
Divided into 4 quadrants
Anatomic Locations of Organs in the Right Upper Quadrants
Liver
Gallbladder
Left Upper Quadrant
Stomach
Spleen
Left lobe of liver
Right Lower Quadrant
Appendix
Right Ovary Tube
Culture and Genetics
Lactose Intolerance
Lack of lactase
Estimated incidence of lactose intolerance is
20% and 30% of whites, 70% of Mexican Americans, and 80% of blacks and 100% American Indians
Celiac Disease (autoimmune disorder)
Intolerant of gluten roughly 1% of 4$ with a diagnosis-most affect Punjab region of India
Subjective Data
Appetite
Dysphagia
Food Intolerance
Abdominal Pain
Nausea and Vomiting
Bowel habits
Past abdominal history
Medications
Nutritional assessment
Development Competence: Aging
Changes of GI system occur with aging, but most do not significantly affect function as long as no disease is present
Salivation decreases = dry mouth / lack of taste
Esophageal emptying and gastric acid secretion are delayed
Incidence of gallstones in creases with age
Drug metabolism impaired
Abdomen Examination (Summary)
Inspection
Contour, symmetry, umbilicus, skin, pulsation or movement, hair distribution, and demeanor
Auscultation
Bowel wounds; note any vascular sounds
Palpation
Light and deep palpation in all four quadrants (for liver and spleen)
Objective Data
Position for comfort to enhance abdominal wall relaxation
Examine painful areas last to prevent guarding
Auscultate prior to palpation
Use distraction to keep patient relaxed and facilitate muscle relaxation
Inspection of the Abdomen
Contour
Determine profile from rib margin to pubic bone; contour describes nutritional state and normally ranges from flat to rounded
Symmetry
Abdomen should be symmetric bilaterally
Umbilicus
Normal it is midline and inverted, with no sign of discoloration, inflammation, or hernia
Skin
Inspect for pigment change and presence of lesions or scars
Pulsation or Movement
Normally, may see pulsations for aorta beneath skin in epigastric area, particularly in thin persons with good muscle wall relaxation
Demeanor
Auscultation
Don’t push stethoscope too hard against body
Auscultate for bruits (swishing during systole) over aorta
Look both renal arteries and iliac arteries
Bowel Sounds
Character and frequency
Happen 5-30 times/min
Abnormal
Hypoactive— from abdominal surgery or with inflammation
Hyperactive— increased motility
Borborgymus from bowel obstruction, gastroenteritis, brisk diarrhea
Listen for 5 minutes before saying bowels sounds are completely absent
Vascular Sounds
User firmer pressures over arteries (especially with people with hypertension)
Light and Deep Palpation
Mild tenderness normally present when palpating sigmoid colon
When identifying a mass, distinguish it from a normally palpable structure or enlarged organ
Noting Mass
Location
Size
Shape
Consistency: soft, firm, hard
Surface: smooth, nodular
Mobility, including movement with respirations
Pulsatility
Tenderness
Palpation of Spleen and Liver
Spleen must be enlarged three time it normal size to be felt
Deep palpation might feel lower edge of liver
Palpation of Aorta
Using your opposing thumb and fingers, palpate aortic pulsation in upper abdomen slightly to left of midline
Pulsates in an anterior direction
Widened in the present of abdominal aortic aneurysm (anormal bulge or ballooning of blood vessel wall)
Aging Adult
May not increased deposits of subcu fat on abdomen and hips because it is redistributed away from extremities
Liver and kidneys easier to palpate
With distended lungs and depressed diaphragm, liver can be palpated lower, descending 1 to 2 cm below costal margin
Costovertebral Angle Tenderness (CVAT)
Positive finding of pain indicates inflammation of the kidney
Urine
Slightly acidic (pH 4.5 - 8.0)
Cloudiness suggests presence of WBC, bacteria, and casts
Daily Fluid Intake
About 15.5 cupts (3.7 L) for men
About 11.5 cups (2/7 L) for women
Water 8-8oz. glasses
Bowl Movement and Stool Characteristics
Usual Elimination pattern (number of stools daily, time of day, routine)
Color
Shape
Consistency (hard, soft)
Changes???
