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perfusion A
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atria
receives blood returning from body and lungs
Ventricles of heart
pumps blood to the body and lungs
Septum of heart
separates right and the left side of the heart
Cardiac output
the amount of blood that is ejected from one ventricle in L/min
Cardiac output equation
stroke volume X heart rate
Stroke volume
the volume of blood that is ejected by one ventricle per heartbeat
What 3 factors determine stroke volume
preload, after load, and contractility
afterload
the RESISTANCE OR PRESSURE that left a ventricle that is needed to help eject blood out and for it to circulate and contract
contractility
the force of the heart that the myocardium generates during a contraction (the force)
Atherosclerosis
a narrowing or occlusion of the arteries that can lead to CAD and stable and unstable angina
Coronary Artery Disease
occurs when there is a build up of plaque in the coronary arteries
Atherosclerosis can lead to
decreased oxygenation and perfusion to the myocardium and stable and unstable angina
Angina
chest pain
Stable angina
when you feel chest pain after working out or moving around. Decreased oxygen delivery to the heart muscle. Pain occurs when the heart needs more oxygen than normal. Manifestations last less than 15 minutes
Unstable angina
when you feel chest pain by not doing anything or moving (like sitting on a couch). when an artery is significantly narrows and the heart is ischemic
Variant angina
chest pain due to coronary artery spasm and occurs during periods of rest
Stable angina is relieved by what
rest or nitroglycerin
Treatment of angina
can be meds like nitroglycerin or rest
CAD risk factors
unhealthy eating, not working out, family history, obesity, sex, lack of sleep, diabetes, hypertension, race, lipid levels, smoking, stress, age, homocysteine levels
CAD risk factors that are chronic diseases
hyperlipidemia, thyroid disease, depression, joint pain, BMI 30 or greater, gout, cancer, osteoarthritis
CAD presents as
can be asymptomatic at first, angina, tightness, fullness, pressure in midsternal area, pain radiating from left arm, neck, jaw, or back. Pain from activity aka stable angina or from rest aka unstable angina
CAD manifestations appear when
appear when there is a decreased blood flow and oxygen to the heart
CAD in females, older adults, and diabetics presents as
may not have chest pain (mainly pain in left arm, neck, jaw, or back)
CAD complication
myocardial infarction aka heart attack
Nitroglycerin
a vasodilator that prevents coronary artery vasospasm and reduces preload and after load to help decrease myocardial oxygen demand.
What is nitroglycerin used to treat
angina and control bp
What to monitor for with nitroglycerin
orthostatic hypotension and bp, change positions slowly, headache is COMMON with side effects
How to take nitroglycerin
place pill under tongue and let is dissolve
What to do if pain is unrelieved with nitroglycerin
if not relieved in 5 minutes, call 911 or go to ED. Can take 3 pills within 15 minutes (5 minute intervals)
Angina or MI med management includes
MONA (morphine (for pain), oxygen, nitrate, and aspirin(to help with preventing vasoconstriction and has antiplatelet effects))
How to diagnose for CAD
CARDIAC CATHERTERS, stress tests, x-ray, ECG
CAD lab studies
cholesterol, homocysteine, C-reactive protein, and cardiac enzyme
Role of the nurse with CAD
get a complete history and physical, go over lifestyle habits, occupation (stress, exposure to toxins, irregular shifts and hours), other health conditions, family history, meds, SDofH, weight, diet, smoking, hypertension, hyperlipidemia, diabetes
Procedures for CAD
angioplasty (balloon), coronary artery bypass graft
Meds to take for CAD
ACE, ARBs, beta blockers, calcium channel blockers, nitrates, statins, non-statins, anticoagulants, antiplatelets
Non-pharm changes for CAD
no smoking, heart healthy diet, maintain healthy weight, physical activity, 7-9 hours of sleep
Heart failure is when
the heart is not able to meet the needs and demands of the systemic circulatory system and pump efficently. Ventricles don’t fill properly. Blood is not ejected into the systemic circulatory system properly
New York heart association scale
determines from scale of 1-4 of the s/s of heart disease based on physical activity. 1 is not limitation of physical activity and can do physical activity with no s/s. 4 is you can’t do physical activity or do more exercise without discomfort and has s/s when resting.
