a long, narrow muscle, running obliquely across the front of each thigh from the hipbone to the inside pf the leg below the knee
40
New cards
function of sartorius? How?
flexes and laterally rotates the hip joint and flexes the knee
41
New cards
antagonist to sartorius?
Gracilis- action flexes the knee, adducts the thigh, and helps to medially rotate the tibia on the femur
42
New cards
tensor fasciae latae, what does it do?
flexes an adducts hip ( moves the thigh forward and outward) rotating the thoughts inward, supports the knee
43
New cards
will the iliotibial tract be visible when the knee is bend back?
no
44
New cards
are there other muscles that act on the fascia lata/it band?
yes gluteus maximus- action principal extension
45
New cards
what does contraction speed tell you?
muscle fiber type
46
New cards
how are skeletal muscle fibers categorized?
speed of contraction, primary mode of ATP production
47
New cards
speed of contraction-
fast- twitch fibers and slow-twitch fibers
48
New cards
primary mode of ATP production-
glycolytic fibers vs. oxidative fibers
49
New cards
what determines speed of fiber?
dependent on rate of myosin atpase activity
50
New cards
ATP hydrolysis=
rate limiting step of cycle . higher rate-faster crossbridge cycling
51
New cards
fast fibers-
myosin with fast ATPase activity
52
New cards
slow fibers-
myosin with slow ATPase activity
53
New cards
comparison of fast and slow fiber contractions?
fast fibers contract two to three times faster than slow fiber, fast fibers also relax more rapidly, rate of Ca2+ ATpase is faster, slow fiber contractions last approximately 10 times longer than fast fiber
many maitochondria( why?)bc their work though it, small diameter(why?) weaker, but power is long term, resistant to fatigue
57
New cards
myoglobin(red) (function? why is it necessary?)
stores, binds pigment, oxygen
58
New cards
glycolytic fibers-
fewer mitochondria(why?) not needed, many glycolytic enzymes, high glycogen stores(why?) independent from the bloodstream, relatively little myoglobin(why?) pink color, use little oxygen, large diameter, quick to fatigue
59
New cards
how can you tell the difference between oxidative and glycolytic fibers?
dark is oxidative, lighter is glycolytic
60
New cards
three skeletal muscle fiber types?
slow oxidative(red slow-twitch), fast glycolytic(white fast-twitch, fast oxidative (intermediate fast-twitch
61
New cards
slow oxidative fibers also called?
slow twitch
62
New cards
what makes slow oxidative fibers slow?
low myosin ATPase activity, high oxidative capacity-aerobic(how?) mitochondria rich blood supply, myoglobin( red color)
63
New cards
what does small diameter in regards to function mean for slow twitch? why is this needed again?
little tension, small diffusion barrier, fatigue slowly
64
New cards
fast glycolytic fibers also called?
white fast twitch
65
New cards
fast glycolytic fibers-
high myosin ATPase activity, high glycolytic capacity, high glycogen stores, many glycolytic enzymes, no myoglobin(white), large diameter 2x, can produce greater tension(why?) cross fibers, fatigue rapidly
66
New cards
fast oxidative fibers
High myosin ATPase activity, High oxidative capacity—aerobic, Myoglobin, Slow to fatigue, but more rapid and powerful than slow oxidative, Intermediate diameter
67
New cards
leg muscles contain white fast-twitch fibers while muscles of the arm contain red slow-twitch?
false
68
New cards
distributions of fiber types in a muscles-
one muscle=mixture of fiber types, proportions vary depending on function, all muscles fibers within a moter unit are of the same type
69
New cards
what would you predict would be the predominant fiber type?
slow-twitch or oxidative type
70
New cards
genetices rules
Individuals born with different ratios of slow- to fast-twitch fibers. Athletic conditioning cannot change the genetic component of ability. Training can only optimize what you have.
71
New cards
what is muscle fatigue?
muscles first give 100% when their contract but that can decrease over time
72
New cards
what are the three main causes of muscle fatigue?
build up of lactic acid, compression of blood vessels, depletion of acetylchline( neuromuscular fatigue)
73
New cards
name for the process that results in enlarged muscles?
adaptation
74
New cards
can you die from pooping too hard?
yes
75
New cards
is lab we reflect the skin with superficial fascia, but we stayed on top of the?
rectus sheath
76
New cards
hypertrophy-
increase in muscle size due to change in size of individual cells, increase in size, production of actin and myosin , not cell division
77
New cards
disuse atrophy-
decrease in size, lose myofibers
78
New cards
endurance exercises=
aerobic exercises, low to moderate intesity can be sustained
79
New cards
increase oxidative capacity of muscle-
more mitochondria, increase blood supply( capillaries), decrease in diameter(why) weaker
80
New cards
adaptation to anaerobic exercise
High intensity exercise , Cannot be sustained , Increases ability of muscle to generate more tension (strength), Increases amount actin and myosin, increases number of myofibrils, Increases diameter of muscle fiber , Increases glycolytic enzymes, Decreases oxidative capacity
81
New cards
How did a feud between two scientists in the late 1700s ultimately lead to the creation of Frankenstein and all battery-powered devices like your cell phone?
curiosity, animal electricity,
82
New cards
luigi galvani
in 1771, he discovered that the muscles of dead frogs legs twitched when struck by a spark, animal electricity
83
New cards
alessandro volta
did not beleive animal was the source of the lectricity, built the fist battery(voltaic pile) to prove it
84
New cards
aldinis gruesome experiments provided one of the inspirations for…?
Mary shelley’s Frankensteinmuscl
85
New cards
muscular dystrophy-
A group of familial diseases of skeletal muscle, classified by age of onset, rate of progression, muscle involvement, and inheritance pattern, Usually the result of one faulty (mutated) gene, Duchenne MD is the most common 1 in 3500 males (usually only in males), Lack of functional dystrophin gene
86
New cards
what does dystrophin do?
acts as a raft, its anchored, the chain is the dystrophin, the bottom of the lake is the membrane
87
New cards
what is the basal lamina?
extracellular matrix(ECM)- allows cell to attach to one another
88
New cards
what is link to ECM?
tug-of-war, protein called laminin
89
New cards
how does the lack of dystrophin affect the muscle fiber?
laminin provides connection, shorten, tension is applied, little holes form
90
New cards
why do children with muscular dystrophy stand up weird?
they walk themselves up their legs because their muscles are too weak, uses hands to push on legs to stand
91
New cards
contracture-
a condition of shortening and hardening of muscles, tendons or other tissue, often leading to deformity and rigidity of joints
92
New cards
why the need for mechanical ventilation? cause of death?
it helps them breath, cause for death is respiration failure not be able to move, bad circulation, lung infections
93
New cards
posterior forearm AKA?
antebrachium
94
New cards
antebrachium means
the part of the arm or forelimb between the branchium and the carpus
95
New cards
what is visible immediately after skin removal?
veins, antebrachial fasiz
96
New cards
analogous structure in legs?
crual fasia
97
New cards
removal of antebrachium exposes what?
superficial extensor muscles as well as the brachioradialis