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Cerumen Impaction
occurs when earwax accumulates and blocks the external auditory canal.
Clinical Manifestations of Cerumen Impaction
Otalgia
Sensation of fullness in the ear
Ear pain
Hearing loss
Local irritation
Tinnitus
Otalgia
pain in the ear
Irrigation
Suction
Instrumentation
Cerumen and foreign objects may be removed through:
Ear Irrigation
flushing the ear canal with warm water to remove impacted cerumen. It is commonly used when cerumen is not tightly packed in the external auditory canal.
Perforated eardrum
Inflamed external ear
Otitis externa
Gentle irrigation with warm water may be used if the patient does not have:
behind the obstructing cerumen.
For successful removal, the water stream should flow
Use Warm water, Gentle irrigation, Low pressure
Do not use Cold or Warm water, and forceful irrigation
What to use in performing irrigation.
Dizziness
Vertigo
Nausea
Vomiting
Effects of cold water irrigation
Burning
Pain
Tissue irritation
Effects of hot water irrigation
Warmed glycerin
Mineral oil
Half-strength hydrogen peroxide
Before irrigation, cerumen may be softened by instilling a few drops of:
Cerumen curette
Aural suction
Binocular microscope for magnification
If irrigation and softening methods are unsuccessful, cerumen may be removed using instruments such as:
External Otitis / Otitis Externa
inflammation of the external auditory canal. It is commonly known as swimmer's ear when related to water exposure. The most common pathogens are Staphylococcus aureus, Pseudomonas species, and Aspergillus.
Aural tenderness
Pain upon palpation of the ear. usually present in external ear infection but usually not present in middle ear infection.
Tympanic Membrane Perforation
a tear or opening in the eardrum usually caused by infection or trauma.
Tympanoplasty
surgical repair or reconstruction of the tympanic membrane. Tissue, commonly Temporalis fascia, is placed across the perforation to allow healing. It may be performed to Prevent infection from water entering the ear, Improve hearing, and Permanently close the perforation
Acute Otitis Media
an acute infection of the middle ear. It usually lasts less than 6 weeks. Most common in children. Pathogens usually enter the middle ear after eustachian tube dysfunction. a purulent exudate is usually present in the middle ear.
Lasts from 2 weeks to 3 months.
Subacute Otitis Media
Myringotomy / Tympanotomy
Surgical incision into the eardrum to allow for drainage and relieve pressure. The incision usually heals within 24 to 72 hours
Ventilating / Pressure-Equalizing Tube
The tube temporarily replaces the function of the eustachian tube by equalizing pressure. The tube may stay in place for 6 to 18 months. It is later extruded through normal skin migration of the tympanic membrane.
Serous Otitis Media
also called middle ear effusion, is the presence of fluid in the middle ear without active infection. It is commonly caused by negative pressure in the middle ear due to eustachian tube obstruction. The tympanic membrane may appear dull and usually shows conductive hearing loss
Chronic Otitis Media
repeated episodes of acute otitis media causing irreversible tissue damage and persistent tympanic membrane perforation. Chronic infection may Destroy the ossicles, Involve the mastoid, or Lead to cholesteatoma formation. Pain is usually absent unless acute mastoiditis occurs.
Cholesteatoma
a cystlike lesion involving the middle ear. It is formed from skin and sebaceous material. commonly caused by a chronic retraction pocket of the tympanic membrane. This creates persistently high negative pressure in the middle ear.
Tympanoplasty
Ossiculoplasty
Mastoidectomy
Surgical Management of Chronic Otitis Media
Ossiculoplasty
surgical reconstruction of the middle ear bones. It is done to restore hearing by reestablishing the sound conduction mechanism.
Mastoidectomy
surgical removal of diseased mastoid air cells. Remove cholesteatoma. Gain access to diseased structures, Create a dry, healthy, noninfected ear, and Reconstruct ossicles if possible
Otosclerosis
an ear disorder involving abnormal bone formation around the stapes, especially near the oval window. This causes fixation of the stapes. Because the stapes cannot vibrate properly, sound transmission from the malleus and incus to the inner ear is reduced.
Stapedectomy
Stapedotomy
Surgical Management of Otosclerosis
Stapedectomy
involves removing the stapes superstructure and part of the footplate. A tissue graft and prosthesis are inserted. The prosthesis bridges the gap between the incus and the inner ear to improve sound conduction.
Stapedotomy
involves drilling a small hole into the stapes instead of removing it. A prosthesis is inserted into the hole. it improves sound conduction by bridging the gap between the incus and the inner ear.
Dizziness
a general term used to describe an altered sensation of orientation in space. It is often described as Lightheadedness, Feeling unsteady, Feeling faint, or Feeling off balance
Vertigo
the false sensation or illusion of motion. Patients may describe it as Spinning sensation, Feeling like the surroundings are moving, or Feeling like objects are moving around them
Syncope
means fainting or loss of consciousness. It usually suggests a problem with the cardiovascular system.
Motion Sickness
a disturbance of equilibrium caused by conflict in motion receptor stimuli. It happens when the brain receives mixed signals from the eyes, inner ear, and body movement receptors. symptoms are caused by vestibular overstimulation
OTC Antihistamines
Anticholinergics
Management of Motion Sickness
Ménière's Disease
an inner ear disorder caused by abnormal inner ear fluid balance. It may result from Malabsorption in the endolymphatic sac or Blockage in the endolymphatic duct. It is associated with endolymphatic hydrops.