Appetite and nutritional intake (fruits, veggies, roughage)
Fluid intake
Medications
Exercise
Living arrangements
Mobility and dexterity
Note:
Abdominal Distention
Feces palpable in Descending colon
Pain
Abnormal Bowel sounds (hyper, hypo)
Consider constipation, impaction, diarrhea, incontinence, flatulence,
hemorrhoids
Heart and Neck Vessels
Position and Surface Landmarks
Precordium: area on anterior chest overlying the heart and great vessels
Superior and Inferior Vena Cava return unoxygenated venous blood
Pulmonary Artery → (Unoxygenated to) Lungs
Pulmonary Veins → (Oxygenated to) Heart
Pulmonary vs Systemic Circulation
Pericardium - Protects
Myocardium - Muscle
Endocardium - Lines inner surface
Heart Chambers
Four chambers separated by valves (prevent backflow)
Four valves
Two atrioventricular (AV) valves - Separate atria and ventricles
Two semilunar (SL) valves
AV Valves
“Tri(cuspid) before you (bi)cuspid”
Chordae tendineae anchor
SL Valves
No valves are present between vena cava (great vessel putting blood into pulmonary side of heart) and right atrium, or between pulmonary veins (into systemic side) and left atrium
High pressure, left side heart = pulmonary congestion
High pressure, right side heart = shows in neck veins and abdomen
Cardiac Cycle
Diastole: 2/3 of cardiac cycle;
Pressure in the atria is higher than ventricles;
Toward the end of diastole, atria contract and push last mount of blood (25% of stroke volume) into ventricles (called presystole / atrial systole / atrial kick)
Atrial systole occurs during ventricualr diastole
Systole 1/3 of cardiac cycle
AV valve closure contribute to S1 (signals beginning of systole)
For a brief moment all four valves are closed and ventricular walls undergo:
Isometric Contraction (First Stage of Systole):
PART OF SYSTOLE THAT AMPS UP VENTRICULAR PRESSURE TO PREPARE TO SHOOT OUT INTO PULMONARY / SYSTEMIC ARTERY
Contraction against closed system works to build high level pressure in ventricles
Left side heart, when the pressure exceeds the pressure in aorta, the aortic valve opens and blood is ejected rapidly
Pressure falls after ventricles empty
When pressure falls below pressure in the aorta, some blood flows backward toward the ventricle causing the aortic valve to swing shut
Closure of the SL valves causes S2 (signals end of systole)
Diastole Again
Isometric contraction
Four valves closed and ventricles relax
Relaxation of the ventricles (decrease pressure lower than the atria) to allow blood to move into the ventricles (from the atria)
Same events occur on both sides of heart
Right side requires lower pressure and sequence occurs slightly later
Characteristics of Heart Sounds
Frequency
Intensity— loud or soft
Duration— very short for heart sounds; silent periods are longer
Timing— Systole or diastole
Extra Heart Sounds
S3
Ventricles resistant to filling during protodiastole
Immediately after S2
Normal in athletes; indicates congestive HF in people >35 years)
S4
During presystole
Caused by vibration of ventricular wall during atrial contraction
Associated with stiffened ventricle (low ventricular compliance)
Hear in patients with ventricular hypertrophy, myocardial ischemia, or in older adults
Murmurs
Create turbulent blood flow
Conditions resulting in murmurs
Velocity of blood increases
Viscosity of blood decreases
Structural defects in valves
6-point (severity) scale
Grade 1: Easy to miss
Grade 2: Quiet by noticeable
Grade 3: Moderately loud
Grade 4, 5, and 6…
Conduction
Specialized cells in sinoatrial (SA) node initiate electrical impulse
SA Node = pacemaker
Pumping Ability
4-6 L of blood per min (resting)
CO = HR x SV (vol of blood pumped from left ventricle per beat)
Neck Vessels
Carotid artery timing coincides with ventricular systole
Jugular Venous Pulse empties unoxygenated blood into superior vena cava
No valve between vena cava and right atrium
jugular veins give info about activity on right side of heart
Volume and pressure increase when right side of heart fails to pump efficiently
Developmental Competence
Pregnant women = More blood vol and CO / decrease blood pressure
Hemodynamic Changes with Aging
Isolated Systolic HTN: increase in systolic BP due to thickening/stiffening of arteries
Ability of heart to augment CO with exercise is decreased
Dysrhythmias
Presence increases with age (ectopic beats common = may compromise CO and BP when disease present)
Tachyarrhythmias may not be tolerated as well in older people
Culture and Genetics
Cardio Vascular Disease (CVD): most common underlying cause of death globally
Risk Factors
HTN
Smoking
Serum Cholesterol (Low HDL, HIGH LDL)
Inactivity
Sex and gender
Nocturia associated with CVD
Assess jugular veins and precordium while patient is supine (head and chest slightly elevated)
Neck Vessel: Inspection
Jugular Vein Pulse
Assess central venous pressure (judges heart’s efficiency as a pump)
Semi-fowler’s position
Observe for distention
Characteristics of jugular versus carotid pulsations
Differentiate between:
Location
Quality
Respiration
Palpable
Pressure
Position of pt
Neck Vessel: Palpation and Auscultation
Palpate carotid
Feel contour and amplitude of pulse (2+)
Auscultate carotid
Carotid bruit— blowing, swishing sound from blood flow turbulence (use bell, ask patient to take a breath, exhale and hold it briefly while you listen)
Carotid bruit is audible when lumen is occluded by ½ to 2/3 (full occlusion = bruit disappears)
Abnormal Pulsations: Precordium
Thrill = incompetent heart valve
Feels like the throat of a purring cat
Lift (heave) at the sternal border
Can be palpated
Indicates right ventricular hypertrophy
Precordium Auscultation
Valve areas are not the anatomic location of the valves but the sites on the chest wall where sounds produced by the valves are best heard
Note rate and rhythm
S1 and S2
Listen for murmurs
Listen for extra heart sounds
Heart Failure
Decreased CO when heart fails as a pump, causing back up
Signs and symptoms
Heart inability to pump enough blood to meet metabolic demands
Abnormal retention of sodium and water (from kidney) to compensate for decreased CO
Increases blood volume and venous return, causing further congestion
Structure and Function of Arteries
Arteries stretch with systole and recoil with diastole
Vascular smooth muscle = Vasodilation/constriction
Structure and Function: Arteries
Accessible Pulses
Temporal
carotid
brachial
ulnar
popliteal
dorsalis pedis
posterior tibial
Peripheral Arterial disease (PAD) affects noncoronary vessels and refers to arteries affecting limbs
Grading the force
0 = Absent
+1 = Weak
+2
+3 = Bounding, hyperthyroidism, etc.