American college of cardiology and AHA staging HF stages
stages a-d (1-4) based on if you have s/s of HF and to what degree. Stage A is that you are at risk for HF due don’t have s/s of it. Stage D is that you have Heart Failure and are hospitalized and can die from it
What is ejection fraction
the amount of blood that is pumped out of the heart / amount of blood in the heart chamber
Ejection fraction helps determine
how well the heart is pumping blood to the body
Normal ejection fraction
50-70% pumped out during each contraction
Borderline ejection fraction
41-49% pumped out during each contraction
Reduced ejection fraction
less than 40% is pumped out during contractions (BAD)
Heart failure causes
main is PULMONARY EDEMA, systemic HTN, myocardial infarction, pulmonary hypertension, dysrhythmias, valvular heart disease, pericarditis, cardiomyopathy
Pulmonary edema in HF
a severe and life threatening accumulation of fluid in the alveoli and interstitial spaces of the lung and can result in HEART FAILURE
HF risk factors
family history of heart disease, chronic pulmonary disease, CAD, HTN, chronic infection, diabetes, obesity, metabolic disease, alcohol abuse, cardiotoxic agents, ethnicity
cardiomyopathy
blood circulation to the lungs is IMPAIRED when the cardiac pump is compromised and leads to HF
Cardiomyopathy risk factors
infection or inflammation of the heart muscle, CAD, various cancer treatments, prolonged alcohol use, heredity
Types of cardiomyopathy
dilated, hypertrophic, restrictive
Most common form of cardomyopathy
dilated
Dilated cardiomyopathy
very dilated ventricles which increases ventricle volume but decreases EF. Ventricles enlarges and fill up with a ton of blood but not a lot leaves.
Hypertrophic cardiomyopathy
increased heart muscle size and mass which reduces the ventricle size
Restrictive cardiomyopathy
rigid and stiff ventricular walls that impair filling and ventricular stretch. They are stiff so they have a hard time getting blood in and out
cardiomyopathy manifestations
fatigue, weakness, HF, angina, dysrhythmias aka heart block, s3 gallop, cardiomegaly aka enlarged heart
Left sided HF s/s
pulmonary congestion, dyspnea, SOB, s3 heart sound, tachycardia, nocturnal dyspnea, orthopnea, fatigue, frothy sputum/blood, nocturne, altered mental status
Right sided HF s/s
JVD, SOB, fatigue, weakness, nausea, anorexia, weight gain, ascites, peripheral edema, enlarged liver and spleen, abdominal distention
Biventricular HF
HF on both the left and right sides
HF diagnostic tests
blood tests (BNP), cardiac catheter, ECG, chest x-ray, egg, tee, cardiac enzymes, electrolytes
Role of nurse with HF
assess clients ability to do ADLs, administer O2, med safety, maintain ordered diet restriction (low fluids and low sodium), monitor daily weights, I/O, VS, SOB, encourage bedrest until pt is stable, give emotional support, position pt to max ventilation aka high-fowlers
Non-pharm HF treatments
elevate head of bed to high-fowlers, anxiety relieving techniques, no smoking, weight reduction, use caffeine and alcohol in moderation, heart-healthy diet, physical activity, monitor fluid intake
Meds for HF
ACE, ARBs, calcium channel blockers, inotropic, beta blockers, vasodilators, diuretics
Surgical interventions of HF
cardiac catheter, implant a cardioverter defibrillator, pacemaker, VAS, heart transplant
HF client education
effective breathing techniques, sleep on left side can cause discomfort and breathing issues, understand meds, continue to take meds, report manifestations, reduce modifiable RF, stay on low-sodium diet, restrict fluids, measure weight daily, notify doc if you gain 1-2lbs in a day or 3lbs in a week. Report swelling of feet or ankles or SOB
HF complications
acute pulmonary edema and cariogenic shock
Acute pulmonary edema w HF
a LIFE-THREATENING EMERGENCY. S/s are anxiety, tachycardia, acute respiratory distress, dyspnea at rest, change in level of consciousness, ascending fluid level w lungs (crackles, cough, etc). Effective intervention results in (diuresis, respiratory distress, improved lung sounds, oxygenation)
PROMPT nursing actions w pulmonary edema w HF
Postion client in H-F
Stay with client
Notify provider
Give oxygen
IV morphine
IV admin of loop diuretics and meds to help with CO
Cardiogenic shock with HF