Endolymphatic Hydrops
dilation of the endolymphatic space of the inner ear; the pathologic correlate of Meniere's disease. may cause: Increased pressure in the inner ear or Rupture of the inner ear membrane
Episodic vertigo
Tinnitus
Fluctuating sensorineural hearing loss
Ménière's disease is characterized by a classic triad:
Cochlear Ménière's Disease
also called cochlear hydrops, is a variant of traditional Ménière's disease characterized by isolated inner-ear fluid swelling. Patients experience fluctuating hearing loss, ringing in the ears (tinnitus), and aural fullness, but without the severe, spinning vertigo typical of standard Ménière's
Vestibular Ménière's Disease
a chronic inner ear disorder characterized by recurrent, severe vertigo attacks, fluctuating hearing loss, tinnitus, and aural fullness. Caused by a buildup of fluid (endolymph) in the inner ear.
Dietary Guidelines for Ménière's Disease
Limit foods high in salt (1000 to 1500 mg/day or less).
Limit foods high in sugar.
Be aware of hidden salt and sugar in foods.
Eat meals and snacks at regular intervals.
Avoid skipping meals.
Eat fresh fruits, vegetables, and whole grains.
Limit canned, frozen, and processed foods high in sodium.
Drink plenty of fluids daily.
Choose water, milk, and low-sugar fruit juices.
Limit coffee, tea, and soft drinks.
Avoid caffeine because of its diuretic effect.
Limit alcohol intake.
Avoid monosodium glutamate, or MSG.
Monitor potassium intake if taking potassium-wasting diuretics.
Avoid aspirin and aspirin-containing medications.
Antihistamines
Tranquilizer
Antiemetics
Diuretics
Intratympanic Gentamicin
Pharmacologic Therapy for Ménière's Disease
Endolymphatic sac decompression
Vestibular nerve sectioning
Surgical Management of Ménière's Disease
Endolymphatic Sac Decompression
also called shunting, is a procedure used to equalize pressure in the endolymphatic space. A shunt or drain is inserted into the endolymphatic sac through a postauricular incision. This is often favored as a first-line surgical approach because it is Relatively simple, Safe, and is Possible to perform on an outpatient basis
Vestibular Nerve Sectioning
involves cutting the vestibular nerve to stop vertigo signals from reaching the brain. Cutting the nerve prevents the brain from receiving abnormal input from the semicircular canals.
Nursing Management and Patient Teaching For Dizziness and Vertigo
Change positions slowly.
Avoid sudden head movements.
Sit or lie down during vertigo attacks.
Avoid walking without assistance during severe dizziness.
Keep the environment free from fall hazards.
Use assistive devices if needed.
Report worsening symptoms.
Nursing Management and Patient Teaching For Motion Sickness
Take medication before exposure to motion.
Apply scopolamine patch 4 hours before travel.
Replace scopolamine patch every 3 days.
Avoid driving if drowsy.
Avoid operating heavy machinery if drowsy
Benign Paroxysmal Positional Vertigo (BPPV)
a brief episode of sudden, incapacitating vertigo that occurs when the position of the head changes in relation to gravity. It usually happens when the patient moves the head backward with the affected ear turned downward. believed to be caused by disrupted debris inside the semicircular canal. This debris comes from small crystals of calcium carbonate from the utricle. These crystals are sometimes called canaliths
Clinical Manifestations of BPPV
Brief episodes of vertigo
Sudden onset of dizziness
Positional vertigo
Nausea
Vomiting
Hearing loss usually does not occur
Bed Rest
Canalith Repositioning Procedure
Meclizine for 1 to 2 weeks
Management of BPPV
1. Place the patient in a sitting position.
2. Turn the head to a *45-degree angle* toward the affected side.
3. Quickly move the patient into a supine position.
4. The quick movements help reposition the canalith debris.
5. Prochlorperazine may be given 1 hour before the canalith repositioning procedure.
Canalith Repositioning Procedure:
Vestibular Rehabilitation
a management strategy used for vestibular disorders. It promotes active use of the vestibular system to help the brain compensate for balance impairment.
Tinnitus
perception of sound in one or both ears without an external sound source. It may be a symptom of an underlying ear disorder associated with hearing loss, or it may be benign.
Labyrinthitis
inflammation of the labyrinth of the inner ear. May affect hearing and balance. May be viral or bacterial.
Ramsay Hunt Syndrome
a neurological disorder caused by the reactivation of the varicella-zoster virus (the virus that causes chickenpox and shingles) in the facial nerve. It is characterized by severe ear pain, a blistering rash around the ear, and facial paralysis on the affected side. It may cause labyrinthitis.
Ototoxicity
Refers to damage to the ear caused by medications or substances. It may affect the Cochlea, Vestibular apparatus, and Cranial nerve VIII. May cause Hearing loss, Tinnitus, Balance problems, or Vertigo
Bilateral tinnitus
Aspirin toxicity can produce:
Aminoglycosides
Antineoplastic agents
Aspirin
Quinine
Common causes of ototoxicity include:
Aminoglycosides and antineoplastic agents
are a common cause of ototoxicity because they destroy hair cells in the organ of Corti.
Acoustic Neuroma
also called vestibular schwannoma, is a slow-growing benign tumor of cranial nerve VIII. It usually arises from Schwann cells of the vestibular portion of the nerve. Most are unilateral.