Structure and Function: Veins
Capacitance vessels
Venous stasis (error in contracting musculoskeletal system and patent lumen)
Risk Factors:
Prolonged standing, sitting, or bed rest (non contracting muscles)
Hypercoagulable states (clots)
Dilated varicose veins = incompetent valves = backflow = increase venous pressure = dilates the vein
Risks: genetic predisposition, obesity, and multiple pregnancies
Structure and Function: Venous Flow
Mechanism to keep blood moving
Contracting skeletal muscles
Pressure gradient from breathing
Intraluminal valves ensure unidirectional flow
Calf pump / peripheral heart
Abnormalities
Varicose veins— dilates and tortuous create incompetent valves → venous pressure → further dilation
DVT— more than 2cm asymmetry, redness and pain
Pitting Edema— bilateral, heart failure, diabetic neuropathy, hepatic cirrhosis
+1 Mild, slight indentation, no perceptible sweeling of leg
+2 Moderate, indentation subsides rapidly
+3 Deep pitting, indentation remains for a short time, leg looks swollen
+4 Very deep pitting, indentation lasts a long time, leg is grossly swollen and distorted
Chronic Arterial Limb Ischemia | Chronic Venous Insufficiency |
Pain- Severe at rest; moving relieves- toes, forefeet, heels | Aching- Relieved by elevation or rest |
Worse at night | Worse later in the day |
Claudication | Cramping- not activity dependent |
Pale feet, rubor red, red to bluish color | Woody, brown, brown pigmented |
Elevation pallor (struggle to move blood up) & dependent rubor | Veins full if leg slightly dependent |
Skin - thin, scaly, dry; Thick nails (deoxygenated tissue) | Skin- Thick, tough, scarring (from blood rushing in periphery) |
Hair loss over calf, ankle, foot (deoxygenated tissue | Premenstrual, salt & water retention (due to pooling and blood secreted in periphery) |
Numbness, Burning, “Toothache” | Itching and burning |
Pulses diminished to absent | Pulses intact |
Ulcers- Distal, concentric, pale | Ulcers- Distal calf, irregular, pink bed, large yellow drainage |
Gangrene & Limb loss | Edema moderate to severe |
Little or no edema |
Structure and Function: Lymphatics
Retrieve excess fluid and plasma proteins
Arteriole hydrostatic pressure leaks out fluid out of the capillaries (more than the venules can absorb)
Colloid osmotic pressure pulls interstitial fluid back into the venules (from plasma proteins being too big to be pushed out of the arterioles, resulting in a force that creates this pressure)
Functions
Vacuum up interstitial fluid
Immune system
Absorb lipids
Spleen Functions
Destroy old RBCs
Produce antibodies
Store RBCs
Filter microbes from blood
Tonsils
Palatine, adenoid, lingual
Aging Adult
Develop arteriosclerosis → more prevalence of PAD
Assess for increased risk for acute DVT and all the risks
Loss of lymphatic tissue leads to fewer numbers of lymph nodes and to decrease in size of remaining nodes
Culture and Genetics
PAD risk for CAD
Environmental factors: Smoking, diabetes, HTN, total levels of cholesterol, obesity
PAD affects blacks more; non-Hispanic blacks highest PAD risk factor
Inspect and Palpate the Arms
Lift the person’s hands in your hands
Inspect hands, including turgor of skin, clubbing, and lesions
Detect early clubbing
Capillary refill
Radial pulse: grade amplitude on a 3 point scale
3+
2+
1+
0
Inspect and Palpate the Legs
Palpate lower extremity pulses using bilateral comparison
Pretibial Edema and Pitting Edema
Check pretibial edema
Depress skin over tibia or medial malleolus for 5 sec and release
Pitting edema scale
1+ Mild pitting, slight indentation, no perceptible swelling
2+ Moderate pitting, indentation subsides rapidly
3+ Deep pitting, indentation remains, leg looks swollen
4+ Very deep pitting, indentation lasts long time, leg grossly swollen and distorted
Use tape measure for more objective data
Pregnant Woman and Aging Adult
Pregnant woman
Expect diffuse bilateral pitting edema in the lower extremities especially at the end of the day
Common findings third trimester
Peripheral pitting edema
Varicose veins
Older adult
Trophic changes associated with arterial insufficiency may be seen
Thin, shiny skin
Thick, ridged nails
Loss of hair on lower legs