a serious complication of pump failure. S/s: tachycardia, htn, inadequate urinary output, altered level of consciousness, respiratory distress (crackles and tachypnea), cool and clammy skin, chest pain
PROMPT nursing actions w cardiogenic shock w HF
Monitor breath sounds for crackles or wheezing
Monitor heart sounds
Admin oxygen and morphine and diuretics to help decrease preload
Administer IC vasopressors or + ionotrophics to increase CO
Congenital heart defects maternal risk factors
infection, alcohol or substance use with pregnancy, diabetes
Congenital heart defects genetic risk factors
family history, syndromes, presence of other abnormalities
Congenital heart defects that are INCREASED PULMONARY BLOOD FLOW and from L to R
Atrial Septal Defect (ASD)
Ventricular septal defect (VSD)
Patent ductus arteriosus (PDA)
Congenital heart defects that are DECREASED PULMONARY BLOOD FLOW and form R to L
Tetralogy of Fallot
Obstructive defects of congenital heart defects
pulmonary stenosis, aortic stenosis, coarctation of aorta
Mixed defects of congenital heart defects
transportation of great arteries, trunks arteriosus, hypo plastic left heart syndrome
Atrial septal defect (ASD)
a hole in the septum between the right and left atria and results in increased pulmonary blood flow (LEFT TO RIGHT shunt). A systolic murmur and fixed split-second heart sound may be present, heart failure, and is asymptomatic
Ventricular septal defect (VSD)
a hole in the septum between the right and left ventricle and results in increased pulmonary blood flow (LEFT TO RIGHT shunt). Is loud and harsh murmur osculated at the left sternal border, heart failure, and come early in life
Patent ductus arteriosus (PDA)
a normal fetal circulation conduit between the pulmonary artery and the aorta fails to close and results in increased pulmonary blood flow (LEFT TO RIGHT shunt). Has systolic murmur or machine hum, wide pulse pressure, bounding pulses, heart failure, rales, can be asymptomatic
How to treat PDA
administer indomethacin to facilitate closure
Non-surgical treatments of L TO R shunt defects
closure during cardiac catheter, diuretic, extra calories if needed
Surgical treatments of L TO R shunt defects
patch closure, pulmonary artery banding (VSD), insert coils to occlude PDA during cardiac Cath, and thoracoscopic repair (ligate vessels in PDA)
Tetralogy of fallot
4 defects that result in mixed blood flow, has cyanosis at birth and get progressive cyanosis over first year of life. Has a systolic murmur and episodes of acute cyanosis and hypoxia. Goes from R TO L
4 defects in heart for tetralogy of ballot
Overriding aorta
Pulmonic stenosis
Ventricular septal defect
Right ventricular hypertrophy
Surgical procedures of tetralogy of fallout
shunt placement until able to undergo primary repair. And complete repair within the first year of life
Diagnosis of congenital heart defects
chest x-ray, ECG, echocardiogram, cardiac catheter
Nursing care for congenital heart failure
keep child hydrated, conserved Childs energy by giving frequent rest periods, clustering care, and give small and frequent meals. Activities not restricted, do daily weights and I/O, monitor vs and labs, monitor nutrition, maintain fluid and electrolyte balance, give prescribed meds, monitor family coping, maintain sodium and fluid restrictions (not usually needed after correction), position in an infant seat or hold at a 45 degree angles, consult a dietician, do GAVAGE FEED the infant if they are unable to consume enough formula or breast milk, and increase caloric density of formula.
Family education for congenital heart failure
pregnant mother should avoid certain substances and prevent infection, encourage genetic counseling for parents of children w CHDs cause of risk to have a subsequent child w CHD, children with small septal are urged to lead a normal life and often require no med intervention, therapeutic management of other forms of CHD focus on palliative care or surgical correction. ANTIBIOTICS ARE RECOMMENEDED BEFORE DNETAL PROCEDURES to reduce the risk of infective endocarditis
Congenital heart defects impact on health
heart failure, hypoxemia, growth retardation, developmental delay, pulmonary vascular disease, failure to thrive
What are are sequelae of hypoxemia
polycythemia, exercise intolerance, hyper cyanotic spells, brain abscess, CVA